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Literature DB >> 6436898

Effects of intermittent haloperidol treatment on dopamine receptor sensitivity in guinea pigs.

Abstract

Intermittent haloperidol treatment in guinea pigs caused an increase in the behavioral response to apomorphine and an increase in 3H-spiroperidol binding in the striatum to the same degree as continual haloperidol treatment. These results do not support the claim that drug holidays can reduce the incidence of tardive dyskinesia.

Entities: Chemical Disease Species

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Year: 1984 PMID： 6436898 DOI： 10.1007/bf00432034

Source DB: PubMed Journal: Psychopharmacology (Berl) ISSN： 0033-3158 Impact factor: 4.530

11 in total

1. Biochemical demonstration of dopaminergic receptors in rat and human brain using [3H]spiroperidol.

Authors: J Z Fields; T D Reisine; H I Yamamura
Journal: Brain Res Date: 1977-11-18 Impact factor: 3.252

2. [On the problem of persistent extrapyramidal hyperkinesia following prolonged use of neuroleptics].

Authors: R Degkwitz; K F Binsack; H Herkert; O Luxenburger; W Wenzel
Journal: Nervenarzt Date: 1967-04 Impact factor: 1.214

3. Behavioural supersensitivity to apomorphine following chronic treatment with drugs which interfere with the synaptic function of catecholamines.

Authors: D Tarsy; R J Baldessarini
Journal: Neuropharmacology Date: 1974-11 Impact factor: 5.250

8. Differential effects of abrupt versus gradual withdrawal of chlorpromazine in hospitalized chronic schizophrenic patients.

Authors: L M Greenberg; S Roth
Journal: Am J Psychiatry Date: 1966-08 Impact factor: 18.112

2. Persistent vacuous chewing in rats following neuroleptic treatment: relationship to dopaminergic and cholinergic function.

Authors: B Glenthøj
Journal: Psychopharmacology (Berl) Date: 1993 Impact factor: 4.530

3. Sensitization to haloperidol-induced suppression of milk intake: effect of interdose interval.

Authors: D L Wolgin; J Moore
Journal: Psychopharmacology (Berl) Date: 1992 Impact factor: 4.530

4. Effect of different neuroleptics in tardive dyskinesia and parkinsonism. A video-controlled multicenter study with chlorprothixene, perphenazine, haloperidol and haloperidol + biperiden. Nordic Dyskinesia Study Group.

Authors:
Journal: Psychopharmacology (Berl) Date: 1986 Impact factor: 4.530

5. Autoradiographic analysis of regional alterations in brain receptors following chronic administration and withdrawal of typical and atypical neuroleptics in rats.

Authors: R E See; A W Toga; G Ellison
Journal: J Neural Transm Gen Sect Date: 1990

5 in total

Effects of intermittent haloperidol treatment on dopamine receptor sensitivity in guinea pigs.

1. Biochemical demonstration of dopaminergic receptors in rat and human brain using [3H]spiroperidol.

2. [On the problem of persistent extrapyramidal hyperkinesia following prolonged use of neuroleptics].

3. Behavioural supersensitivity to apomorphine following chronic treatment with drugs which interfere with the synaptic function of catecholamines.

4. Animal models of tardive dyskinesias.

5. The effect of drug holidays in an animal model of tardive dyskinesia.

6. The effects of a drug holiday on relapse and tardive dyskinesia in chronic schizophrenics [proceedings].

7. Tardive dyskinesia--reversible and persistent.

8. Differential effects of abrupt versus gradual withdrawal of chlorpromazine in hospitalized chronic schizophrenic patients.

Review 9. Tardive dyskinesia: prevalence and risk factors, 1959 to 1979.

10. Tardive dyskinesia: review and update.

1. Intermittent and continuous haloperidol regimens produce different types of oral dyskinesias in rats.

2. Persistent vacuous chewing in rats following neuroleptic treatment: relationship to dopaminergic and cholinergic function.

3. Sensitization to haloperidol-induced suppression of milk intake: effect of interdose interval.

4. Effect of different neuroleptics in tardive dyskinesia and parkinsonism. A video-controlled multicenter study with chlorprothixene, perphenazine, haloperidol and haloperidol + biperiden. Nordic Dyskinesia Study Group.

5. Autoradiographic analysis of regional alterations in brain receptors following chronic administration and withdrawal of typical and atypical neuroleptics in rats.