Literature DB >> 30983543

The effect of intestinal glucose load on neural regulation of food craving.

Marion A Stopyra1,2, Hans-Christoph Friederich1, Sebastian Sailer1, Sabina Pauen3, Martin Bendszus4, Wolfgang Herzog1, Joe J Simon1.   

Abstract

Objectives: Excess sugar consumption, particularly in the form of sweetened beverages, has been identified as a pivotal contributor to the epidemic of obesity and associated metabolic disorders. However, the impact of sugar-sweetened beverages on food craving is still inconclusive. Therefore, the present study aimed to specifically investigate the effects of an intestinal glucose load on neural processing of food cues.
Methods: Using a single-blind fMRI design, 26 normal-weight women were scanned on two occasions, after receiving either a glucose or water infusion directly into the stomach using a nasogastric tube, without being aware of the type of infusion. Participants had to either view neutral and food images, or were asked to distract themselves from these images by solving an arithmetic task.
Results: In response to viewing high-caloric food cues, we observed increased activation in reward-related brain areas. During food distraction, fronto-parietal brain regions were recruited, which are commonly related to attentional deployment and hedonic valuation. Furthermore, activity in the dorsolateral prefrontal cortex showed increased functional connectivity with the insula and was correlated with subjective craving levels to food cues. Despite an increase of blood glucose levels in response to the glucose compared to the water infusion, neither subjective food craving nor neural regulation of food craving showed significant differences. Conclusions: These findings support a decreased satiation effect of sweet beverages, as intestinal glucose ingestion and signalling showed no significant effect on cortical brain circuits associated with food craving. This trial was registered at clinicaltrials.gov as NCT03075371.

Entities:  

Keywords:  FMRI; craving; food distraction; glucose ingestion

Mesh:

Substances:

Year:  2019        PMID: 30983543     DOI: 10.1080/1028415X.2019.1600275

Source DB:  PubMed          Journal:  Nutr Neurosci        ISSN: 1028-415X            Impact factor:   4.994


  6 in total

1.  Neuroimaging of hypothalamic mechanisms related to glucose metabolism in anorexia nervosa and obesity.

Authors:  Joe J Simon; Marion A Stopyra; Esther Mönning; Sebastian Sailer; Nora Lavandier; Lars P Kihm; Martin Bendszus; Hubert Preissl; Wolfgang Herzog; Hans-Christoph Friederich
Journal:  J Clin Invest       Date:  2020-08-03       Impact factor: 14.808

2.  Evidence of a Role for the Hippocampus in Food-Cue Processing and the Association with Body Weight and Dietary Added Sugar.

Authors:  Sabrina Jones; Shan Luo; Hilary M Dorton; Brendan Angelo; Alexandra G Yunker; John R Monterosso; Kathleen A Page
Journal:  Obesity (Silver Spring)       Date:  2021-02       Impact factor: 5.002

3.  The influence of homeostatic mechanisms on neural regulation of food craving in anorexia nervosa.

Authors:  Marion A Stopyra; Hans-Christoph Friederich; Esther Mönning; Nora Lavandier; Martin Bendszus; Wolfgang Herzog; Joe J Simon
Journal:  Psychol Med       Date:  2020-01-14       Impact factor: 7.723

4.  Homeostasis and food craving in obesity: a functional MRI study.

Authors:  M A Stopyra; H-C Friederich; N Lavandier; E Mönning; M Bendszus; W Herzog; J J Simon
Journal:  Int J Obes (Lond)       Date:  2021-08-17       Impact factor: 5.095

Review 5.  Endoscopic intragastric balloon: a gimmick or a viable option for obesity?

Authors:  Katja Susanne Claudia Gollisch; Dirk Raddatz
Journal:  Ann Transl Med       Date:  2020-03

Review 6.  The Gut-Brain Axis and Its Role in Controlling Eating Behavior in Intestinal Inflammation.

Authors:  Gordon William Moran; Gita Thapaliya
Journal:  Nutrients       Date:  2021-03-18       Impact factor: 5.717

  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.