Literature DB >> 33461618

Topographic patterns of white matter hyperintensities are associated with multimodal neuroimaging biomarkers of Alzheimer's disease.

Malo Gaubert1,2, Catharina Lange3,4, Gaël Chételat5, Miranka Wirth6, Antoine Garnier-Crussard7,8, Theresa Köbe1, Salma Bougacha7, Julie Gonneaud7, Robin de Flores7, Clémence Tomadesso7, Florence Mézenge7, Brigitte Landeau7, Vincent de la Sayette9.   

Abstract

BACKGROUND: White matter hyperintensities (WMH) are frequently found in Alzheimer's disease (AD). Commonly considered as a marker of cerebrovascular disease, regional WMH may be related to pathological hallmarks of AD, including beta-amyloid (Aβ) plaques and neurodegeneration. The aim of this study was to examine the regional distribution of WMH associated with Aβ burden, glucose hypometabolism, and gray matter volume reduction.
METHODS: In a total of 155 participants (IMAP+ cohort) across the cognitive continuum from normal cognition to AD dementia, FLAIR MRI, AV45-PET, FDG-PET, and T1 MRI were acquired. WMH were automatically segmented from FLAIR images. Mean levels of neocortical Aβ deposition (AV45-PET), temporo-parietal glucose metabolism (FDG-PET), and medial-temporal gray matter volume (GMV) were extracted from processed images using established AD meta-signature templates. Associations between AD brain biomarkers and WMH, as assessed in region-of-interest and voxel-wise, were examined, adjusting for age, sex, education, and systolic blood pressure.
RESULTS: There were no significant associations between global Aβ burden and region-specific WMH. Voxel-wise WMH in the splenium of the corpus callosum correlated with greater Aβ deposition at a more liberal threshold. Region- and voxel-based WMH in the posterior corpus callosum, along with parietal, occipital, and frontal areas, were associated with lower temporo-parietal glucose metabolism. Similarly, lower medial-temporal GMV correlated with WMH in the posterior corpus callosum in addition to parietal, occipital, and fontal areas.
CONCLUSIONS: This study demonstrates that local white matter damage is correlated with multimodal brain biomarkers of AD. Our results highlight modality-specific topographic patterns of WMH, which converged in the posterior white matter. Overall, these cross-sectional findings corroborate associations of regional WMH with AD-typical Aß deposition and neurodegeneration.

Entities:  

Keywords:  Alzheimer’s disease; Alzheimer’s disease pathology; Cerebrovascular disease; White matter lesion

Mesh:

Substances:

Year:  2021        PMID: 33461618      PMCID: PMC7814451          DOI: 10.1186/s13195-020-00759-3

Source DB:  PubMed          Journal:  Alzheimers Res Ther            Impact factor:   6.982


  51 in total

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