Literature DB >> 34284251

Dichotic listening deficits in amblyaudia are characterized by aberrant neural oscillations in auditory cortex.

Sara Momtaz1, Deborah Moncrieff2, Gavin M Bidelman3.   

Abstract

OBJECTIVE: Children diagnosed with auditory processing disorder (APD) show deficits in processing complex sounds that are associated with difficulties in higher-order language, learning, cognitive, and communicative functions. Amblyaudia (AMB) is a subcategory of APD characterized by abnormally large ear asymmetries in dichotic listening tasks.
METHODS: Here, we examined frequency-specific neural oscillations and functional connectivity via high-density electroencephalography (EEG) in children with and without AMB during passive listening of nonspeech stimuli.
RESULTS: Time-frequency maps of these "brain rhythms" revealed stronger phase-locked beta-gamma (~35 Hz) oscillations in AMB participants within bilateral auditory cortex for sounds presented to the right ear, suggesting a hypersynchronization and imbalance of auditory neural activity. Brain-behavior correlations revealed neural asymmetries in cortical responses predicted the larger than normal right-ear advantage seen in participants with AMB. Additionally, we found weaker functional connectivity in the AMB group from right to left auditory cortex, despite their stronger neural responses overall.
CONCLUSION: Our results reveal abnormally large auditory sensory encoding and an imbalance in communication between cerebral hemispheres (ipsi- to -contralateral signaling) in AMB. SIGNIFICANCE: These neurophysiological changes might lead to the functionally poorer behavioral capacity to integrate information between the two ears in children with AMB. Published by Elsevier B.V.

Entities:  

Keywords:  Auditory processing disorders (APD); Event-related brain potentials (ERPs); Gamma/beta band response; Hemispheric asymmetries; Phase-locking; Time-frequency analysis

Mesh:

Year:  2021        PMID: 34284251      PMCID: PMC8384684          DOI: 10.1016/j.clinph.2021.04.022

Source DB:  PubMed          Journal:  Clin Neurophysiol        ISSN: 1388-2457            Impact factor:   4.861


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