Literature DB >> 33351088

Association of Optical Coherence Tomography With Longitudinal Neurodegeneration in Veterans With Chronic Mild Traumatic Brain Injury.

Casey S Gilmore1,2, Kelvin O Lim1,2,3, Mona K Garvin4,5, Jui-Kai Wang4,5, Johannes Ledolter5,6, Alicia L Fenske1,2, Carolyn L Gentz1,2, Julie Nellis4,7, Michael T Armstrong1,2, Randy H Kardon4,7.   

Abstract

Importance: Mild traumatic brain injury (TBI) may predispose individuals to progressive neurodegeneration. Objective: To identify evidence of neurodegeneration through longitudinal evaluation of changes in retinal layer thickness using optical coherence tomography in veterans with a history of mild TBI. Design, Setting, and Participants: This longitudinal cohort study evaluated veterans who were receiving services at the Minneapolis Veterans Affairs Health Care System. Symptomatic or mild TBI was diagnosed according to the Mayo TBI Severity Classification System. Participants in the age-matched control group had no history of TBI. Participants with any history or evidence of retinal or optic nerve disease that could affect retinal thickness were excluded. Data analysis was performed from July 2019 to February 2020. Exposures: The presence and severity of mild TBI were determined through consensus review of self-report responses during the Minnesota Blast Exposure Screening Tool semistructured interview. Main Outcomes and Measures: Change over time of retinal nerve fiber layer (RNFL) thickness.
Results: A total of 139 veterans (117 men [84%]; mean [SD] age, 49.9 [11.1] years) were included in the study, 69 in the TBI group and 70 in the control group. Veterans with mild TBI showed significantly greater RNFL thinning compared with controls (mean [SE] RNFL slope, -1.47 [0.24] μm/y vs -0.31 [0.32] μm/y; F1,122 = 8.42; P = .004; Cohen d = 0.52). Functionally, veterans with mild TBI showed greater declines in visual field mean deviation (mean [SE] slope, -0.09 [0.14] dB/y vs 0.46 [0.23] dB/y; F1,122 = 4.08; P = .046; Cohen d = 0.36) and pattern standard deviation (mean [SE] slope, 0.09 [0.06] dB/y vs -0.10 [0.07] dB/y; F1,122 = 4.78; P = .03; Cohen d = 0.39) and high spatial frequency (12 cycles/degree) contrast sensitivity compared with controls. Cognitively, there was a significantly greater decrease in the number of errors over time during the Groton Maze Learning Test (GMLT) in controls compared with veterans with mild TBI (mean [SE] slope, -9.30 [1.48] errors/y vs -5.23 [1.24] errors/y; F1,127 = 4.43; P = .04; Cohen d = 0.37). RNFL tissue loss was significantly correlated with both worsening performance on the GMLT over time (Spearman ρ = -0.20; P = .03) and mild TBI severity (Spearman ρ = -0.25; P = .006). The more severe the mild TBI (larger Minnesota Blast Exposure Screening Tool severity score), the faster the reduction in RNFL thickness (ie, the more negative the slope) across time. Conclusions and Relevance: This cohort study found longitudinal evidence for significant, progressive neural degeneration over time in veterans with mild TBI, as indicated by greater RNFL tissue loss in patients with mild TBI vs controls, as well as measures of function. These results suggest that these longitudinal measures may be useful biomarkers of neurodegeneration. Changes in this biomarker may provide early detection of subsequent cognitive and functional deficits that may impact veterans' independence and need for care.

Entities:  

Mesh:

Year:  2020        PMID: 33351088      PMCID: PMC7756235          DOI: 10.1001/jamanetworkopen.2020.30824

Source DB:  PubMed          Journal:  JAMA Netw Open        ISSN: 2574-3805


  51 in total

Review 1.  Traumatic brain injury: a disease process, not an event.

Authors:  Brent E Masel; Douglas S DeWitt
Journal:  J Neurotrauma       Date:  2010-08       Impact factor: 5.269

2.  Indirect Traumatic Optic Neuropathy in Mild Chronic Traumatic Brain Injury.

Authors:  Jane W Chan; Nancy K Hills; Benjamin Bakall; Brian Fernandez
Journal:  Invest Ophthalmol Vis Sci       Date:  2019-05-01       Impact factor: 4.799

3.  Association of Retinal Neurodegeneration on Optical Coherence Tomography With Dementia: A Population-Based Study.

Authors:  Unal Mutlu; Johanna M Colijn; M Arfan Ikram; Pieter W M Bonnemaijer; Silvan Licher; Frank J Wolters; Henning Tiemeier; Peter J Koudstaal; Caroline C W Klaver; M Kamran Ikram
Journal:  JAMA Neurol       Date:  2018-10-01       Impact factor: 18.302

Review 4.  Traumatic brain injury: a risk factor for neurodegenerative diseases.

Authors:  Rajaneesh Gupta; Nilkantha Sen
Journal:  Rev Neurosci       Date:  2016-01       Impact factor: 4.353

5.  Retinal thinning associates with nigral dopaminergic loss in de novo Parkinson disease.

Authors:  Jeeyun Ahn; Jee-Young Lee; Tae Wan Kim; Eun Jin Yoon; Sohee Oh; Yu Kyeong Kim; Jong-Min Kim; Se Joon Woo; Ki Woong Kim; Beomseok Jeon
Journal:  Neurology       Date:  2018-08-15       Impact factor: 9.910

Review 6.  Biomarker modeling of Alzheimer's disease.

Authors:  Clifford R Jack; David M Holtzman
Journal:  Neuron       Date:  2013-12-18       Impact factor: 17.173

7.  The mayo classification system for traumatic brain injury severity.

Authors:  James F Malec; Allen W Brown; Cynthia L Leibson; Julie Testa Flaada; Jayawant N Mandrekar; Nancy N Diehl; Patricia K Perkins
Journal:  J Neurotrauma       Date:  2007-09       Impact factor: 5.269

8.  Retinal thickness in Alzheimer's disease: A systematic review and meta-analysis.

Authors:  Jurre den Haan; Frank D Verbraak; Pieter Jelle Visser; Femke H Bouwman
Journal:  Alzheimers Dement (Amst)       Date:  2017-01-25

9.  Change in retinal structural anatomy during the preclinical stage of Alzheimer's disease.

Authors:  Cláudia Y Santos; Lenworth N Johnson; Stuart E Sinoff; Elena K Festa; William C Heindel; Peter J Snyder
Journal:  Alzheimers Dement (Amst)       Date:  2018-02-07

10.  Blast-Mediated Traumatic Brain Injury Exacerbates Retinal Damage and Amyloidosis in the APPswePSENd19e Mouse Model of Alzheimer's Disease.

Authors:  Matthew M Harper; Adam Hedberg-Buenz; Judith Herlein; Eric E Abrahamson; Michael G Anderson; Markus H Kuehn; Randy H Kardon; Pieter Poolman; Milos D Ikonomovic
Journal:  Invest Ophthalmol Vis Sci       Date:  2019-06-03       Impact factor: 4.799

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2.  Tandem Mass Tag-based proteomics analysis reveals the vital role of inflammation in traumatic brain injury in a mouse model.

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3.  Network topology changes in chronic mild traumatic brain injury (mTBI).

Authors:  Elias Boroda; Michael Armstrong; Casey S Gilmore; Carrie Gentz; Alicia Fenske; Mark Fiecas; Tim Hendrickson; Donovan Roediger; Bryon Mueller; Randy Kardon; Kelvin Lim
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4.  Report From the National Eye Institute Workshop on Neuro-Ophthalmic Disease Clinical Trial Endpoints: Optic Neuropathies.

Authors:  Leonard A Levin; Mohor Sengupta; Laura J Balcer; Mark J Kupersmith; Neil R Miller
Journal:  Invest Ophthalmol Vis Sci       Date:  2021-11-01       Impact factor: 4.799

5.  Structure and function of retinal ganglion cells in subjects with a history of repeated traumatic brain injury.

Authors:  Kelly R Klimo; Elizabeth A Stern-Green; Erica Shelton; Elizabeth Day; Lisa Jordan; Matthew Robich; Julie Racine; Catherine E McDaniel; Dean A VanNasdale; Phillip T Yuhas
Journal:  Front Neurol       Date:  2022-08-12       Impact factor: 4.086

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