Literature DB >> 32278790

Characterization of the clinical and immunologic phenotype and management of 157 individuals with 56 distinct heterozygous NFKB1 mutations.

Tiziana Lorenzini1, Manfred Fliegauf2, Nils Klammer3, Natalie Frede3, Michele Proietti3, Alla Bulashevska3, Nadezhda Camacho-Ordonez3, Markku Varjosalo4, Matias Kinnunen4, Esther de Vries5, Jos W M van der Meer6, Rohan Ameratunga7, Chaim M Roifman8, Yael D Schejter8, Robin Kobbe9, Timo Hautala10, Faranaz Atschekzei11, Reinhold E Schmidt11, Claudia Schröder12, Polina Stepensky13, Bella Shadur14, Luis A Pedroza15, Michiel van der Flier16, Mónica Martínez-Gallo17, Luis Ignacio Gonzalez-Granado18, Luis M Allende19, Anna Shcherbina20, Natalia Kuzmenko20, Victoria Zakharova21, João Farela Neves22, Peter Svec23, Ute Fischer24, Winnie Ip25, Oliver Bartsch26, Safa Barış27, Christoph Klein28, Raif Geha29, Janet Chou29, Mohammed Alosaimi29, Lauren Weintraub30, Kaan Boztug31, Tatjana Hirschmugl31, Maria Marluce Dos Santos Vilela32, Dirk Holzinger33, Maximilian Seidl34, Vassilios Lougaris35, Alessandro Plebani35, Laia Alsina36, Monica Piquer-Gibert36, Angela Deyà-Martínez36, Charlotte A Slade37, Asghar Aghamohammadi38, Hassan Abolhassani39, Lennart Hammarström40, Outi Kuismin41, Merja Helminen42, Hana Lango Allen43, James E Thaventhiran44, Alexandra F Freeman45, Matthew Cook46, Shahrzad Bakhtiar47, Mette Christiansen48, Charlotte Cunningham-Rundles49, Niraj C Patel50, William Rae51, Tim Niehues52, Nina Brauer52, Jaana Syrjänen53, Mikko R J Seppänen54, Siobhan O Burns55, Paul Tuijnenburg56, Taco W Kuijpers56, Klaus Warnatz57, Bodo Grimbacher58.   

Abstract

BACKGROUND: An increasing number of NFKB1 variants are being identified in patients with heterogeneous immunologic phenotypes.
OBJECTIVE: To characterize the clinical and cellular phenotype as well as the management of patients with heterozygous NFKB1 mutations.
METHODS: In a worldwide collaborative effort, we evaluated 231 individuals harboring 105 distinct heterozygous NFKB1 variants. To provide evidence for pathogenicity, each variant was assessed in silico; in addition, 32 variants were assessed by functional in vitro testing of nuclear factor of kappa light polypeptide gene enhancer in B cells (NF-κB) signaling.
RESULTS: We classified 56 of the 105 distinct NFKB1 variants in 157 individuals from 68 unrelated families as pathogenic. Incomplete clinical penetrance (70%) and age-dependent severity of NFKB1-related phenotypes were observed. The phenotype included hypogammaglobulinemia (88.9%), reduced switched memory B cells (60.3%), and respiratory (83%) and gastrointestinal (28.6%) infections, thus characterizing the disorder as primary immunodeficiency. However, the high frequency of autoimmunity (57.4%), lymphoproliferation (52.4%), noninfectious enteropathy (23.1%), opportunistic infections (15.7%), autoinflammation (29.6%), and malignancy (16.8%) identified NF-κB1-related disease as an inborn error of immunity with immune dysregulation, rather than a mere primary immunodeficiency. Current treatment includes immunoglobulin replacement and immunosuppressive agents.
CONCLUSIONS: We present a comprehensive clinical overview of the NF-κB1-related phenotype, which includes immunodeficiency, autoimmunity, autoinflammation, and cancer. Because of its multisystem involvement, clinicians from each and every medical discipline need to be made aware of this autosomal-dominant disease. Hematopoietic stem cell transplantation and NF-κB1 pathway-targeted therapeutic strategies should be considered in the future.
Copyright © 2020. Published by Elsevier Inc.

Entities:  

Keywords:  NF-κB1-related phenotype; NFKB1 mutation; NFKB1 variant; autosomal dominant; common variable immunodeficiency; reduced penetrance

Mesh:

Substances:

Year:  2020        PMID: 32278790     DOI: 10.1016/j.jaci.2019.11.051

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


  24 in total

1.  Droplet digital PCR for identifying copy number variations in patients with primary immunodeficiency disorders.

Authors:  See-Tarn Woon; Julia Mayes; Alexander Quach; Hilary Longhurst; Antonio Ferrante; Rohan Ameratunga
Journal:  Clin Exp Immunol       Date:  2022-05-12       Impact factor: 4.330

2.  Diagnostic Yield and Therapeutic Consequences of Targeted Next-Generation Sequencing in Sporadic Primary Immunodeficiency.

Authors:  Georgios Sogkas; Natalia Dubrowinskaja; Katharina Schütz; Lars Steinbrück; Jasper Götting; Nicolaus Schwerk; Ulrich Baumann; Bodo Grimbacher; Torsten Witte; Reinhold E Schmidt; Faranaz Atschekzei
Journal:  Int Arch Allergy Immunol       Date:  2021-10-07       Impact factor: 2.749

3.  Lymphoma as an Exclusion Criteria for CVID Diagnosis Revisited.

Authors:  Vincent Allain; Virginie Grandin; Véronique Meignin; Rémi Bertinchamp; David Boutboul; Claire Fieschi; Lionel Galicier; Laurence Gérard; Marion Malphettes; Jacinta Bustamante; Mathieu Fusaro; Nathalie Lambert; Jérémie Rosain; Christelle Lenoir; Sven Kracker; Frédéric Rieux-Laucat; Sylvain Latour; Jean-Pierre de Villartay; Capucine Picard; Eric Oksenhendler
Journal:  J Clin Immunol       Date:  2022-09-26       Impact factor: 8.542

4.  Common Variable Immunodeficiency and Neurodevelopmental Delay Due to a 13Mb Deletion on Chromosome 4 Including the NFKB1 Gene: A Case Report.

Authors:  Clara Franco-Jarava; Irene Valenzuela; Jacques G Riviere; Marina Garcia-Prat; Mónica Martínez-Gallo; Romina Dieli-Crimi; Neus Castells; Laura Batlle-Masó; Pere Soler-Palacin; Roger Colobran
Journal:  Front Immunol       Date:  2022-06-17       Impact factor: 8.786

5.  Analysis of Glaucoma Associated Genes in Response to Inflammation, an Examination of a Public Data Set Derived from Peripheral Blood from Patients with Hepatitis C.

Authors:  Jacob K Player; Sean M Riordan; R Scott Duncan; Peter Koulen
Journal:  Clin Ophthalmol       Date:  2022-06-23

6.  Droplet digital PCR for identifying copy number variations in patients with primary immunodeficiency disorders.

Authors:  See-Tarn Woon; Julia Mayes; Alexander Quach; Hilary Longhurst; Antonio Ferrante; Rohan Ameratunga
Journal:  Clin Exp Immunol       Date:  2021-12-21       Impact factor: 5.732

7.  A Pathogenic Missense Variant in NFKB1 Causes Common Variable Immunodeficiency Due to Detrimental Protein Damage.

Authors:  Manfred Fliegauf; Renate Krüger; Sophie Steiner; Leif Gunnar Hanitsch; Sarah Büchel; Volker Wahn; Horst von Bernuth; Bodo Grimbacher
Journal:  Front Immunol       Date:  2021-04-27       Impact factor: 7.561

8.  Bronchiectasis is associated with delayed diagnosis and adverse outcomes in the New Zealand Common Variable Immunodeficiency Disorders cohort study.

Authors:  R Ameratunga; A Jordan; A Cavadino; S Ameratunga; T Hills; R Steele; M Hurst; B McGettigan; I Chua; M Brewerton; N Kennedy; W Koopmans; Y Ahn; R Barker; C Allan; P Storey; C Slade; A Baker; L Huang; S-T Woon
Journal:  Clin Exp Immunol       Date:  2021-04-12       Impact factor: 5.732

9.  Establishing the Molecular Diagnoses in a Cohort of 291 Patients With Predominantly Antibody Deficiency by Targeted Next-Generation Sequencing: Experience From a Monocentric Study.

Authors:  Jessica Rojas-Restrepo; Andrés Caballero-Oteyza; Katrin Huebscher; Hanna Haberstroh; Manfred Fliegauf; Baerbel Keller; Robin Kobbe; Klaus Warnatz; Stephan Ehl; Michele Proietti; Bodo Grimbacher
Journal:  Front Immunol       Date:  2021-12-17       Impact factor: 7.561

10.  Aplastic anemia in a patient with CVID due to NFKB1 haploinsufficiency.

Authors:  Tammarah Sklarz; Stephanie N Hurwitz; Natasha L Stanley; Jane Juusola; Adam Bagg; Daria Babushok
Journal:  Cold Spring Harb Mol Case Stud       Date:  2020-12-17
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.