Literature DB >> 31197603

Gastric Sleeve Surgery Alleviates Obesity-Associated Insulin Resistance and Suppresses Endoplasmic Reticulum Stress in Adipose Tissue of db/db Mice.

Ming Sun1, Wenyan Zhao2, Shuqiang Li2, Chunfei Li2, Yong Feng2, Donghua Geng3.   

Abstract

OBJECTIVE: The aims of this study were to evaluate the effects of gastric sleeve surgery on diabetes remission in db/db mice as well as to determine the underlying mechanisms.
METHODS: Thirty spontaneously obese, diabetic mice (C57BL/Ksj-db/db) were randomly divided into three groups: sleeve gastrectomy group, sham-operated group, and control db/db group. Ten db/m lean mice were used as nondiabetic littermate controls. All mice were sacrificed on day 28. The fasting plasma glucose, serum insulin, lipid profile, and oral glucose tolerance were measured pre- and postoperatively. Inflammatory cytokines (TNF-α and IL-6), endoplasmic reticulum (ER) stress-related markers (GRP78, PERK, IRE-1, and ATF6), and glucose transporter 4 (GLUT4) in the adipose tissue were assayed.
RESULTS: Sleeve gastrectomy significantly reduced the body weight and food intake in the db/db mice. This surgery improved glucose and lipid metabolism, as manifested by the decrease in the fasting plasma glucose level and partial restoration of lipid abnormalities. Also, the surgery improved glucose tolerance and alleviated insulin resistance in db/db mice. Sleeve gastrectomy surgery induced downregulation of the inflammatory adipocytokines TNF-α and IL-6; suppressed expression of the ER stress-related markers GRP78, PERK, IRE-1, and ATF-6; and increased the expression and distribution of GLUT4 in adipose tissue of db/db mice.
CONCLUSION: The improvement in glucose tolerance following sleeve gastrectomy is associated with alleviation of insulin resistance, reduction of inflammatory adipocytokine levels, and suppression of ER stress. Further studies are needed to assess whether these effects have a causal role.

Entities:  

Keywords:  Endoplasmic reticulum stress; Gastric sleeve surgery; Obesity; Type 2 diabetes mellitus

Mesh:

Substances:

Year:  2019        PMID: 31197603     DOI: 10.1007/s11695-019-03966-6

Source DB:  PubMed          Journal:  Obes Surg        ISSN: 0960-8923            Impact factor:   4.129


  31 in total

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9.  GLUT4 defects in adipose tissue are early signs of metabolic alterations in Alms1GT/GT, a mouse model for obesity and insulin resistance.

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Journal:  PLoS One       Date:  2014-10-09       Impact factor: 3.240

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Authors:  Noritaka Kawasaki; Rie Asada; Atsushi Saito; Soshi Kanemoto; Kazunori Imaizumi
Journal:  Sci Rep       Date:  2012-11-12       Impact factor: 4.379

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Journal:  Biomolecules       Date:  2020-02-17

2.  Prolonged preoperative fasting induces postoperative insulin resistance by ER-stress mediated Glut4 down-regulation in skeletal muscles.

Authors:  Ming-Wei Lin; Chih-I Chen; Tzu-Ting Cheng; Chien-Chi Huang; Jen-Wei Tsai; Guan-Ming Feng; Tzer-Zen Hwang; Chen-Fuh Lam
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3.  Hydrogen Sulfide Ameliorates Angiotensin II-Induced Atrial Fibrosis Progression to Atrial Fibrillation Through Inhibition of the Warburg Effect and Endoplasmic Reticulum Stress.

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