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Literature DB >> 6209739

Control of replication of bacterial plasmids: genetics, molecular biology, and physiology of the plasmid R1 system.

Abstract

Plasmids are autonomously replicating DNA molecules that are present in defined copy numbers in bacteria. This number may for some plasmids be very low (2-5 per average cell). In order to be stably inherited, replication and partitioning of the plasmid have to be strictly controlled. Plasmids carry genetic information for both processes. In the present paper we summarize what is known about the replication control system of one low-copy-number plasmid, R1, belonging to the FII incompatibility group. We do so because the FII group seems to be one of the best understood examples with respect to genetics, molecular biology, and physiology of the replication control system. The paper is not a classical review, but rather an essay in which we discuss the aspects of replication control that we regard as being important.

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Year: 1984 PMID： 6209739 DOI： 10.1016/0147-619x(84)90054-4

Source DB: PubMed Journal: Plasmid ISSN： 0147-619X Impact factor: 3.466

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Cited

67 in total

1. Progression of a loop-loop complex to a four-way junction is crucial for the activity of a regulatory antisense RNA.

Authors: F A Kolb; H M Engdahl; J G Slagter-Jäger; B Ehresmann; C Ehresmann; E Westhof; E G Wagner; P Romby
Journal: EMBO J Date: 2000-11-01 Impact factor: 11.598

2. Natural selection, infectious transfer and the existence conditions for bacterial plasmids.

Authors: C T Bergstrom; M Lipsitch; B R Levin
Journal: Genetics Date: 2000-08 Impact factor: 4.562

3. Bulged residues promote the progression of a loop-loop interaction to a stable and inhibitory antisense-target RNA complex.

Authors: F A Kolb; E Westhof; C Ehresmann; B Ehresmann; E G Wagner; P Romby
Journal: Nucleic Acids Res Date: 2001-08-01 Impact factor: 16.971

10. Suppression of replication-deficient mutants of IncFII plasmid NR1 can occur by two different mechanisms that increase expression of the repA1 gene.

Authors: R Wu; X Wang; D D Womble; R H Rownd
Journal: J Bacteriol Date: 1993-05 Impact factor: 3.490

Control of replication of bacterial plasmids: genetics, molecular biology, and physiology of the plasmid R1 system.

1. Progression of a loop-loop complex to a four-way junction is crucial for the activity of a regulatory antisense RNA.

2. Natural selection, infectious transfer and the existence conditions for bacterial plasmids.

3. Bulged residues promote the progression of a loop-loop interaction to a stable and inhibitory antisense-target RNA complex.

4. High affinity nucleic acid aptamers for streptavidin incorporated into bi-specific capture ligands.

5. The effect of loop size in antisense and target RNAs on the efficiency of antisense RNA control.

6. Evaluating quantitative methods for measuring plasmid copy numbers in single cells.

7. Inhibition of protein synthesis transiently stimulates initiation of minichromosome replication in Escherichia coli.

8. Nucleotide sequence and replication characteristics of RepFIB, a basic replicon of IncF plasmids.

Review 9. Copy-number control of the Escherichia coli chromosome: a plasmidologist's view.

10. Suppression of replication-deficient mutants of IncFII plasmid NR1 can occur by two different mechanisms that increase expression of the repA1 gene.