Literature DB >> 34999880

Trans-ethnic Mendelian-randomization study reveals causal relationships between cardiometabolic factors and chronic kidney disease.

Jie Zheng1, Yuemiao Zhang2, Humaira Rasheed1,3, Venexia Walker1,4, Yuka Sugawara5, Jiachen Li6, Yue Leng7, Benjamin Elsworth1, Robyn E Wootton1, Si Fang1, Qian Yang1, Stephen Burgess8,9, Philip C Haycock1, Maria Carolina Borges1, Yoonsu Cho1, Rebecca Carnegie1, Amy Howell1, Jamie Robinson1, Laurent F Thomas3,10, Ben Michael Brumpton1,3,11, Kristian Hveem3, Stein Hallan10,12, Nora Franceschini13, Andrew P Morris14, Anna Köttgen15, Cristian Pattaro16, Matthias Wuttke15, Masayuki Yamamoto17, Naoki Kashihara18, Masato Akiyama19,20, Masahiro Kanai21,22, Koichi Matsuda23, Yoichiro Kamatani19,24, Yukinori Okada22,25,26, Robin Walters27, Iona Y Millwood27, Zhengming Chen27, George Davey Smith1,28, Sean Barbour29,30, Canqing Yu6, Bjørn Olav Åsvold3,31, Hong Zhang2, Tom R Gaunt1,28.   

Abstract

BACKGROUND: This study was to systematically test whether previously reported risk factors for chronic kidney disease (CKD) are causally related to CKD in European and East Asian ancestries using Mendelian randomization.
METHODS: A total of 45 risk factors with genetic data in European ancestry and 17 risk factors in East Asian participants were identified as exposures from PubMed. We defined the CKD by clinical diagnosis or by estimated glomerular filtration rate of <60 ml/min/1.73 m2. Ultimately, 51 672 CKD cases and 958 102 controls of European ancestry from CKDGen, UK Biobank and HUNT, and 13 093 CKD cases and 238 118 controls of East Asian ancestry from Biobank Japan, China Kadoorie Biobank and Japan-Kidney-Biobank/ToMMo were included.
RESULTS: Eight risk factors showed reliable evidence of causal effects on CKD in Europeans, including genetically predicted body mass index (BMI), hypertension, systolic blood pressure, high-density lipoprotein cholesterol, apolipoprotein A-I, lipoprotein(a), type 2 diabetes (T2D) and nephrolithiasis. In East Asians, BMI, T2D and nephrolithiasis showed evidence of causality on CKD. In two independent replication analyses, we observed that increased hypertension risk showed reliable evidence of a causal effect on increasing CKD risk in Europeans but in contrast showed a null effect in East Asians. Although liability to T2D showed consistent effects on CKD, the effects of glycaemic phenotypes on CKD were weak. Non-linear Mendelian randomization indicated a threshold relationship between genetically predicted BMI and CKD, with increased risk at BMI of >25 kg/m2.
CONCLUSIONS: Eight cardiometabolic risk factors showed causal effects on CKD in Europeans and three of them showed causality in East Asians, providing insights into the design of future interventions to reduce the burden of CKD.
© The Author(s) 2021. Published by Oxford University Press on behalf of the International Epidemiological Association.

Entities:  

Keywords:  Mendelian randomization; cardiometabolic risk factors; causality; chronic kidney disease; trans-ethnic study

Mesh:

Year:  2021        PMID: 34999880      PMCID: PMC8743120          DOI: 10.1093/ije/dyab203

Source DB:  PubMed          Journal:  Int J Epidemiol        ISSN: 0300-5771            Impact factor:   7.196


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