Literature DB >> 35556214

Harnessing NAD+ Metabolism as Therapy for Cardiometabolic Diseases.

Akash Chakraborty1,2, Keaton E Minor1,3, Hina Lateef Nizami1, Ying Ann Chiao2,4, Chi Fung Lee5,6.   

Abstract

PURPOSE OF THE REVIEW: This review summarizes current understanding on the roles of nicotinamide adenine dinucleotide (NAD+) metabolism in the pathogeneses and treatment development of metabolic and cardiac diseases. RECENT
FINDINGS: NAD+ was identified as a redox cofactor in metabolism and a co-substrate for a wide range of NAD+-dependent enzymes. NAD+ redox imbalance and depletion are associated with many pathologies where metabolism plays a key role, for example cardiometabolic diseases. This review is to delineate the current knowledge about harnessing NAD+ metabolism as potential therapy for cardiometabolic diseases. The review has summarized how NAD+ redox imbalance and depletion contribute to the pathogeneses of cardiometabolic diseases. Therapeutic evidence involving activation of NAD+ synthesis in pre-clinical and clinical studies was discussed. While activation of NAD+ synthesis shows great promise for therapy, the field of NAD+ metabolism is rapidly evolving. Therefore, it is expected that new mechanisms will be discovered as therapeutic targets for cardiometabolic diseases.
© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

Entities:  

Keywords:  Cardiometabolic diseases; Heart failure; NAD+ metabolism; Redox balance

Mesh:

Substances:

Year:  2022        PMID: 35556214      PMCID: PMC9339518          DOI: 10.1007/s11897-022-00550-5

Source DB:  PubMed          Journal:  Curr Heart Fail Rep        ISSN: 1546-9530


  123 in total

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Authors:  Dan Huang; Cristel V Camacho; Rohit Setlem; Keun Woo Ryu; Balaji Parameswaran; Rana K Gupta; W Lee Kraus
Journal:  Mol Cell       Date:  2020-08-20       Impact factor: 17.970

2.  Inhibition of CD38 with the Thiazoloquin(az)olin(on)e 78c Protects the Heart against Postischemic Injury.

Authors:  James Boslett; Nikhil Reddy; Yasmin A Alzarie; Jay L Zweier
Journal:  J Pharmacol Exp Ther       Date:  2019-01-11       Impact factor: 4.030

3.  Sirt1 acts in association with PPARα to protect the heart from hypertrophy, metabolic dysregulation, and inflammation.

Authors:  Ana Planavila; Roser Iglesias; Marta Giralt; Francesc Villarroya
Journal:  Cardiovasc Res       Date:  2010-11-29       Impact factor: 10.787

4.  SIRT3 regulates mitochondrial fatty-acid oxidation by reversible enzyme deacetylation.

Authors:  Matthew D Hirschey; Tadahiro Shimazu; Eric Goetzman; Enxuan Jing; Bjoern Schwer; David B Lombard; Carrie A Grueter; Charles Harris; Sudha Biddinger; Olga R Ilkayeva; Robert D Stevens; Yu Li; Asish K Saha; Neil B Ruderman; James R Bain; Christopher B Newgard; Robert V Farese; Frederick W Alt; C Ronald Kahn; Eric Verdin
Journal:  Nature       Date:  2010-03-04       Impact factor: 49.962

5.  Sirt3 blocks the cardiac hypertrophic response by augmenting Foxo3a-dependent antioxidant defense mechanisms in mice.

Authors:  Nagalingam R Sundaresan; Madhu Gupta; Gene Kim; Senthilkumar B Rajamohan; Ayman Isbatan; Mahesh P Gupta
Journal:  J Clin Invest       Date:  2009-08-03       Impact factor: 14.808

6.  Mitochondrial complex I deficiency increases protein acetylation and accelerates heart failure.

Authors:  Georgios Karamanlidis; Chi Fung Lee; Lorena Garcia-Menendez; Stephen C Kolwicz; Wichit Suthammarak; Guohua Gong; Margaret M Sedensky; Philip G Morgan; Wang Wang; Rong Tian
Journal:  Cell Metab       Date:  2013-08-06       Impact factor: 27.287

7.  CD38 deficiency suppresses adipogenesis and lipogenesis in adipose tissues through activating Sirt1/PPARγ signaling pathway.

Authors:  Ling-Fang Wang; Lian-Jie Miao; Xiao-Nv Wang; Cong-Cong Huang; Yi-Song Qian; Xuan Huang; Xiao-Lei Wang; Wan-Zhu Jin; Guang-Ju Ji; Mingui Fu; Ke-Yu Deng; Hong-Bo Xin
Journal:  J Cell Mol Med       Date:  2017-08-16       Impact factor: 5.310

8.  DNA damage-induced PARP1 activation confers cardiomyocyte dysfunction through NAD+ depletion in experimental atrial fibrillation.

Authors:  Deli Zhang; Xu Hu; Jin Li; Jia Liu; Luciënne Baks-Te Bulte; Marit Wiersma; Noor-Ul-Ann Malik; Denise M S van Marion; Marziyeh Tolouee; Femke Hoogstra-Berends; Eva A H Lanters; Arie M van Roon; Antoine A F de Vries; Daniël A Pijnappels; Natasja M S de Groot; Robert H Henning; Bianca J J M Brundel
Journal:  Nat Commun       Date:  2019-03-21       Impact factor: 14.919

9.  Nicotinamide mononucleotide (NMN) supplementation ameliorates the impact of maternal obesity in mice: comparison with exercise.

Authors:  Golam Mezbah Uddin; Neil A Youngson; Bronte M Doyle; David A Sinclair; Margaret J Morris
Journal:  Sci Rep       Date:  2017-11-08       Impact factor: 4.379

10.  Sirtuin 3 is essential for hypertension-induced cardiac fibrosis via mediating pericyte transition.

Authors:  Han Su; Heng Zeng; Bo Liu; Jian-Xiong Chen
Journal:  J Cell Mol Med       Date:  2020-05-28       Impact factor: 5.310

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  1 in total

1.  Sexually dimorphic effects of SARM1 deletion on cardiac NAD+ metabolism and function.

Authors:  Hina Lateef Nizami; Keaton E Minor; Ying Ann Chiao; Christine M Light; Chi Fung Lee
Journal:  Am J Physiol Heart Circ Physiol       Date:  2022-09-02       Impact factor: 5.125

  1 in total

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