Literature DB >> 31737518

Prevention of neointimal formation after angioplasty using tetramethylpyrazine-coated balloon catheters in a rabbit iliac artery model.

Lijuan Chen1, Si Pang1, Chunshu Hao1, Aiming Xie1, Kongbo Zhu1, Yanru He1, Xiaoguo Zhang1, Wenbing Lu1, Genshan Ma1, Zhong Chen2,3.   

Abstract

BACKGROUND: Restenosis remains a clinical problem; drug-coated balloons (DCBs) have demonstrated high efficiency in this situation. DCBs prevent neointimal hyperplasia by inhibiting cell proliferation and migration. Tetramethylpyrazine (TMP) is a traditional Chinese medicine originally isolated from the rhizome of Ligusticum Walliichii, which can inhibit platelet aggregation and smooth muscle cell proliferation. We hypothesized that TMP-coated balloons (TCB) could reduce neointimal hyperplasia through the NF-κB signalling pathway.
METHODS: Twenty-one New-Zealand White rabbits (2.5-3.0 kg, male) were fed high-fat diets; 36 bilateral iliac artery stenosis models were successfully established by balloon straining. Rabbits were randomly treated with TCB (n=20) or plain balloons (PBA, n=16) (3 died during model construction). Angiographies were recorded at baseline, the immediate period, and 4 weeks later. Animals were euthanized and arteries collected for histological analysis and immunohistochemical staining. Protein expression of proliferating cell nuclear antigen (PCNA) and nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) p65 of the vessel samples were analyzed using Western blotting.
RESULTS: No difference existed in the baseline lesion characteristics or procedural results. Angiographic follow-up was successfully performed on 18 rabbits (TCB: n=20, PBA: n=16), except for 3 deaths related to the operation. Treatment with TCB was superior to that with PBA, with lower late lumen loss (0.45±0.23 vs. 0.84±0.17 mm, P<0.01). Pathological analysis confirmed the efficiency of TCB through decreasing the area stenosis rate compared with PBA (46.48%±8.22% vs. 75.24%±6.10%, P<0.01). As determined by Western blotting, significant reductions occurred in PCNA and NF-κB p65 protein intensity in the TCB group versus the PBA group (all P<0.01). TCB efficiently mitigated restenosis in the rabbit iliac artery model.
CONCLUSIONS: This study elucidated that TCB could restrain intimal hyperplasia of vessels by inhibiting the activation of the NF-κB pathway to reduce inflammatory response and decrease the rate of cell proliferation through suppressing PCNA expression. 2019 Cardiovascular Diagnosis and Therapy. All rights reserved.

Entities:  

Keywords:  Tetramethylpyrazine (TMP); inflammatory; prevention; proliferation; restenosis

Year:  2019        PMID: 31737518      PMCID: PMC6837916          DOI: 10.21037/cdt.2019.09.04

Source DB:  PubMed          Journal:  Cardiovasc Diagn Ther        ISSN: 2223-3652


  19 in total

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8.  Tetramethylpyrazine-eluting stents prevented in-stent restenosis in a porcine model.

Authors:  Genshan Ma; Shu Ding; Yi Feng; Chengxing Shen; Lijuan Chen; Zhong Chen
Journal:  J Cardiovasc Pharmacol       Date:  2007-08       Impact factor: 3.105

9.  [Effect of tetramethylpyrazine on the proliferation and collagen synthesis of vascular smooth muscle cells].

Authors:  Jun-Yi Hua; Yu-Zhou He; Xu-Hong Jiang; Wu Ye; Mei-Ya Yang
Journal:  Zhongguo Zhong Xi Yi Jie He Za Zhi       Date:  2013-09

10.  Tetramethylpyrazine protects against high glucose-induced vascular smooth muscle cell injury through inhibiting the phosphorylation of JNK, p38MAPK, and ERK.

Authors:  Yutao Liu; Xu Li; Shanling Jiang; Quanli Ge
Journal:  J Int Med Res       Date:  2018-07-12       Impact factor: 1.671

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