Literature DB >> 33208540

PM2.5 associated with gray matter atrophy reflecting increased Alzheimers risk in older women.

Diana Younan1, Xinhui Wang2, Ramon Casanova2, Ryan Barnard2, Sarah A Gaussoin2, Santiago Saldana2, Andrew J Petkus2, Daniel P Beavers2, Susan M Resnick2, JoAnn E Manson2, Marc L Serre2, William Vizuete2, Victor W Henderson2, Bonnie C Sachs2, Joel A Salinas2, Margaret Gatz2, Mark A Espeland2, Helena C Chui2, Sally A Shumaker2, Stephen R Rapp2, Jiu-Chiuan Chen2.   

Abstract

OBJECTIVE: To examine whether late-life exposure to PM2.5 (particulate matter with aerodynamic diameters <2.5-µm) contributes to progressive brain atrophy predictive of Alzheimer's disease (AD) using a community-dwelling cohort of women (aged 70-89) with up to two brain MRI scans (MRI-1: 2005-6; MRI-2: 2010-11).
METHODS: AD pattern similarity (AD-PS) scores, developed by supervised machine learning and validated with MRI data from the AD Neuroimaging Initiative, was used to capture high-dimensional gray matter atrophy in brain areas vulnerable to AD (e.g., amygdala, hippocampus, parahippocampal gyrus, thalamus, inferior temporal lobe areas and midbrain). Based on participants' addresses and air monitoring data, we implemented a spatiotemporal model to estimate 3-year average exposure to PM2.5 preceding MRI-1. General linear models were used to examine the association between PM2.5 and AD-PS scores (baseline and 5-year standardized change), accounting for potential confounders and white matter lesion volumes.
RESULTS: For 1365 women aged 77.9±3.7 years in 2005-6, there was no association between PM2.5 and baseline AD-PS score in cross-sectional analyses (β=-0.004; 95% CI: -0.019, 0.011). Longitudinally, each interquartile range increase of PM2.5 (2.82-µg/m3) was associated with increased AD-PS scores during the follow-up, equivalent to a 24% (hazard ratio=1.24; 95% CI: 1.14, 1.34) increase in AD risk over 5-years (n=712; aged 77.4±3.5 years). This association remained after adjustment for socio-demographics, intracranial volume, lifestyle, clinical characteristics, and white matter lesions, and was present with levels below US regulatory standards (<12-µg/m3).
CONCLUSIONS: Late-life exposure to PM2.5 is associated with increased neuroanatomical risk of AD, which may not be explained by available indicators of cerebrovascular damage.
© 2020 American Academy of Neurology.

Entities:  

Year:  2020        PMID: 33208540      PMCID: PMC8055348          DOI: 10.1212/WNL.0000000000011149

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  51 in total

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