Literature DB >> 31424985

Blood Leukocyte DNA Methylation Predicts Risk of Future Myocardial Infarction and Coronary Heart Disease.

Golareh Agha1, Michael M Mendelson2,3,4, Cavin K Ward-Caviness5,6, Roby Joehanes2,7, TianXiao Huan8, Rahul Gondalia9, Elias Salfati10, Jennifer A Brody11, Giovanni Fiorito12, Jan Bressler13, Brian H Chen14, Symen Ligthart15, Simonetta Guarrera12, Elena Colicino16, Allan C Just16, Simone Wahl17, Christian Gieger17, Amy R Vandiver18, Toshiko Tanaka14, Dena G Hernandez19, Luke C Pilling20, Andrew B Singleton19, Carlotta Sacerdote21, Vittorio Krogh22, Salvatore Panico23, Rosario Tumino24, Yun Li25, Guosheng Zhang26, James D Stewart27, James S Floyd11, Kerri L Wiggins11, Jerome I Rotter28, Michael Multhaup18, Kelly Bakulski29, Steven Horvath30, Philip S Tsao10, Devin M Absher31, Pantel Vokonas32, Joel Hirschhorn33,34, M Daniele Fallin35, Chunyu Liu36, Stefania Bandinelli37, Eric Boerwinkle38,39, Abbas Dehghan40, Joel D Schwartz41, Bruce M Psaty42,43, Andrew P Feinberg44, Lifang Hou45, Luigi Ferrucci46, Nona Sotoodehnia47, Giuseppe Matullo12, Annette Peters48,49, Myriam Fornage50, Themistocles L Assimes51, Eric A Whitsel52, Daniel Levy2,3, Andrea A Baccarelli1.   

Abstract

BACKGROUND: DNA methylation is implicated in coronary heart disease (CHD), but current evidence is based on small, cross-sectional studies. We examined blood DNA methylation in relation to incident CHD across multiple prospective cohorts.
METHODS: Nine population-based cohorts from the United States and Europe profiled epigenome-wide blood leukocyte DNA methylation using the Illumina Infinium 450k microarray, and prospectively ascertained CHD events including coronary insufficiency/unstable angina, recognized myocardial infarction, coronary revascularization, and coronary death. Cohorts conducted race-specific analyses adjusted for age, sex, smoking, education, body mass index, blood cell type proportions, and technical variables. We conducted fixed-effect meta-analyses across cohorts.
RESULTS: Among 11 461 individuals (mean age 64 years, 67% women, 35% African American) free of CHD at baseline, 1895 developed CHD during a mean follow-up of 11.2 years. Methylation levels at 52 CpG (cytosine-phosphate-guanine) sites were associated with incident CHD or myocardial infarction (false discovery rate<0.05). These CpGs map to genes with key roles in calcium regulation (ATP2B2, CASR, GUCA1B, HPCAL1), and genes identified in genome- and epigenome-wide studies of serum calcium (CASR), serum calcium-related risk of CHD (CASR), coronary artery calcified plaque (PTPRN2), and kidney function (CDH23, HPCAL1), among others. Mendelian randomization analyses supported a causal effect of DNA methylation on incident CHD; these CpGs map to active regulatory regions proximal to long non-coding RNA transcripts.
CONCLUSION: Methylation of blood-derived DNA is associated with risk of future CHD across diverse populations and may serve as an informative tool for gaining further insight on the development of CHD.

Entities:  

Keywords:  coronary artery disease; coronary heart disease; epigenetics; gene expression regulation; genomics

Mesh:

Year:  2019        PMID: 31424985      PMCID: PMC6812683          DOI: 10.1161/CIRCULATIONAHA.118.039357

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


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