Literature DB >> 31934802

Nanomolar Potency Aminophenyltriazine CFTR Activator Reverses Corneal Epithelial Injury in a Mouse Model of Dry Eye.

Xiaolan Chen1,2, Sujin Lee1, Tianyi Zhang1, Tianying Duan1, Neel D Pasricha3, Julie M Schallhorn3, Marc H Levin4, Vuk Koprivica5, Alan S Verkman1.   

Abstract

Purpose: Dry eye disorders are a major health care burden. We previously reported the identification of N-methyl-N-phenyl-6-(2,2,3,3-tetrafluoropropoxy)-1,3,5-triazine-2,4-diamine [cystic fibrosis transmembrane conductance regulator (CFTR)act-K267], which activated human wild-type CFTR chloride conductance with EC50 ∼ 30 nM. Here, we report in vivo evidence for CFTRact-K267 efficacy in an experimental mouse model of dry eye using a human compatible ophthalmic vehicle.
Methods: CFTR activation in mice in vivo was demonstrated by ocular surface potential difference (OSPD) measurements. Ocular surface pharmacodynamics was measured in tear fluid samples obtained at different times after topical administration of CFTRact-K267. Dry eye was produced by lacrimal duct cautery (LDC) and corneal epithelial injury and was assessed by Lissamine green (LG) staining.
Results: OSPD measurements demonstrated a hyperpolarization of -8.6 ± 3 mV (standard error of the mean, 5 mice) in response to CFTRact-K267 exposure in low chloride solution that was reversed by a CFTR inhibitor. Following single-dose topical administration of 2 nmol CFTRact-K267, tear fluid CFTRact-K267 concentration was >500 nM for more than 6 h. Following LDC, corneal surface epithelial injury, as assessed by LG staining, was substantially reversed in 10 of 12 eyes receiving 2 nmol CFTRact-K267 3 times daily starting on day 2, when marked epithelial injury had already occurred. Improvement was seen in 3 of 12 vehicle-treated eyes.
Conclusion: These studies provide in vivo evidence in mice for the efficacy of a topical, human use compatible CFTRact-K267 formulation in stimulating chloride secretion and reversing corneal epithelial injury in dry eye.

Entities:  

Keywords:  CFTR; chloride channel; keratoconjunctivitis sicca; lacrimal duct cautery

Year:  2020        PMID: 31934802      PMCID: PMC7175624          DOI: 10.1089/jop.2019.0087

Source DB:  PubMed          Journal:  J Ocul Pharmacol Ther        ISSN: 1080-7683            Impact factor:   2.671


  27 in total

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3.  Extraorbital lacrimal gland excision: a reproducible model of severe aqueous tear-deficient dry eye disease.

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6.  Presence of CFTR in the conjunctival epithelium.

Authors:  Helen C Turner; Audrey Bernstein; Oscar A Candia
Journal:  Curr Eye Res       Date:  2002-03       Impact factor: 2.424

7.  Establishment of a continuous untransfected human corneal endothelial cell line and its biocompatibility to denuded amniotic membrane.

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8.  Novel Insight Into the Role of CFTR in Lacrimal Gland Duct Function in Mice.

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Journal:  Invest Ophthalmol Vis Sci       Date:  2018-01-01       Impact factor: 4.799

Review 9.  Over the counter (OTC) artificial tear drops for dry eye syndrome.

Authors:  Andrew D Pucker; Sueko M Ng; Jason J Nichols
Journal:  Cochrane Database Syst Rev       Date:  2016-02-23

10.  Topical administration of interleukin-1 receptor antagonist as a therapy for aqueous-deficient dry eye in autoimmune disease.

Authors:  Trinka Vijmasi; Feeling Y T Chen; Ying Ting Chen; Marianne Gallup; Nancy McNamara
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2.  Novel CFTR Activator Cact-3 Ameliorates Ocular Surface Dysfunctions in Scopolamine-Induced Dry Eye Mice.

Authors:  Dongkyu Jeon; Ikhyun Jun; Ho K Lee; Jinhong Park; Bo-Rahm Kim; Kunhi Ryu; Hongchul Yoon; Tae-Im Kim; Wan Namkung
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Review 3.  Ocular Surface Ion-Channels Are Closely Related to Dry Eye: Key Research Focus on Innovative Drugs for Dry Eye.

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4.  Ocular Surface Potential Difference Measured in Human Subjects to Study Ocular Surface Ion Transport.

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5.  Genistein-Calcitriol Mitigates Hyperosmotic Stress-Induced TonEBP, CFTR Dysfunction, VDR Degradation and Inflammation in Dry Eye Disease.

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  5 in total

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