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Literature DB >> 31992647

Reply to Bernardi: The mitochondrial permeability transition pore and the ATP synthase.

John E Walker¹, Joe Carroll², Jiuya He².

Abstract

Entities: Chemical

Mesh：

Substances：

Year: 2020 PMID： 31992647 PMCID： PMC7022154 DOI： 10.1073/pnas.1921409117

Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN： 0027-8424 Impact factor: 11.205

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11 in total

1. The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release.

Authors: D R Hunter; R A Haworth
Journal: Arch Biochem Biophys Date: 1979-07 Impact factor: 4.013

2. The Ca2+-induced membrane transition in mitochondria. II. Nature of the Ca2+ trigger site.

Authors: R A Haworth; D R Hunter
Journal: Arch Biochem Biophys Date: 1979-07 Impact factor: 4.013

3. Persistence of the mitochondrial permeability transition in the absence of subunit c of human ATP synthase.

Authors: Jiuya He; Holly C Ford; Joe Carroll; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2017-03-13 Impact factor: 11.205

4. Permeability transition in human mitochondria persists in the absence of peripheral stalk subunits of ATP synthase.

Authors: Jiuya He; Joe Carroll; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2017-08-07 Impact factor: 11.205

5. Mechanisms for Ca²⁺-dependent permeability transition in mitochondria.

Authors: Paolo Bernardi
Journal: Proc Natl Acad Sci U S A Date: 2020-01-28 Impact factor: 11.205

6. Persistence of the permeability transition pore in human mitochondria devoid of an assembled ATP synthase.

Authors: Joe Carroll; Jiuya He; Shujing Ding; Ian M Fearnley; John E Walker
Journal: Proc Natl Acad Sci U S A Date: 2019-06-18 Impact factor: 11.205

7. Dimers of mitochondrial ATP synthase form the permeability transition pore.

Authors: Valentina Giorgio; Sophia von Stockum; Manuela Antoniel; Astrid Fabbro; Federico Fogolari; Michael Forte; Gary D Glick; Valeria Petronilli; Mario Zoratti; Ildikó Szabó; Giovanna Lippe; Paolo Bernardi
Journal: Proc Natl Acad Sci U S A Date: 2013-03-25 Impact factor: 11.205

8. Allosteric inhibition of the Ca2+-activated hydrophilic channel of the mitochondrial inner membrane by nucleotides.

Authors: R A Haworth; D R Hunter
Journal: J Membr Biol Date: 1980-06-15 Impact factor: 1.843

9. Relationship between configuration, function, and permeability in calcium-treated mitochondria.

Authors: D R Hunter; R A Haworth; J H Southard
Journal: J Biol Chem Date: 1976-08-25 Impact factor: 5.157

10. Induction of the permeability transition pore in cells depleted of mitochondrial DNA.

Authors: Ionica Masgras; Andrea Rasola; Paolo Bernardi
Journal: Biochim Biophys Acta Date: 2012-02-28

9 in total

1. Symbiotic Origin of Apoptosis.

Authors: Szymon Kaczanowski
Journal: Results Probl Cell Differ Date: 2020

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Authors: Alexey V Vlasov; Stepan D Osipov; Nikolay A Bondarev; Vladimir N Uversky; Valentin I Borshchevskiy; Mikhail F Yanyushin; Ilya V Manukhov; Andrey V Rogachev; Anastasiia D Vlasova; Nikolay S Ilyinsky; Alexandr I Kuklin; Norbert A Dencher; Valentin I Gordeliy
Journal: Cell Mol Life Sci Date: 2022-03-06 Impact factor: 9.261

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Authors: Yuting Cui; Mingyue Pan; Jing Ma; Xinhua Song; Weiling Cao; Peng Zhang
Journal: Mol Cell Biochem Date: 2020-09-30 Impact factor: 3.396

Review 4. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

Authors: Galina D Mironova; Evgeny V Pavlov
Journal: Cells Date: 2021-01-11 Impact factor: 6.600

5. Bisindolylpyrrole Induces a Cpr3- and Porin1/2-Dependent Transition in Yeast Mitochondrial Permeability in a Low Conductance State via the AACs-Associated Pore.

Authors: Masami Koushi; Rei Asakai
Journal: Int J Mol Sci Date: 2021-01-26 Impact factor: 5.923

9. Mitochondrial ATP synthase inhibitory factor 1 interacts with the p53-cyclophilin D complex and promotes opening of the permeability transition pore.

Authors: Lishu Guo
Journal: J Biol Chem Date: 2022-03-23 Impact factor: 5.486

9 in total

Reply to Bernardi: The mitochondrial permeability transition pore and the ATP synthase.

1. The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release.

2. The Ca2+-induced membrane transition in mitochondria. II. Nature of the Ca2+ trigger site.

3. Persistence of the mitochondrial permeability transition in the absence of subunit c of human ATP synthase.

4. Permeability transition in human mitochondria persists in the absence of peripheral stalk subunits of ATP synthase.

5. Mechanisms for Ca²⁺-dependent permeability transition in mitochondria.

6. Persistence of the permeability transition pore in human mitochondria devoid of an assembled ATP synthase.

7. Dimers of mitochondrial ATP synthase form the permeability transition pore.

8. Allosteric inhibition of the Ca2+-activated hydrophilic channel of the mitochondrial inner membrane by nucleotides.

9. Relationship between configuration, function, and permeability in calcium-treated mitochondria.

10. Induction of the permeability transition pore in cells depleted of mitochondrial DNA.

1. Symbiotic Origin of Apoptosis.

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Review 4. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

5. Bisindolylpyrrole Induces a Cpr3- and Porin1/2-Dependent Transition in Yeast Mitochondrial Permeability in a Low Conductance State via the AACs-Associated Pore.

Review 6. Mitochondrial Ion Channels of the Inner Membrane and Their Regulation in Cell Death Signaling.

Review 7. Calcium influx through the mitochondrial calcium uniporter holocomplex, MCU_cx.

Review 8. Mitochondrial Permeability Transition: A Pore Intertwines Brain Aging and Alzheimer's Disease.

9. Mitochondrial ATP synthase inhibitory factor 1 interacts with the p53-cyclophilin D complex and promotes opening of the permeability transition pore.

Review 2. ATP synthase FOF1 structure, function, and structure-based drug design.

Review 3. Recent progress in the use of mitochondrial membrane permeability transition pore in mitochondrial dysfunction-related disease therapies.

Review 4. Mitochondrial Cyclosporine A-Independent Palmitate/Ca2+-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

Review 6. Mitochondrial Ion Channels of the Inner Membrane and Their Regulation in Cell Death Signaling.

Review 7. Calcium influx through the mitochondrial calcium uniporter holocomplex, MCUcx.

Review 8. Mitochondrial Permeability Transition: A Pore Intertwines Brain Aging and Alzheimer's Disease.

Review 2. ATP synthase F_OF₁ structure, function, and structure-based drug design.

Review 4. Mitochondrial Cyclosporine A-Independent Palmitate/Ca²⁺-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity.

Review 7. Calcium influx through the mitochondrial calcium uniporter holocomplex, MCU_cx.