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Literature DB >> 31992648

Mechanisms for Ca²⁺-dependent permeability transition in mitochondria.

Paolo Bernardi¹.

Abstract

Entities: Chemical

Year: 2020 PMID： 31992648 PMCID： PMC7022152 DOI： 10.1073/pnas.1921035117

Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN： 0027-8424 Impact factor: 11.205

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9 in total

1. The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore.

Authors: Jason E Kokoszka; Katrina G Waymire; Shawn E Levy; James E Sligh; Jiyang Cai; Dean P Jones; Grant R MacGregor; Douglas C Wallace
Journal: Nature Date: 2004-01-29 Impact factor: 49.962

Review 2. Mitochondrial channels: ion fluxes and more.

Authors: Ildiko Szabo; Mario Zoratti
Journal: Physiol Rev Date: 2014-04 Impact factor: 37.312

3. Persistence of the mitochondrial permeability transition in the absence of subunit c of human ATP synthase.

Authors: Jiuya He; Holly C Ford; Joe Carroll; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2017-03-13 Impact factor: 11.205

4. Permeability transition in human mitochondria persists in the absence of peripheral stalk subunits of ATP synthase.

Authors: Jiuya He; Joe Carroll; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2017-08-07 Impact factor: 11.205

5. Persistence of the permeability transition pore in human mitochondria devoid of an assembled ATP synthase.

Authors: Joe Carroll; Jiuya He; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2019-06-18 Impact factor: 11.205

6. A large Ca2+-dependent channel formed by recombinant ADP/ATP carrier from Neurospora crassa resembles the mitochondrial permeability transition pore.

Authors: Nickolay Brustovetsky; Maximilian Tropschug; Simone Heimpel; Doerthe Heidkämper; Martin Klingenberg
Journal: Biochemistry Date: 2002-10-01 Impact factor: 3.162

7. Inhibition of mitochondrial permeability transition by deletion of the ANT family and CypD.

Authors: Jason Karch; Michael J Bround; Hadi Khalil; Michelle A Sargent; Nadina Latchman; Naohiro Terada; Pablo M Peixoto; Jeffery D Molkentin
Journal: Sci Adv Date: 2019-08-28 Impact factor: 14.136

8. Purified F-ATP synthase forms a Ca²⁺-dependent high-conductance channel matching the mitochondrial permeability transition pore.

Authors: Andrea Urbani; Valentina Giorgio; Andrea Carrer; Cinzia Franchin; Giorgio Arrigoni; Chimari Jiko; Kazuhiro Abe; Shintaro Maeda; Kyoko Shinzawa-Itoh; Janna F M Bogers; Duncan G G McMillan; Christoph Gerle; Ildikò Szabò; Paolo Bernardi
Journal: Nat Commun Date: 2019-09-25 Impact factor: 14.919

9. ATP Synthase C-Subunit-Deficient Mitochondria Have a Small Cyclosporine A-Sensitive Channel, but Lack the Permeability Transition Pore.

Authors: Maria A Neginskaya; Maria E Solesio; Elena V Berezhnaya; Giuseppe F Amodeo; Nelli Mnatsakanyan; Elizabeth A Jonas; Evgeny V Pavlov
Journal: Cell Rep Date: 2019-01-02 Impact factor: 9.423

9 in total

10 in total

1. Reply to Bernardi: The mitochondrial permeability transition pore and the ATP synthase.

Authors: John E Walker; Joe Carroll; Jiuya He
Journal: Proc Natl Acad Sci U S A Date: 2020-01-28 Impact factor: 11.205

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Authors: Alexey V Vlasov; Stepan D Osipov; Nikolay A Bondarev; Vladimir N Uversky; Valentin I Borshchevskiy; Mikhail F Yanyushin; Ilya V Manukhov; Andrey V Rogachev; Anastasiia D Vlasova; Nikolay S Ilyinsky; Alexandr I Kuklin; Norbert A Dencher; Valentin I Gordeliy
Journal: Cell Mol Life Sci Date: 2022-03-06 Impact factor: 9.261

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Authors: Yuting Cui; Mingyue Pan; Jing Ma; Xinhua Song; Weiling Cao; Peng Zhang
Journal: Mol Cell Biochem Date: 2020-09-30 Impact factor: 3.396

Review 4. Electrophysiological properties of the mitochondrial permeability transition pores: Channel diversity and disease implication.

Authors: M A Neginskaya; E V Pavlov; S-S Sheu
Journal: Biochim Biophys Acta Bioenerg Date: 2020-12-24 Impact factor: 3.991

5. The effect of mitochondrial calcium uniporter and cyclophilin D knockout on resistance of brain mitochondria to Ca²⁺-induced damage.

Authors: James Hamilton; Tatiana Brustovetsky; Nickolay Brustovetsky
Journal: J Biol Chem Date: 2021-04-14 Impact factor: 5.157

Review 6. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

Authors: Galina D Mironova; Evgeny V Pavlov
Journal: Cells Date: 2021-01-11 Impact factor: 6.600

7. Bisindolylpyrrole Induces a Cpr3- and Porin1/2-Dependent Transition in Yeast Mitochondrial Permeability in a Low Conductance State via the AACs-Associated Pore.

Authors: Masami Koushi; Rei Asakai
Journal: Int J Mol Sci Date: 2021-01-26 Impact factor: 5.923

Mechanisms for Ca²⁺-dependent permeability transition in mitochondria.

1. The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore.

Review 2. Mitochondrial channels: ion fluxes and more.

3. Persistence of the mitochondrial permeability transition in the absence of subunit c of human ATP synthase.

4. Permeability transition in human mitochondria persists in the absence of peripheral stalk subunits of ATP synthase.

5. Persistence of the permeability transition pore in human mitochondria devoid of an assembled ATP synthase.

6. A large Ca2+-dependent channel formed by recombinant ADP/ATP carrier from Neurospora crassa resembles the mitochondrial permeability transition pore.

7. Inhibition of mitochondrial permeability transition by deletion of the ANT family and CypD.

8. Purified F-ATP synthase forms a Ca²⁺-dependent high-conductance channel matching the mitochondrial permeability transition pore.

9. ATP Synthase C-Subunit-Deficient Mitochondria Have a Small Cyclosporine A-Sensitive Channel, but Lack the Permeability Transition Pore.

1. Reply to Bernardi: The mitochondrial permeability transition pore and the ATP synthase.

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Review 4. Electrophysiological properties of the mitochondrial permeability transition pores: Channel diversity and disease implication.

5. The effect of mitochondrial calcium uniporter and cyclophilin D knockout on resistance of brain mitochondria to Ca²⁺-induced damage.

Review 6. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

7. Bisindolylpyrrole Induces a Cpr3- and Porin1/2-Dependent Transition in Yeast Mitochondrial Permeability in a Low Conductance State via the AACs-Associated Pore.

8. Formation of High-Conductive C Subunit Channels upon Interaction with Cyclophilin D.

9. PK11195 Protects From Cell Death Only When Applied During Reperfusion: Succinate-Mediated Mechanism of Action.

10. Coupling/Uncoupling Reversibility in Isolated Mitochondria from Saccharomyces cerevisiae.

Review 2. Mitochondrial channels: ion fluxes and more.

Review 2. ATP synthase FOF1 structure, function, and structure-based drug design.

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Review 4. Electrophysiological properties of the mitochondrial permeability transition pores: Channel diversity and disease implication.

Review 6. Mitochondrial Cyclosporine A-Independent Palmitate/Ca2+-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Review 6. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.