Literature DB >> 7583141

Transcriptional regulation. Flipping the Myc switch.

R Bernards1.   

Abstract

When certain cells differentiate, Myc in Myc-Max heterodimers is replaced by Mad or Mxi, generating heterodimers that suppress transcription by interacting with the repressor Sin3.

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Year:  1995        PMID： 7583141     DOI： 10.1016/s0960-9822(95)00173-4

Source DB:  PubMed          Journal:  Curr Biol        ISSN： 0960-9822            Impact factor:   10.834


  5 in total

1.  MondoA, a novel basic helix-loop-helix-leucine zipper transcriptional activator that constitutes a positive branch of a max-like network.

Authors:  A N Billin; A L Eilers; K L Coulter; J S Logan; D E Ayer
Journal:  Mol Cell Biol       Date:  2000-12       Impact factor: 4.272

2.  A role for c-myc in chemically induced renal-cell death.

Authors:  Y Zhan; J L Cleveland; J L Stevens
Journal:  Mol Cell Biol       Date:  1997-11       Impact factor: 4.272

3.  N-myc over-expression downregulates alpha3beta1 integrin expression in human Saos-2 osteosarcoma cells.

Authors:  R Judware; L A Culp
Journal:  Clin Exp Metastasis       Date:  1997-05       Impact factor: 5.150

4.  Mad proteins contain a dominant transcription repression domain.

Authors:  D E Ayer; C D Laherty; Q A Lawrence; A P Armstrong; R N Eisenman
Journal:  Mol Cell Biol       Date:  1996-10       Impact factor: 4.272

5.  Fast and systematic genome-wide discovery of conserved regulatory elements using a non-alignment based approach.

Authors:  Olivier Elemento; Saeed Tavazoie
Journal:  Genome Biol       Date:  2005-01-26       Impact factor: 13.583
  5 in total

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