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Literature DB >> 3800982

Sex- and phenotype-dependent metabolism of N tau-methylhistidine by mice.

C I Harris, G J Rucklidge, R M McDiarmid, G Milne.

Abstract

The metabolism of N tau-[Me-14C]methylhistidine by mice was influenced by both the sex and the phenotype of the animal. After subcutaneous injection of labelled N tau-methylhistidine, recoveries of radioactivity in excreta were incomplete, and the distributions of radioactivity showed that different mouse phenotypes degraded N tau-methylhistidine in vivo to different extents. Hence the excretion of N tau-methylhistidine by mice cannot be used as an index of protein breakdown in vivo.

Entities: Chemical Species

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Year: 1986 PMID： 3800982 PMCID： PMC1147265 DOI： 10.1042/bj2390229

Source DB: PubMed Journal: Biochem J ISSN： 0264-6021 Impact factor: 3.857

8 in total

1. Growth of adipose and lean tissue mass in hindlimbs of genetically obese mice during preobese and obese phases of development.

Authors: W G Bergen; M L Kaplan; R A Merkel; G A Leveille
Journal: Am J Clin Nutr Date: 1975-02 Impact factor: 7.045

2. Metabolism of administered 3-methylhistidine. Lack of muscle transfer ribonucleic acid charging and quantitative excretion as 3-methylhistidine and its N-acetyl derivative.

Authors: V R Young; S D Alexis; B S Baliga; H N Munro; W Muecke
Journal: J Biol Chem Date: 1972-06-10 Impact factor: 5.157

1 in total

1. Effect of postdevelopmental myostatin depletion on myofibrillar protein metabolism.

Authors: Stephen Welle; Sangeeta Mehta; Kerri Burgess
Journal: Am J Physiol Endocrinol Metab Date: 2011-03-15 Impact factor: 4.310

1 in total

Sex- and phenotype-dependent metabolism of N tau-methylhistidine by mice.

1. Growth of adipose and lean tissue mass in hindlimbs of genetically obese mice during preobese and obese phases of development.

2. Metabolism of administered 3-methylhistidine. Lack of muscle transfer ribonucleic acid charging and quantitative excretion as 3-methylhistidine and its N-acetyl derivative.

3. A new metabolism cage suitable for the study of mice.

4. The occurrence of the N tau-methylhistidine-containing dipeptide, balenine, in muscle extracts of various mammals.

5. Skeletal muscle accretion and turnover in lean and obese (ob/ob) mice.

6. The urinary excretion of N tau-methyl histidine in sheep: an invalid index of muscle protein breakdown.

7. The inadequacy of urinary N tau-methyl histidine excretion in the pig as a measure of muscle protein breakdown.

8. The urinary excretion of N tau-methyl histidine by cattle: validation as an index of muscle protein breakdown.

1. Effect of postdevelopmental myostatin depletion on myofibrillar protein metabolism.