Literature DB >> 7796227

Excitatory amino acids in neonatal brain: contributions to pathology and therapeutic strategies.

S W D'Souza1, P Slater.   

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Year:  1995        PMID： 7796227      PMCID： PMC2528440          DOI： 10.1136/fn.72.3.f147

Source DB:  PubMed          Journal:  Arch Dis Child Fetal Neonatal Ed        ISSN： 1359-2998            Impact factor:   5.747


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Review 2.  The TiPS/TINS lecture: the molecular biology of mammalian glutamate receptor channels.

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3.  Autoradiographic characterization of N-methyl-D-aspartate-, quisqualate- and kainate-sensitive glutamate binding sites.

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4.  Cloning, functional coexpression, and pharmacological characterisation of human cDNAs encoding NMDA receptor NR1 and NR2A subunits.

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6.  Glycine site of the excitatory amino acid N-methyl-D-aspartate receptor in neonatal and adult brain.

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7.  Extracellular overflow of neuroactive amino acids during severe insulin-induced hypoglycemia: in vivo dialysis of the rat hippocampus.

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8.  The dissociative anaesthetics, ketamine and phencyclidine, selectively reduce excitation of central mammalian neurones by N-methyl-aspartate.

Authors:  N A Anis; S C Berry; N R Burton; D Lodge
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9.  Ischemia-induced shift of inhibitory and excitatory amino acids from intra- to extracellular compartments.

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10.  CPP, a new potent and selective NMDA antagonist. Depression of central neuron responses, affinity for [3H]D-AP5 binding sites on brain membranes and anticonvulsant activity.

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Review 1.  Treating disorders of the neonatal central nervous system: pharmacokinetic and pharmacodynamic considerations with a focus on antiepileptics.

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Review 2.  Newer concepts and approaches to neonatal brain asphyxia.

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4.  Lentiviral expression of calpain-1 C2-like domain peptide prevents glutamate-induced cell death in mouse hippocampal neuronal HT22 cells.

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Review 5.  Pharmacotherapy for Neonatal Seizures: Current Knowledge and Future Perspectives.

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6.  Early Changes in Glutamate Metabolism and Perfusion in Basal Ganglia following Hypoxia-Ischemia in Neonatal Piglets: A Multi-Sequence 3.0T MR Study.

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