Literature DB >> 34415468

Identification of Cellular Voids in the Human Otic Capsule.

Lars Juul Hansen1, Sune Land Bloch2, Mads Sølvsten Sørensen2.   

Abstract

The otic capsule consists of dense highly mineralized compact bone. Inner ear osteoprotegerin (OPG) effectively inhibits perilabyrinthine remodeling and otic capsular bone turnover is very low compared to other bone. Consequently, degenerative changes like dead osteocytes and microcracks accumulate around the inner ear. Osteocytes are connected via canaliculi and need a certain connectivity to sustain life. Consequently, stochastic osteocyte apoptosis may disrupt the osteocytic network in unsustainable patterns leading to widespread cell death. When studying bulk-stained undecalcified human temporal bone, large clusters of dead osteocytes have been observed. Such "cellular voids" may disrupt the perilabyrinthine OPG mediated remodeling inhibition possibly leading to local remodeling. In the common ear disease otosclerosis pathological bone remodeling foci are found exclusively in the otic capsule. We believe the pathogenesis of otosclerosis is linked to the unique bony dynamics of perilabyrinthine bone and cellular voids may represent a starting point for otosclerotic remodeling. This study aims to identify and characterize cellular voids of the human otic capsule. This would allow future cellular void quantification and comparison of void and otosclerotic distribution to further elucidate the yet unknown pathogenesis of otosclerosis.
© 2021. Association for Research in Otolaryngology.

Entities:  

Mesh:

Year:  2021        PMID: 34415468      PMCID: PMC8476705          DOI: 10.1007/s10162-021-00810-6

Source DB:  PubMed          Journal:  J Assoc Res Otolaryngol        ISSN: 1438-7573


  38 in total

1.  Demonstration of intravital microfissures in undecalcified plastic-embedded temporal bones with the prestaining technique.

Authors:  T Frisch; M S Sørensen; P Bretlau
Journal:  Ann Otol Rhinol Laryngol       Date:  2001-08       Impact factor: 1.547

2.  Intravital microlesions in the human otic capsule. Detection, classification and pathogenetic significance revisited.

Authors:  Thomas Frisch; Poul Bretlau; Mads Solvsten Sorensen
Journal:  ORL J Otorhinolaryngol Relat Spec       Date:  2008-04-08       Impact factor: 1.538

Review 3.  The cell biology of bone metabolism.

Authors:  H K Datta; W F Ng; J A Walker; S P Tuck; S S Varanasi
Journal:  J Clin Pathol       Date:  2008-05       Impact factor: 3.411

4.  Prevalence, size and distribution of microdamage in the human otic capsule.

Authors:  Thomas Frisch; Sune Land Bloch; Mads Sølvsten Sørensen
Journal:  Acta Otolaryngol       Date:  2015-04-10       Impact factor: 1.494

5.  Autofluorescence of bone tissues.

Authors:  A I Prentice
Journal:  J Clin Pathol       Date:  1967-09       Impact factor: 3.411

6.  Unbiased estimation of particle density in the tandem scanning reflected light microscope.

Authors:  V Howard; S Reid; A Baddeley; A Boyde
Journal:  J Microsc       Date:  1985-05       Impact factor: 1.758

Review 7.  Osteocyte apoptosis.

Authors:  Robert L Jilka; Brendon Noble; Robert S Weinstein
Journal:  Bone       Date:  2012-12-11       Impact factor: 4.398

8.  Decline in osteocyte lacunar density in human cortical bone is associated with accumulation of microcracks with age.

Authors:  D Vashishth; O Verborgt; G Divine; M B Schaffler; D P Fyhrie
Journal:  Bone       Date:  2000-04       Impact factor: 4.398

9.  Microcrack surface density in the human otic capsule: An unbiased stereological quantification.

Authors:  Lars Juul Hansen; Sune Land Bloch; Thomas Frisch; Mads Sølvsten Sørensen
Journal:  Anat Rec (Hoboken)       Date:  2020-10-19       Impact factor: 2.064

10.  Degeneration of the osteocyte network in the C57BL/6 mouse model of aging.

Authors:  LeAnn M Tiede-Lewis; Yixia Xie; Molly A Hulbert; Richard Campos; Mark R Dallas; Vladimir Dusevich; Lynda F Bonewald; Sarah L Dallas
Journal:  Aging (Albany NY)       Date:  2017-10-26       Impact factor: 5.682

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