Literature DB >> 33743851

Olaparib tablets as maintenance therapy in patients with platinum-sensitive relapsed ovarian cancer and a BRCA1/2 mutation (SOLO2/ENGOT-Ov21): a final analysis of a double-blind, randomised, placebo-controlled, phase 3 trial.

Andrés Poveda1, Anne Floquet2, Jonathan A Ledermann3, Rebecca Asher4, Richard T Penson5, Amit M Oza6, Jacob Korach7, Tomasz Huzarski8, Sandro Pignata9, Michael Friedlander10, Alessandra Baldoni11, Tjoung-Won Park-Simon12, Kenji Tamura13, Gabe S Sonke14, Alla Lisyanskaya15, Jae-Hoon Kim16, Elias Abdo Filho17, Tsveta Milenkova18, Elizabeth S Lowe19, Phil Rowe18, Ignace Vergote20, Eric Pujade-Lauraine21.   

Abstract

BACKGROUND: Olaparib, a poly (ADP-ribose) polymerase (PARP) inhibitor, has previously been shown to extend progression-free survival versus placebo when given to patients with relapsed high-grade serous or endometrioid ovarian cancer who were platinum sensitive and who had a BRCA1 or BRCA2 (BRCA1/2) mutation, as part of the SOLO2/ENGOT-Ov21 trial. The aim of this final analysis is to investigate the effect of olaparib on overall survival.
METHODS: This double-blind, randomised, placebo-controlled, phase 3 trial was done across 123 medical centres in 16 countries. Eligible patients were aged 18 years or older, had an Eastern Cooperative Oncology Group performance status at baseline of 0-1, had histologically confirmed, relapsed, high-grade serous or high-grade endometrioid ovarian cancer, including primary peritoneal or fallopian tube cancer, and had received two or more previous platinum regimens. Patients were randomly assigned (2:1) to receive olaparib tablets (300 mg in two 150 mg tablets twice daily) or matching placebo tablets using an interactive web or voice-response system. Stratification was by response to previous chemotherapy and length of platinum-free interval. Treatment assignment was masked to patients, treatment providers, and data assessors. The primary endpoint of progression-free survival has been reported previously. Overall survival was a key secondary endpoint and was analysed in all patients as randomly allocated. Safety was assessed in all patients who received at least one treatment dose. This trial is registered with ClinicalTrials.gov, NCT01874353, and is no longer recruiting patients.
FINDINGS: Between Sept 3, 2013 and Nov 21, 2014, 295 patients were enrolled. Patients were randomly assigned to receive either olaparib (n=196 [66%]) or placebo (n=99 [34%]). One patient, randomised in error, did not receive olaparib. Median follow-up was 65·7 months (IQR 63·6-69·3) with olaparib and 64·5 months (63·4-68·7) with placebo. Median overall survival was 51·7 months (95% CI 41·5-59·1) with olaparib and 38·8 months (31·4-48·6) with placebo (hazard ratio 0·74 [95% CI 0·54-1·00]; p=0·054), unadjusted for the 38% of patients in the placebo group who received subsequent PARP inhibitor therapy. The most common grade 3 or worse treatment-emergent adverse event was anaemia (which occurred in 41 [21%] of 195 patients in the olaparib group and two [2%] of 99 patients in the placebo group). Serious treatment-emergent adverse events were reported in 50 (26%) of 195 patients receiving olaparib and eight (8%) of 99 patients receiving placebo. Treatment-emergent adverse events with a fatal outcome occurred in eight (4%) of the 195 patients receiving olaparib, six of which were judged to be treatment-related (attributed to myelodysplastic syndrome [n=3] and acute myeloid leukaemia [n=3]).
INTERPRETATION: Olaparib provided a median overall survival benefit of 12·9 months compared with placebo in patients with platinum-sensitive, relapsed ovarian cancer and a BRCA1/2 mutation. Although statistical significance was not reached, these findings are arguably clinically meaningful and support the use of maintenance olaparib in these patients. FUNDING: AstraZeneca and Merck.
Copyright © 2021 Elsevier Ltd. All rights reserved.

Entities:  

Year:  2021        PMID: 33743851     DOI: 10.1016/S1470-2045(21)00073-5

Source DB:  PubMed          Journal:  Lancet Oncol        ISSN: 1470-2045            Impact factor:   41.316


  44 in total

1.  Clonal Hematopoiesis-Associated Gene Mutations in a Clinical Cohort of 448 Patients With Ovarian Cancer.

Authors:  Konstantin Weber-Lassalle; Corinna Ernst; Alexander Reuss; Kathrin Möllenhoff; Klaus Baumann; Christian Jackisch; Jan Hauke; Dimo Dietrich; Julika Borde; Tjoung-Won Park-Simon; Lars Hanker; Katharina Prieske; Sandra Schmidt; Nana Weber-Lassalle; Esther Pohl-Rescigno; Stefan Kommoss; Frederik Marmé; Florian Heitz; Julia C Stingl; Rita K Schmutzler; Philipp Harter; Eric Hahnen
Journal:  J Natl Cancer Inst       Date:  2022-04-11       Impact factor: 13.506

Review 2.  Implementation of preventive and predictive BRCA testing in patients with breast, ovarian, pancreatic, and prostate cancer: a position paper of Italian Scientific Societies.

Authors:  A Russo; L Incorvaia; E Capoluongo; P Tagliaferri; S Gori; L Cortesi; M Genuardi; D Turchetti; U De Giorgi; M Di Maio; M Barberis; M Dessena; M Del Re; A Lapini; C Luchini; B A Jereczek-Fossa; A Sapino; S Cinieri
Journal:  ESMO Open       Date:  2022-05-19

3.  Perspectives on Ovarian Cancer 1809 to 2022 and Beyond.

Authors:  Frank G Lawton; Edward J Pavlik
Journal:  Diagnostics (Basel)       Date:  2022-03-24

Review 4.  Poly(ADP-ribose) polymerase (PARP) inhibitors for the treatment of ovarian cancer.

Authors:  Abigail Tattersall; Neil Ryan; Alison J Wiggans; Ewelina Rogozińska; Jo Morrison
Journal:  Cochrane Database Syst Rev       Date:  2022-02-16

5.  Targeting the ALK-CDK9-Tyr19 kinase cascade sensitizes ovarian and breast tumors to PARP inhibition via destabilization of the P-TEFb complex.

Authors:  Yu-Yi Chu; Mei-Kuang Chen; Yongkun Wei; Heng-Huan Lee; Weiya Xia; Ying-Nai Wang; Clinton Yam; Jennifer L Hsu; Hung-Ling Wang; Wei-Chao Chang; Hirohito Yamaguchi; Zhou Jiang; Chunxiao Liu; Ching-Fei Li; Lei Nie; Li-Chuan Chan; Yuan Gao; Shao-Chun Wang; Jinsong Liu; Shannon N Westin; Sanghoon Lee; Anil K Sood; Liuqing Yang; Gabriel N Hortobagyi; Dihua Yu; Mien-Chie Hung
Journal:  Nat Cancer       Date:  2022-10-17

6.  Olaparib and rucaparib for the treatment of DNA repair-deficient metastatic castration-resistant prostate cancer.

Authors:  Benjamin L Maughan; Emmanuel S Antonarakis
Journal:  Expert Opin Pharmacother       Date:  2021-04-07       Impact factor: 4.103

7.  Editorial: The Role of DNA Repair Pathways in Resistance to Chemotherapy and Radiotherapy in Cancer.

Authors:  David Y Lee; Rosa María Bermúdez-Cruz; José Díaz-Chávez
Journal:  Front Oncol       Date:  2022-04-08       Impact factor: 5.738

Review 8.  Differences in PARP Inhibitors for the Treatment of Ovarian Cancer: Mechanisms of Action, Pharmacology, Safety, and Efficacy.

Authors:  Giorgio Valabrega; Giulia Scotto; Valentina Tuninetti; Arianna Pani; Francesco Scaglione
Journal:  Int J Mol Sci       Date:  2021-04-19       Impact factor: 5.923

Review 9.  Extracellular vesicles in ovarian cancer chemoresistance, metastasis, and immune evasion.

Authors:  Wanjia Tian; Ningjing Lei; Junying Zhou; Mengyu Chen; Ruixia Guo; Bo Qin; Yong Li; Lei Chang
Journal:  Cell Death Dis       Date:  2022-01-18       Impact factor: 8.469

Review 10.  PARP Inhibitors and Myeloid Neoplasms: A Double-Edged Sword.

Authors:  Clifford M Csizmar; Antoine N Saliba; Elizabeth M Swisher; Scott H Kaufmann
Journal:  Cancers (Basel)       Date:  2021-12-20       Impact factor: 6.639

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