Literature DB >> 32372669

Desmoplakin Cardiomyopathy, a Fibrotic and Inflammatory Form of Cardiomyopathy Distinct From Typical Dilated or Arrhythmogenic Right Ventricular Cardiomyopathy.

Eric D Smith1, Neal K Lakdawala2, Nikolaos Papoutsidakis3, Gregory Aubert4, Andrea Mazzanti5, Anthony C McCanta6, Prachi P Agarwal7, Patricia Arscott1, Lisa M Dellefave-Castillo8, Esther E Vorovich9, Kavitha Nutakki2, Lisa D Wilsbacher8, Silvia G Priori5, Daniel L Jacoby3, Elizabeth M McNally8, Adam S Helms1.   

Abstract

BACKGROUND: Mutations in desmoplakin (DSP), the primary force transducer between cardiac desmosomes and intermediate filaments, cause an arrhythmogenic form of cardiomyopathy that has been variably associated with arrhythmogenic right ventricular cardiomyopathy. Clinical correlates of DSP cardiomyopathy have been limited to small case series.
METHODS: Clinical and genetic data were collected on 107 patients with pathogenic DSP mutations and 81 patients with pathogenic plakophilin 2 (PKP2) mutations as a comparison cohort. A composite outcome of severe ventricular arrhythmia was assessed.
RESULTS: DSP and PKP2 cohorts included similar proportions of probands (41% versus 42%) and patients with truncating mutations (98% versus 100%). Left ventricular (LV) predominant cardiomyopathy was exclusively present among patients with DSP (55% versus 0% for PKP2, P<0.001), whereas right ventricular cardiomyopathy was present in only 14% of patients with DSP versus 40% for PKP2 (P<0.001). Arrhythmogenic right ventricular cardiomyopathy diagnostic criteria had poor sensitivity for DSP cardiomyopathy. LV late gadolinium enhancement was present in a primarily subepicardial distribution in 40% of patients with DSP (23/57 with magnetic resonance images). LV late gadolinium enhancement occurred with normal LV systolic function in 35% (8/23) of patients with DSP. Episodes of acute myocardial injury (chest pain with troponin elevation and normal coronary angiography) occurred in 15% of patients with DSP and were strongly associated with LV late gadolinium enhancement (90%), even in cases of acute myocardial injury with normal ventricular function (4/5, 80% with late gadolinium enhancement). In 4 DSP cases with 18F-fluorodeoxyglucose positron emission tomography scans, acute LV myocardial injury was associated with myocardial inflammation misdiagnosed initially as cardiac sarcoidosis or myocarditis. Left ventricle ejection fraction <55% was strongly associated with severe ventricular arrhythmias for DSP cases (P<0.001, sensitivity 85%, specificity 53%). Right ventricular ejection fraction <45% was associated with severe arrhythmias for PKP2 cases (P<0.001) but was poorly associated for DSP cases (P=0.8). Frequent premature ventricular contractions were common among patients with severe arrhythmias for both DSP (80%) and PKP2 (91%) groups (P=non-significant).
CONCLUSIONS: DSP cardiomyopathy is a distinct form of arrhythmogenic cardiomyopathy characterized by episodic myocardial injury, left ventricular fibrosis that precedes systolic dysfunction, and a high incidence of ventricular arrhythmias. A genotype-specific approach for diagnosis and risk stratification should be used.

Entities:  

Keywords:  arrhythmogenic right ventricular cardiomyopathy; cardiomyopathy; desmoplakin; desmosome

Year:  2020        PMID: 32372669      PMCID: PMC7286080          DOI: 10.1161/CIRCULATIONAHA.119.044934

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  47 in total

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Journal:  Circulation       Date:  2019-06-03       Impact factor: 29.690

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6.  Impact of genotype on clinical course in arrhythmogenic right ventricular dysplasia/cardiomyopathy-associated mutation carriers.

Authors:  Aditya Bhonsale; Judith A Groeneweg; Cynthia A James; Dennis Dooijes; Crystal Tichnell; Jan D H Jongbloed; Brittney Murray; Anneline S J M te Riele; Maarten P van den Berg; Hennie Bikker; Douwe E Atsma; Natasja M de Groot; Arjan C Houweling; Jeroen F van der Heijden; Stuart D Russell; Pieter A Doevendans; Toon A van Veen; Harikrishna Tandri; Arthur A Wilde; Daniel P Judge; J Peter van Tintelen; Hugh Calkins; Richard N Hauer
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Authors:  T Maruthappu; A Posafalvi; S Castelletti; P J Delaney; P Syrris; E A O'Toole; K J Green; P M Elliott; P D Lambiase; A Tinker; W J McKenna; D P Kelsell
Journal:  Br J Dermatol       Date:  2019-01-02       Impact factor: 9.302

8.  A pattern-based approach to assessment of delayed enhancement in nonischemic cardiomyopathy at MR imaging.

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Journal:  Radiographics       Date:  2009 Jan-Feb       Impact factor: 5.333

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Authors:  Nadine Kawel-Boehm; Alicia Maceira; Emanuela R Valsangiacomo-Buechel; Jens Vogel-Claussen; Evrim B Turkbey; Rupert Williams; Sven Plein; Michael Tee; John Eng; David A Bluemke
Journal:  J Cardiovasc Magn Reson       Date:  2015-04-18       Impact factor: 5.364

10.  Association of fibrosis with mortality and sudden cardiac death in patients with nonischemic dilated cardiomyopathy.

Authors:  Ankur Gulati; Andrew Jabbour; Tevfik F Ismail; Kaushik Guha; Jahanzaib Khwaja; Sadaf Raza; Kishen Morarji; Tristan D H Brown; Nizar A Ismail; Marc R Dweck; Elisa Di Pietro; Michael Roughton; Ricardo Wage; Yousef Daryani; Rory O'Hanlon; Mary N Sheppard; Francisco Alpendurada; Alexander R Lyon; Stuart A Cook; Martin R Cowie; Ravi G Assomull; Dudley J Pennell; Sanjay K Prasad
Journal:  JAMA       Date:  2013-03-06       Impact factor: 56.272

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2.  Clinical and genetic features of arrhythmogenic cardiomyopathy: diagnosis, management and the heart failure perspective.

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9.  Clinical Findings and Diagnostic Yield of Arrhythmogenic Cardiomyopathy Through Genomic Screening of Pathogenic or Likely Pathogenic Desmosome Gene Variants.

Authors:  Eric D Carruth; Dominik Beer; Amro Alsaid; Marci L B Schwartz; Megan McMinn; Melissa A Kelly; Adam H Buchanan; Christopher D Nevius; Hugh Calkins; Cynthia A James; Brittney Murray; Crystal Tichnell; Martin E Matsumura; H Lester Kirchner; Brandon K Fornwalt; Amy C Sturm; Christopher M Haggerty
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