Literature DB >> 31326317

Identification of risk loci and a polygenic risk score for lung cancer: a large-scale prospective cohort study in Chinese populations.

Juncheng Dai1, Jun Lv2, Meng Zhu3, Yuzhuo Wang4, Na Qin4, Hongxia Ma5, Yong-Qiao He6, Ruoxin Zhang7, Wen Tan8, Jingyi Fan4, Tianpei Wang4, Hong Zheng9, Qi Sun4, Lijuan Wang4, Mingtao Huang4, Zijun Ge4, Canqing Yu2, Yu Guo10, Tong-Min Wang6, Jie Wang11, Lin Xu11, Weibing Wu12, Liang Chen12, Zheng Bian10, Robin Walters13, Iona Y Millwood13, Xi-Zhao Li6, Xin Wang14, Rayjean J Hung15, David C Christiani16, Haiquan Chen17, Mengyun Wang7, Cheng Wang18, Yue Jiang3, Kexin Chen9, Zhengming Chen13, Guangfu Jin5, Tangchun Wu19, Dongxin Lin8, Zhibin Hu5, Christopher I Amos20, Chen Wu8, Qingyi Wei21, Wei-Hua Jia6, Liming Li2, Hongbing Shen22.   

Abstract

BACKGROUND: Genetic variation has an important role in the development of non-small-cell lung cancer (NSCLC). However, genetic factors for lung cancer have not been fully identified, especially in Chinese populations, which limits the use of existing polygenic risk scores (PRS) to identify subpopulations at high risk of lung cancer for prevention. We therefore aimed to identify novel loci associated with NSCLC risk, and generate a PRS and evaluate its utility and effectiveness in the prediction of lung cancer risk in Chinese populations.
METHODS: To systematically identify genetic variants for NSCLC risk, we newly genotyped 19 546 samples from Chinese NSCLC cases and controls from the Nanjing Medical University Global Screening Array Project and did a meta-analysis of genome-wide association studies (GWASs) of 27 120 individuals with NSCLC and 27 355 without NSCLC (13 327 cases and 13 328 controls of Chinese descent as well as 13 793 cases and 14 027 controls of European descent). We then built a PRS for Chinese populations from all reported single-nucleotide polymorphisms that have been reported to be associated with lung cancer risk at genome-wide significance level. We evaluated the utility and effectiveness of the generated PRS in predicting subpopulations at high-risk of lung cancer in an independent prospective cohort of 95 408 individuals from the China Kadoorie Biobank (CKB) with more than 10 years' follow-up.
FINDINGS: We identified 19 susceptibility loci to be significantly associated with NSCLC risk at p≤5·0 × 10-8, including six novel loci. When applied to the CKB cohort, the PRS of the risk loci successfully predicted lung cancer incident cases in a dose-response manner in participants at a high genetic risk (top 10%) than those at a low genetic risk (bottom 10%; adjusted hazard ratio 1·96, 95% CI 1·53-2·51; ptrend=2·02 × 10-9). Specially, we observed consistently separated curves of lung cancer events in individuals at low, intermediate, and high genetic risk, respectively, and PRS was an independent effective risk stratification indicator beyond age and smoking pack-years.
INTERPRETATION: We have shown for the first time that GWAS-derived PRS can be effectively used in discriminating subpopulations at high risk of lung cancer, who might benefit from a practically feasible PRS-based lung cancer screening programme for precision prevention in Chinese populations. FUNDING: National Natural Science Foundation of China, the Priority Academic Program for the Development of Jiangsu Higher Education Institutions, National Key R&D Program of China, Science Foundation for Distinguished Young Scholars of Jiangsu, and China's Thousand Talents Program.
Copyright © 2019 Elsevier Ltd. All rights reserved.

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Year:  2019        PMID: 31326317      PMCID: PMC7015703          DOI: 10.1016/S2213-2600(19)30144-4

Source DB:  PubMed          Journal:  Lancet Respir Med        ISSN: 2213-2600            Impact factor:   30.700


  32 in total

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5.  A Novel Pathway-Based Approach Improves Lung Cancer Risk Prediction Using Germline Genetic Variations.

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Review 6.  Epidemiology of lung cancer: Diagnosis and management of lung cancer, 3rd ed: American College of Chest Physicians evidence-based clinical practice guidelines.

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8.  A flexible and accurate genotype imputation method for the next generation of genome-wide association studies.

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10.  Breast Cancer Risk From Modifiable and Nonmodifiable Risk Factors Among White Women in the United States.

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Journal:  JAMA Oncol       Date:  2016-10-01       Impact factor: 31.777

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  42 in total

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2.  Association Analysis of Driver Gene-Related Genetic Variants Identified Novel Lung Cancer Susceptibility Loci with 20,871 Lung Cancer Cases and 15,971 Controls.

Authors:  Yuzhuo Wang; Olga Y Gorlova; Ivan P Gorlov; Meng Zhu; Juncheng Dai; Demetrius Albanes; Stephen Lam; Adonina Tardon; Chu Chen; Gary E Goodman; Stig E Bojesen; Maria Teresa Landi; Mattias Johansson; Angela Risch; Heunz-Erich Wichmann; Heike Bickeboller; David C Christiani; Gad Rennert; Susanne M Arnold; Paul Brennan; John K Field; Sanjay Shete; Loïc Le Marchand; Olle Melander; Hans Brunnstrom; Geoffrey Liu; Rayjean J Hung; Angeline S Andrew; Lambertus A Kiemeney; Shanbeh Zienolddiny; Kjell Grankvist; Mikael Johansson; Neil E Caporaso; Penella J Woll; Philip Lazarus; Matthew B Schabath; Melinda C Aldrich; Victoria L Stevens; Hongxia Ma; Guangfu Jin; Zhibin Hu; Christopher I Amos; Hongbing Shen
Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2020-04-10       Impact factor: 4.254

3.  Cross-ancestry genome-wide meta-analysis of 61,047 cases and 947,237 controls identifies new susceptibility loci contributing to lung cancer.

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4.  A causal variant rs3769823 in 2q33.1 involved in apoptosis pathway leading to a decreased risk of non-small cell lung cancer.

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Review 5.  The genetic and evolutionary determinants of COVID-19 susceptibility.

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6.  Incorporating both genetic and tobacco smoking data to identify high-risk smokers for lung cancer screening.

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7.  Potential Genes Associated with the Survival of Lung Adenocarcinoma Were Identified by Methylation.

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8.  Association of smoking and polygenic risk with the incidence of lung cancer: a prospective cohort study.

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9.  Whole-exome sequencing reveals common and rare variants in immunologic and neurological genes implicated in achalasia.

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10.  Mapping the human genetic architecture of COVID-19.

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