Literature DB >> 7502522

Inflammation, acid and ulcers.

M J Muller1, R H Hunt.   

Abstract

Chronic active type B gastritis is invariably the result of Helicobacter pylori infection and is an important factor in duodenal ulcer disease. The actions of mediators produced (a protein factor, a lipid soluble "pore-forming factor" and urease) or induced (immune/inflammatory cell mediators) by this bacterium on the control of gastric acid secretion are currently being investigated. These studies are reviewed in light of our current knowledge of the physiological control of gastric acid secretion.

Entities:  

Mesh：

Year:  1994        PMID： 7502522      PMCID： PMC2588934     

Source DB:  PubMed          Journal:  Yale J Biol Med        ISSN： 0044-0086


  59 in total

1.  Helicobacter and hypergastrinemia: the Quisling option.

Authors:  P K Rangachari
Journal:  Scand J Gastroenterol Suppl       Date:  1991

2.  The identification of urogastrone in serum, saliva, and gastric juice.

Authors:  H Gregory; S Walsh; C R Hopkins
Journal:  Gastroenterology       Date:  1979-08       Impact factor: 22.682

3.  Unidentified curved bacilli on gastric epithelium in active chronic gastritis.

Authors:  J R Warren; B Marshall
Journal:  Lancet       Date:  1983-06-04       Impact factor: 79.321

4.  Eradicating Helicobacter pylori infection lowers gastrin mediated acid secretion by two thirds in patients with duodenal ulcer.

Authors:  E el-Omar; I Penman; C A Dorrian; J E Ardill; K E McColl
Journal:  Gut       Date:  1993-08       Impact factor: 23.059

Review 5.  Biology of interleukin 1.

Authors:  C A Dinarello
Journal:  FASEB J       Date:  1988-02       Impact factor: 5.191

6.  Mucosal tumour necrosis factor alpha and interleukin-6 in patients with Helicobacter pylori associated gastritis.

Authors:  J E Crabtree; T M Shallcross; R V Heatley; J I Wyatt
Journal:  Gut       Date:  1991-12       Impact factor: 23.059

7.  Mechanisms underlying the protective effects of interleukin 1 in experimental nonsteroidal anti-inflammatory drug gastropathy.

Authors:  J L Wallace; C M Keenan; M Cucala; K G Mugridge; L Parente
Journal:  Gastroenterology       Date:  1992-04       Impact factor: 22.682

8.  Helicobacter pylori infection induces a decrease in immunoreactive-somatostatin concentrations of human stomach.

Authors:  H Kaneko; K Nakada; T Mitsuma; K Uchida; A Furusawa; Y Maeda; K Morise
Journal:  Dig Dis Sci       Date:  1992-03       Impact factor: 3.199

Review 9.  Hypotheses on the pathogenesis and natural history of Helicobacter pylori-induced inflammation.

Authors:  M J Blaser
Journal:  Gastroenterology       Date:  1992-02       Impact factor: 22.682

10.  Campylobacter pyloridis and gastritis: association with intercellular spaces and adaptation to an environment of mucus as important factors in colonization of the gastric epithelium.

Authors:  S L Hazell; A Lee; L Brady; W Hennessy
Journal:  J Infect Dis       Date:  1986-04       Impact factor: 5.226
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  1 in total

Review 1.  pH, healing rate and symptom relief in acid-related diseases.

Authors:  J Q Huang; R H Hunt
Journal:  Yale J Biol Med       Date:  1996 Mar-Apr
  1 in total

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