Literature DB >> 31405905

Community-informed connectomics of the thalamocortical system in generalized epilepsy.

Zhengge Wang1, Sara Larivière1, Qiang Xu1, Reinder Vos de Wael1, Seok-Jun Hong1, Zhongyuan Wang1, Yun Xu1, Bin Zhu1, Neda Bernasconi1, Andrea Bernasconi1, Bing Zhang1, Zhiqiang Zhang1, Boris C Bernhardt2.   

Abstract

OBJECTIVE: To study the intrinsic organization of the thalamocortical circuitry in patients with generalized epilepsy with tonic-clonic seizures (GTCS) via resting-state fMRI (rs-fMRI) connectome analysis and to evaluate its relation to drug response.
METHODS: In a prospectively followed-up sample of 41 patients and 27 healthy controls, we obtained rs-fMRI and structural MRI. After 1 year of follow-up, 27 patients were classified as seizure-free and 14 as drug-resistant. We examined connectivity within and between resting-state communities in cortical and thalamic subregions. In addition to comparing patients to controls, we examined associations with seizure control. We assessed reproducibility in an independent cohort of 21 patients.
RESULTS: Compared to controls, patients showed a more constrained network embedding of the thalamus, while frontocentral neocortical regions expressed increased functional diversity. Findings remained significant after regressing out thalamic volume and cortical thickness, suggesting independence from structural alterations. We observed more marked network imbalances in drug-resistant compared to seizure-free patients. Findings were similar in the reproducibility dataset.
CONCLUSIONS: Our findings suggest a pathoconnectomic mechanism of generalized epilepsy centered on diverging changes in cortical and thalamic connectivity. More restricted thalamic connectivity could reflect the tendency to engage in recursive thalamocortical loops, which may contribute to hyperexcitability. Conversely, increased connectional diversity of frontocentral networks may relay abnormal activity to an extended bilateral territory. Network imbalances were observed shortly after diagnosis and related to future drug response, suggesting clinical utility.
© 2019 American Academy of Neurology.

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Year:  2019        PMID: 31405905      PMCID: PMC6746209          DOI: 10.1212/WNL.0000000000008096

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  43 in total

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Authors:  S Murray Sherman
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Review 4.  Cellular and network mechanisms of spike-wave seizures.

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5.  Altered thalamocortical functional connectivity in idiopathic generalized epilepsy.

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Journal:  Epilepsia       Date:  2014-03-20       Impact factor: 5.864

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  16 in total

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5.  Connective profiles and antagonism between dynamic and static connectivity underlying generalized epilepsy.

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6.  Cortico-striato-thalamo-cerebellar networks of structural covariance underlying different epilepsy syndromes associated with generalized tonic-clonic seizures.

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Review 8.  Cognitive Function in Genetic Generalized Epilepsies: Insights From Neuropsychology and Neuroimaging.

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Journal:  Sci Adv       Date:  2020-11-18       Impact factor: 14.136

10.  Disrupted topological properties of functional networks in epileptic children with generalized tonic-clonic seizures.

Authors:  Yongxin Li; Qian Chen; Wenhua Huang
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