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7 in total

1. External biliary drainage plus bile acid feeding is not equal to internal drainage in preserving the cellular immunity following prolonged obstructive jaundice.

Authors: K T Mok; B W Wang; H C Chang; S L Lin
Journal: Dig Dis Sci Date: 2001-09 Impact factor: 3.199

2. Increased concentrations of tumour necrosis factor (TNF) and soluble TNF receptors in biliary obstruction in mice; soluble TNF receptors as prognostic factors for mortality.

Authors: M H Bemelmans; J W Greve; D J Gouma; W A Buurman
Journal: Gut Date: 1996-03 Impact factor: 23.059

3. Soluble interleukin-2 receptor and soluble CD8 in liver cirrhosis and obstructive jaundice.

Authors: F Wagner; C Assemi; C Lersch; R Hart; M Classen
Journal: Clin Exp Immunol Date: 1990-11 Impact factor: 4.330

4. Obstructive jaundice expands intrahepatic regulatory T cells, which impair liver T lymphocyte function but modulate liver cholestasis and fibrosis.

Authors: Steven C Katz; Kristin Ryan; Naseem Ahmed; George Plitas; Umer I Chaudhry; T Peter Kingham; Seema Naheed; Cang Nguyen; Ponnandai Somasundar; N Joseph Espat; Richard P Junghans; Ronald P Dematteo
Journal: J Immunol Date: 2011-06-22 Impact factor: 5.422

Immune status in mice with experimental biliary obstruction.

1. External biliary drainage plus bile acid feeding is not equal to internal drainage in preserving the cellular immunity following prolonged obstructive jaundice.

2. Increased concentrations of tumour necrosis factor (TNF) and soluble TNF receptors in biliary obstruction in mice; soluble TNF receptors as prognostic factors for mortality.

3. Soluble interleukin-2 receptor and soluble CD8 in liver cirrhosis and obstructive jaundice.

4. Obstructive jaundice expands intrahepatic regulatory T cells, which impair liver T lymphocyte function but modulate liver cholestasis and fibrosis.

5. Role of the gut in the pathophysiology of extrahepatic biliary obstruction.

6. Effect of arginine on immune function in rats with obstructive jaundice.

7. Interleukin 2 production and its relationship with T lymphocyte subsets in patients with obstructive jaundice.