Literature DB >> 32702169

Randomised clinical trial: significant biochemical and colonic transit effects of the farnesoid X receptor agonist tropifexor in patients with primary bile acid diarrhoea.

Michael Camilleri1, Sara Linker Nord1, Duane Burton1, Ibironke Oduyebo1, Yiming Zhang2, Jin Chen3, Koeun Im2, Prafulla Bhad2, Michael K Badman2, David S Sanders4, Julian R F Walters5.   

Abstract

BACKGROUND: In primary bile acid diarrhoea, feedback by farnesoid X receptor (FXR) and fibroblast growth hormone 19 (FGF19) on hepatic bile acid production is impaired. AIMS: To evaluate the safety, mechanisms and efficacy of negative feedback by FXR activation with tropifexor, a non-bile acid FXR agonist, in patients with primary bile acid diarrhoea.
METHODS: In this double-blind, multicentre, randomised, cross-over study, patients received tropifexor 60 µg or placebo once daily for 14 days in each of two treatment periods. Primary objectives included tropifexor safety and tolerability, and on stool frequency and form. Other assessments included pharmacokinetic and pharmacodynamic measures, biochemical markers and gastrointestinal transit.
RESULTS: Twenty patients (tropifexor 60 µg/placebo [N = 10]; placebo/tropifexor 60 µg [N = 10]) were enrolled. Adverse event rates were lower with tropifexor vs placebo (52.9% vs 73.7%). No patient had pruritus during tropifexor intake. There were no significant differences in stool frequency, stool form or loperamide use between treatments. Tropifexor increased FGF19 and decreased 7α-hydroxy-4-cholesten-3-one (C4) levels for up to 8 h. Plasma tropifexor concentrations peaked at 5 hours post-dose on days 1 and 12. At day 12, tropifexor caused reduction in peak total bile acid concentration (33%, P = 0.032) and exposure (36%, P = 0.005). Moreover, tropifexor showed a significant increase in ascending colon half-emptying time (P = 0.036).
CONCLUSIONS: Tropifexor 60 µg once daily had acceptable safety and tolerability. Changes in FGF19 and C4 showed effective target engagement; however, higher doses may be required to observe stool frequency changes. Slowing of ascending colon emptying suggests therapeutic potential of tropifexor in patients with primary bile acid diarrhoea. ClinicalTrials.gov number: NCT02713243.
© 2020 John Wiley & Sons Ltd.

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Year:  2020        PMID: 32702169     DOI: 10.1111/apt.15967

Source DB:  PubMed          Journal:  Aliment Pharmacol Ther        ISSN: 0269-2813            Impact factor:   8.171


  10 in total

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Journal:  Pharm Res       Date:  2022-03-21       Impact factor: 4.200

2.  Contributions of bile acids to gastrointestinal physiology as receptor agonists and modifiers of ion channels.

Authors:  Stephen J Keely; Andreacarola Urso; Alexandr V Ilyaskin; Christoph Korbmacher; Nigel W Bunnett; Daniel P Poole; Simona E Carbone
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Review 4.  Pathophysiology and Clinical Management of Bile Acid Diarrhea.

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Review 8.  Bile Acid-Related Regulation of Mucosal Inflammation and Intestinal Motility: From Pathogenesis to Therapeutic Application in IBD and Microscopic Colitis.

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Journal:  Nutrients       Date:  2022-06-27       Impact factor: 6.706

9.  Farnesoid X receptor agonist tropifexor attenuates cholestasis in a randomised trial in patients with primary biliary cholangitis.

Authors:  Christoph Schramm; Heiner Wedemeyer; Andrew Mason; Gideon M Hirschfield; Cynthia Levy; Kris V Kowdley; Piotr Milkiewicz; Ewa Janczewska; Elena Sergeevna Malova; Johanne Sanni; Phillip Koo; Jin Chen; Subhajit Choudhury; Lloyd B Klickstein; Michael K Badman; David Jones
Journal:  JHEP Rep       Date:  2022-07-21

Review 10.  Redox-Dependent Effects in the Physiopathological Role of Bile Acids.

Authors:  Josué Orozco-Aguilar; Felipe Simon; Claudio Cabello-Verrugio
Journal:  Oxid Med Cell Longev       Date:  2021-09-04       Impact factor: 6.543

  10 in total

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