Literature DB >> 33178868

Examining the Frequency of the JAK2 (V617F) Mutation in Patients with Myeloproliferative Diseases in North Eastern Iran and the Effect of Treatment Intervention.

Mojila Nasseri1,2, Fatemeh Keyfi3,2, Raheleh Rahbarian1, Majid Rajabian4, Mohammad Reza Abbaszadegan2,5.   

Abstract

BACKGROUND: Janus kinase 2 (JAK2) is a tyrosine kinase located in the cytoplasm that plays a critical role in the signal transduction of cytokines and growth hormones. The conversion of valine to phenylalanine at the polypeptide position 617 results in the JAK2 (V617F) mutation, which often found in patients with myeloproliferative neoplasms (MPNs). As a result of this mutation, JAK2 is constitutively activated leading to uncontrolled cell growth. The present study aimed to investigate the frequency and relationship of the JAK2 (V617F) mutation in a population of patients with MPNs in Iran.
METHODS: A total of 213 patients with myeloproliferative diseases (MPDs), were included in the study. Real-time PCR was used to detect the presence of the JAK2 (V617F) mutation in the genomic DNA isolated from patient peripheral blood samples.
RESULTS: Of the 213 patients with MPDs, approximately 60 (28%) patients were positive for the JAK2 (V617F) mutation. Polycythemia Vera (PV, 42.11%) was the most common MPD, followed by Essential Thrombocythemia (ET, 29.82%), Primary Myelofibrosis (MF, 12.28%), and Chronic Myeloid Leukemia (CML, 10.5%). A significant relationship between all types of MPDs and the clinical course (p< 0.05) was observed. The relationship between age and gender among all types of MPD disease was not significant (p> 0.05).
CONCLUSION: Of the examined cohort in North Eastern Iran, 28% of the patients with MPNs were found to have the JAK2 (V617F) mutation which determining the presence of the JAK2 (V617F) mutation helps to decide the correct form of treatment.

Entities:  

Keywords:  AK2 (V617F); JAK-STAT pathway; MPD Real-Time PCR; Treatment

Year:  2020        PMID: 33178868      PMCID: PMC7603260          DOI: 10.29252/rbmb.9.2.188

Source DB:  PubMed          Journal:  Rep Biochem Mol Biol        ISSN: 2322-3480


  10 in total

1.  Regulation of the Jak2 tyrosine kinase by its pseudokinase domain.

Authors:  P Saharinen; K Takaluoma; O Silvennoinen
Journal:  Mol Cell Biol       Date:  2000-05       Impact factor: 4.272

Review 2.  Vertebrate non-receptor protein-tyrosine kinase families.

Authors:  K Neet; T Hunter
Journal:  Genes Cells       Date:  1996-02       Impact factor: 1.891

3.  JAK2 V617F Mutation in Adult T Cell Leukemia-Lymphoma.

Authors:  Hossein Ayatollahi; Mohammad Hadi Sadeghian; Mohammad Reza Keramati; Ehsan Ghayoor Karimiani; Amir Hossein Jafarian; Abbas Shirdel; Hossein Rahimi; Mohammad Esmaeel Zangane-Far; Arezoo Shajiei; Maryam Sheikhi
Journal:  Indian J Hematol Blood Transfus       Date:  2015-12-17       Impact factor: 0.900

4.  A family with erythrocytosis establishes a role for prolyl hydroxylase domain protein 2 in oxygen homeostasis.

Authors:  Melanie J Percy; Quan Zhao; Adrian Flores; Claire Harrison; Terence R J Lappin; Patrick H Maxwell; Mary Frances McMullin; Frank S Lee
Journal:  Proc Natl Acad Sci U S A       Date:  2006-01-09       Impact factor: 11.205

5.  Prevalence of the frequency of JAK2 (V617F) mutation in different myeloproliferative disorders in Egyptian patients.

Authors:  Gamal T Ebid; Mohamed Ghareeb; Omina Salaheldin; Mahmoud M Kamel
Journal:  Int J Clin Exp Pathol       Date:  2015-09-01

6.  Proposed criteria for the diagnosis of post-polycythemia vera and post-essential thrombocythemia myelofibrosis: a consensus statement from the International Working Group for Myelofibrosis Research and Treatment.

Authors:  G Barosi; R A Mesa; J Thiele; F Cervantes; P J Campbell; S Verstovsek; B Dupriez; R L Levine; F Passamonti; J Gotlib; J T Reilly; A M Vannucchi; C A Hanson; L A Solberg; A Orazi; A Tefferi
Journal:  Leukemia       Date:  2007-08-30       Impact factor: 11.528

7.  Improved targeting of JAK2 leads to increased therapeutic efficacy in myeloproliferative neoplasms.

Authors:  Neha Bhagwat; Priya Koppikar; Matthew Keller; Sachie Marubayashi; Kaitlyn Shank; Raajit Rampal; Jun Qi; Maria Kleppe; Hardik J Patel; Smit K Shah; Tony Taldone; James E Bradner; Gabriela Chiosis; Ross L Levine
Journal:  Blood       Date:  2014-01-27       Impact factor: 22.113

8.  Role of Ubiquitylation in Controlling Suppressor of Cytokine Signalling 3 (SOCS3) Function and Expression.

Authors:  Jamie J L Williams; Kirsten M A Munro; Timothy M Palmer
Journal:  Cells       Date:  2014-05-30       Impact factor: 6.600

Review 9.  Mechanistic Insights into Regulation of JAK2 Tyrosine Kinase.

Authors:  Stevan R Hubbard
Journal:  Front Endocrinol (Lausanne)       Date:  2018-01-05       Impact factor: 5.555

10.  The JAK2 V617F mutation in isolated neutropenia.

Authors:  Stephen E Langabeer
Journal:  EXCLI J       Date:  2018-01-02       Impact factor: 4.068

  10 in total

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