Literature DB >> 6884512

Stimulation of glycogenolysis in hepatocytes by angiotensin II may involve both calcium release and calcium influx.

L M DeWitt, J W Putney.   

Abstract

The stimulation of [3H]glucose release (a measure of glycogenolysis) from isolated guinea pig hepatocytes by Ca-mobilizing agonists can be resolved into two phases. The initial transient phase is independent of extracellular Ca, and is probably a result of Ca released from an intracellular pool. The second phase occurs only in the presence of extracellular Ca, which suggests that Ca-influx is also involved in the mechanism of Ca-mobilization by these agents in the guinea pig hepatocyte.

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Year:  1983        PMID： 6884512     DOI： 10.1016/0014-5793(83)80978-8

Source DB:  PubMed          Journal:  FEBS Lett        ISSN： 0014-5793            Impact factor:   4.124


  11 in total

1.  Gluconeogenesis stimulated by extracellular ATP is triggered by the initial increase in the intracellular Ca2+ concentration of the periphery of hepatocytes.

Authors:  M Koike; T Kashiwagura; N Takeguchi
Journal:  Biochem J       Date:  1992-04-01       Impact factor: 3.857

2.  Effects of vasopressin and La3+ on plasma-membrane Ca2+ inflow and Ca2+ disposition in isolated hepatocytes. Evidence that vasopressin inhibits Ca2+ disposition.

Authors:  B P Hughes; S E Milton; G J Barritt
Journal:  Biochem J       Date:  1986-09-15       Impact factor: 3.857

3.  P2-purinergic control of liver glycogenolysis.

Authors:  S Keppens; H De Wulf
Journal:  Biochem J       Date:  1985-11-01       Impact factor: 3.857

4.  Effect of cyclic AMP-dependent hormones and Ca2+-mobilizing hormones on the Ca2+ influx and polyphosphoinositide metabolism in isolated rat hepatocytes.

Authors:  J Poggioli; J P Mauger; M Claret
Journal:  Biochem J       Date:  1986-05-01       Impact factor: 3.857

5.  Stimulation of glycogenolysis by adenine nucleotides in the perfused rat liver.

Authors:  D B Buxton; S M Robertson; M S Olson
Journal:  Biochem J       Date:  1986-08-01       Impact factor: 3.857

6.  Release of Ca2+ from the endoplasmic reticulum is not the mechanism for bile acid-induced cholestasis and hepatotoxicity in the intact rat liver.

Authors:  G C Farrell; S K Duddy; G E Kass; J Llopis; A Gahm; S Orrenius
Journal:  J Clin Invest       Date:  1990-04       Impact factor: 14.808

7.  Stimulation of glucose production from glycogen by glucagon, noradrenaline and non-degradable adenosine analogues is counteracted by adenosine and ATP in cultured rat hepatocytes.

Authors:  E Oetjen; C Schweickhardt; K Unthan-Fechner; I Probst
Journal:  Biochem J       Date:  1990-10-15       Impact factor: 3.857

8.  The effects of the renin-angiotensin-aldosterone system gene polymorphisms on insulin resistance in hypertensive families.

Authors:  Chin-Fu Hsiao; Wayne W H Sheu; Yi-Jen Hung; Ming-Wei Lin; David Curb; Koustubh Ranadex; Thomas Quertermous; Yue-Ming Chen; Ida Yi-Der Chen; Kwan-Dun Wu
Journal:  J Renin Angiotensin Aldosterone Syst       Date:  2012-03-14       Impact factor: 1.636

9.  Evidence for adrenergic control of transcellular calcium distribution in liver.

Authors:  C E Hill; A P Dawson; J S Pryor
Journal:  Biochem J       Date:  1985-09-15       Impact factor: 3.857

10.  Evidence that a pertussis-toxin-sensitive substrate is involved in the stimulation by epidermal growth factor and vasopressin of plasma-membrane Ca2+ inflow in hepatocytes.

Authors:  B P Hughes; J N Crofts; A M Auld; L C Read; G J Barritt
Journal:  Biochem J       Date:  1987-12-15       Impact factor: 3.857
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