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Literature DB >> 6766900

Temporary increase in chromosome breakage in an infant prenatally exposed to lead.

Abstract

An infant exposed to high levels of lead in utero was found to have increased numbers of cells with chromosome breaks in blood samples obtained at 6 weeks and 3 months of life. Later samples did not show significant abnormality. Physical and neurological examinations of the patient up to 18 months of age gave results within normal limits.

Entities: Species

Mesh：

Substances：
Lead
Edetic Acid

Year: 1980 PMID： 6766900 DOI： 10.1007/bf00273496

Source DB: PubMed Journal: Hum Genet ISSN： 0340-6717 Impact factor: 4.132

13 in total

1. LEAD POISONING DURING PREGNANCY. FETAL TOLERANCE OF CALCIUM DISODIUM EDETATE.

Authors: C R ANGLE; M S MCINTIRE
Journal: Am J Dis Child Date: 1964-10

2. Chromosome preparations of leukocytes cultured from human peripheral blood.

Authors: P S MOORHEAD; P C NOWELL; W J MELLMAN; D M BATTIPS; D A HUNGERFORD
Journal: Exp Cell Res Date: 1960-09 Impact factor: 3.905

3. Distribution of spontaneous chromosome breaks in human chromosomes.

Authors: P Aula; H von Koskull
Journal: Hum Genet Date: 1976-05-19 Impact factor: 4.132

4. [Chromosome investigations in subjects with occupational lead exposure (author's transl)].

Authors: G Schwanitz; E Gebhart; H D Rott; K H Schaller; H G Essing; O Lauer; H Prestele
Journal: Dtsch Med Wochenschr Date: 1975-05-02 Impact factor: 0.628

5. The Blastophthoric Effect of Chronic Lead Poisoning : I. Introduction. Clinical and experimental observations from the Literature.

Authors: C V Weller
Journal: J Med Res Date: 1915-11

Temporary increase in chromosome breakage in an infant prenatally exposed to lead.

1. LEAD POISONING DURING PREGNANCY. FETAL TOLERANCE OF CALCIUM DISODIUM EDETATE.

2. Chromosome preparations of leukocytes cultured from human peripheral blood.

3. Distribution of spontaneous chromosome breaks in human chromosomes.

4. [Chromosome investigations in subjects with occupational lead exposure (author's transl)].

5. The Blastophthoric Effect of Chronic Lead Poisoning : I. Introduction. Clinical and experimental observations from the Literature.

6. Chromosome damage in experimental lead poisoning.

7. Maternal lead exposure and blood lead concentration in infancy.

8. Transport of lead 203 and calcium 47 from mother to offspring.

9. Neonatal lead intoxication in a prenatally exposed infant.

10. Developmental malformations resulting from the administration of lead salts.

1. Neonatal lead poisoning from maternal pica behavior during pregnancy.