Literature DB >> 31479507

The effects of insecticides on two splice variants of the glutamate-gated chloride channel receptor of the major malaria vector, Anopheles gambiae.

Mohammed Atif1, Joseph W Lynch1, Angelo Keramidas1.   

Abstract

BACKGROUND AND
PURPOSE: Between half to 1 million people die annually from malaria. Anopheles gambiae mosquitoes are major malaria vectors. Unfortunately, resistance has emerged to the agents currently used to control A. gambiae, creating a demand for novel control measures. The pentameric glutamate-gated chloride channel (GluCl) expressed in the muscle and nerve cells of these organisms are a potentially important biological target for malaria control. The pharmacological properties of Anophiline GluCl receptors are, however, largely unknown. Accordingly, we compared the efficacy of four insecticides (lindane, fipronil, picrotoxin, and ivermectin) on two A. gambiae GluCl receptor splice variants with the aim of providing a molecular basis for designing novel anti-malaria treatments. EXPERIMENTAL APPROACH: The A. gambiae GluCl receptor b1 and c splice variants were expressed homomerically in Xenopus laevis oocytes and studied with electrophysiological techniques, using two-electrode voltage-clamp. KEY
RESULTS: The b1 and c GluCl receptors were activated with similar potencies by glutamate and ivermectin. Fipronil was more potent than picrotoxin and lindane at inhibiting glutamate- and ivermectin-gated currents. Importantly, b1 GluCl receptors exhibited reduced sensitivity to picrotoxin and lindane. They also recovered from these effects to a greater extent than c GluCl receptors CONCLUSIONS AND IMPLICATIONS: The two splice variant subunits exhibited differential sensitivities to multiple, structurally divergent insecticides, without accompanying changes in the sensitivity to the endogenous neurotransmitter, glutamate, implying that drug resistance may be caused by alterations in relative subunit expression levels, without affecting physiological function. Our results strongly suggest that it should be feasible to develop novel subunit-specific pharmacological agents.
© 2019 The British Pharmacological Society.

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Year:  2019        PMID: 31479507      PMCID: PMC6976876          DOI: 10.1111/bph.14855

Source DB:  PubMed          Journal:  Br J Pharmacol        ISSN: 0007-1188            Impact factor:   8.739


  63 in total

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3.  Mechanism of action of the insecticides, lindane and fipronil, on glycine receptor chloride channels.

Authors:  Robiul Islam; Joseph W Lynch
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4.  Point mutations in the M2 region of the alpha, beta, or gamma subunit of the GABAA channel that abolish block by picrotoxin.

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5.  Species specific RNA A-to-I editing of mosquito RDL modulates GABA potency and influences agonistic, potentiating and antagonistic actions of ivermectin.

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8.  Identification and distribution of a GABA receptor mutation conferring dieldrin resistance in the malaria vector Anopheles funestus in Africa.

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9.  GluClR-mediated inhibitory postsynaptic currents reveal targets for ivermectin and potential mechanisms of ivermectin resistance.

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10.  Preliminary efficacy investigations of oral fipronil against Anopheles arabiensis when administered to Zebu cattle (Bos indicus) under field conditions.

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  3 in total

1.  The effects of insecticides on two splice variants of the glutamate-gated chloride channel receptor of the major malaria vector, Anopheles gambiae.

Authors:  Mohammed Atif; Joseph W Lynch; Angelo Keramidas
Journal:  Br J Pharmacol       Date:  2019-10-31       Impact factor: 8.739

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Journal:  Pestic Biochem Physiol       Date:  2020-11-08       Impact factor: 3.963

3.  The molecular targets of ivermectin and lotilaner in the human louse Pediculus humanus humanus: New prospects for the treatment of pediculosis.

Authors:  Nicolas Lamassiaude; Berthine Toubate; Cédric Neveu; Pierre Charnet; Catherine Dupuy; Françoise Debierre-Grockiego; Isabelle Dimier-Poisson; Claude L Charvet
Journal:  PLoS Pathog       Date:  2021-02-18       Impact factor: 6.823

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