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Literature DB >> 5839721

Formal discussion of: somatic mutations and carcinogenesis.

H J Curtis.

Abstract

Mesh：

Year: 1965 PMID： 5839721

Source DB: PubMed Journal: Cancer Res ISSN： 0008-5472 Impact factor: 12.701

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9 in total

1. The occurrence of mitotic irregularities during a normal epithelial proliferation.

Authors: J G Forsberg
Journal: Experientia Date: 1967-10-15

Review 2. Changing mutational and adaptive landscapes and the genesis of cancer.

Authors: L Alexander Liggett; James DeGregori
Journal: Biochim Biophys Acta Rev Cancer Date: 2017-02-04 Impact factor: 10.680

Review 3. Cancer attractors: a systems view of tumors from a gene network dynamics and developmental perspective.

Authors: Sui Huang; Ingemar Ernberg; Stuart Kauffman
Journal: Semin Cell Dev Biol Date: 2009-07-10 Impact factor: 7.727

4. Significance and limitations of the use of next-generation sequencing technologies for detecting mutational signatures.

Authors: Ammal Abbasi; Ludmil B Alexandrov
Journal: DNA Repair (Amst) Date: 2021-08-05

Review 5. Clonal Hematopoiesis Analyses in Clinical, Epidemiologic, and Genetic Aging Studies to Unravel Underlying Mechanisms of Age-Related Dysfunction in Humans.

Authors: Kenneth Walsh; Nalini Raghavachari; Candace Kerr; Alexander G Bick; Steven R Cummings; Todd Druley; Cynthia E Dunbar; Giulio Genovese; Margaret A Goodell; Siddhartha Jaiswal; Jaroslaw Maciejewski; Pradeep Natarajan; Anastasia V Shindyapina; Alan R Shuldiner; Erik B Van Den Akker; Jan Vijg
Journal: Front Aging Date: 2022-03-08

Formal discussion of: somatic mutations and carcinogenesis.

1. The occurrence of mitotic irregularities during a normal epithelial proliferation.

Review 2. Changing mutational and adaptive landscapes and the genesis of cancer.

Review 3. Cancer attractors: a systems view of tumors from a gene network dynamics and developmental perspective.

4. Significance and limitations of the use of next-generation sequencing technologies for detecting mutational signatures.

Review 5. Clonal Hematopoiesis Analyses in Clinical, Epidemiologic, and Genetic Aging Studies to Unravel Underlying Mechanisms of Age-Related Dysfunction in Humans.

Review 6. Non-genetic heterogeneity--a mutation-independent driving force for the somatic evolution of tumours.

Review 7. Emergentism as a default: cancer as a problem of tissue organization.

8. Loss of function of e-cadherin in embryonic stem cells and the relevance to models of tumorigenesis.

9. Synonymous mutations in oncogenesis and apoptosis versus survival unveiled by network modeling.