Literature DB >> 30936005

Does Tranexamic Acid Improve Clot Strength in Severely Injured Patients Who Have Elevated Fibrin Degradation Products and Low Fibrinolytic Activity, Measured by Thrombelastography?

Hunter B Moore1, Ernest E Moore2, Michael P Chapman3, Kirk C Hansen4, Mitchell J Cohen5, Frederic M Pieracci5, James Chandler5, Angela Sauaia6.   

Abstract

BACKGROUND: Elevated d-dimers in injured patients with paradoxically low fibrinolysis activity measured by viscoelastic assays have been speculated to be "occult" fibrinolysis. However, an alternative explanation is that these patients have previously activated their fibrinolytic system and have shut it down by the time of blood draw, and would gain no benefit in clot strength with tranexamic acid (TXA). We hypothesize that TXA will not increase clot strength in injured patients with low fibrinolytic activity measured by thrombelastography (TEG), despite biomarkers of fibrinolysis activation. STUDY
DESIGN: Three TEG assays (rapid, tissue plasminogen activator, and functional fibrinogen) were run on trauma patients. The tissue plasminogen activator TEG served as a functional assay to quantify depletion of fibrinolysis inhibitors (DFI). Patients were stratified by DFI vs non-DFI and then by rapid TEG lysis at 30 minutes phenotype cutoffs. Response to TXA was evaluated with functional fibrinogen TEG by calculating percent change in clot strength with the addition of exogenous TXA in the TEG cup.
RESULTS: Six hundred and thirty patients with a median new injury severity score of 20 were analyzed. Depletion of fibrinolysis inhibitors was present in 116 (18%). The DFI patients had significantly increased d-dimer (p < 0.001) and lower fibrinogen (p < 0.001). The DFI patients had increased rates of massive transfusion (33% vs 3.3%; p < 0.001) and mortality (40% vs 6.2%; p < 0.001). Among DFI patients, TXA significantly improved fibrin clot strength with hyperfibrinolysis (+19% clot strength; p < 0.001) but not with shutdown (+1.2%) or physiologic (-2.5%).
CONCLUSIONS: Patients with DFI have multiple abnormalities of their coagulation system, but only DFI patients with hyperfibrinolysis have improved fibrin clot strength with TXA.
Copyright © 2019. Published by Elsevier Inc.

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Year:  2019        PMID: 30936005      PMCID: PMC6874093          DOI: 10.1016/j.jamcollsurg.2019.03.015

Source DB:  PubMed          Journal:  J Am Coll Surg        ISSN: 1072-7515            Impact factor:   6.113


  39 in total

1.  Mechanism of action of fibrinolysin in the prevention of irreversible hemorrhagic shock.

Authors:  R M HARDAWAY; J W BURNS
Journal:  Ann Surg       Date:  1963-02       Impact factor: 12.969

2.  Prevention of "irreversible" hemorrhagic shock with fibrinolysin.

Authors:  R M HARDAWAY; D C DRAKE
Journal:  Ann Surg       Date:  1963-01       Impact factor: 12.969

3.  Tranexamic acid mediates proinflammatory and anti-inflammatory signaling via complement C5a regulation in a plasminogen activator-dependent manner.

Authors:  Christopher D Barrett; Hunter B Moore; Yi-Wen Kong; Michael P Chapman; Ganapathy Sriram; Dan Lim; Ernest E Moore; Michael B Yaffe
Journal:  J Trauma Acute Care Surg       Date:  2019-01       Impact factor: 3.313

4.  Rationale for the selective administration of tranexamic acid to inhibit fibrinolysis in the severely injured patient.

Authors:  Ernest E Moore; Hunter B Moore; Eduardo Gonzalez; Angela Sauaia; Anirban Banerjee; Christopher C Silliman
Journal:  Transfusion       Date:  2016-04       Impact factor: 3.157

5.  Fibrinolytic shutdown: fascinating theory but randomized controlled trial data are needed.

Authors:  Ian Roberts
Journal:  Transfusion       Date:  2016-04       Impact factor: 3.157

6.  Persistent Fibrinolysis Shutdown Is Associated with Increased Mortality in Severely Injured Trauma Patients.

Authors:  Jonathan P Meizoso; Charles A Karcutskie; Juliet J Ray; Nicholas Namias; Carl I Schulman; Kenneth G Proctor
Journal:  J Am Coll Surg       Date:  2016-12-23       Impact factor: 6.113

7.  TEG Lysis Shutdown Represents Coagulopathy in Bleeding Trauma Patients: Analysis of the PROPPR Cohort.

Authors:  Jessica C Cardenas; Charles E Wade; Bryan A Cotton; Mitchell J George; John B Holcomb; Martin A Schreiber; Nathan J White
Journal:  Shock       Date:  2019-03       Impact factor: 3.454

8.  Plasma-first resuscitation to treat haemorrhagic shock during emergency ground transportation in an urban area: a randomised trial.

Authors:  Hunter B Moore; Ernest E Moore; Michael P Chapman; Kevin McVaney; Gary Bryskiewicz; Robert Blechar; Theresa Chin; Clay Cothren Burlew; Fredric Pieracci; F Bernadette West; Courtney D Fleming; Arsen Ghasabyan; James Chandler; Christopher C Silliman; Anirban Banerjee; Angela Sauaia
Journal:  Lancet       Date:  2018-07-20       Impact factor: 79.321

9.  Plasma QconCATs reveal a gender-specific proteomic signature in apheresis platelet plasma supernatants.

Authors:  Monika Dzieciatkowska; Angelo D'Alessandro; Ryan C Hill; Kirk C Hansen
Journal:  J Proteomics       Date:  2015-03-02       Impact factor: 4.044

10.  Label-Free Kinetic Studies of Hemostasis-Related Biomarkers Including D-Dimer Using Autologous Serum Transfusion.

Authors:  Heiko Rühl; Christina Berens; Anna Winterhagen; Jens Müller; Johannes Oldenburg; Bernd Pötzsch
Journal:  PLoS One       Date:  2015-12-14       Impact factor: 3.240

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  7 in total

1.  Detection of early allograft dysfunction at 30 min of reperfusion in liver transplantation: An intraoperative diagnostic tool with real time assessment of graft function.

Authors:  Hunter B Moore; Hillary Yaffe; James J Pomposelli; Michael Wachs; Thomas Bak; Peter Kennealey; Kendra Conzen; Megan Adams; Thomas Pshak; Rashikh Choudhury; Carson Walker; Alexander Schulick; Tanner Ferrell; Michael P Chapman; Elizabeth A Pomfret; Trevor L Nydam
Journal:  Am J Surg       Date:  2020-08-27       Impact factor: 2.565

2.  A clinical coagulopathy score concurrent with viscoelastic testing defines opportunities to improve hemostatic resuscitation and enhance blood product utilization during liver transplantation.

Authors:  Alexander C Schulick; Hunter B Moore; Carson B Walker; Hillary Yaffe; James J Pomposelli; Fareed Azam; Michael Wachs; Thomas Bak; Peter Kennealey; Kendra Conzen; Megan Adams; Thomas Pshak; Rashikh Choudhury; Michael P Chapman; Elizabeth A Pomfret; Trevor L Nydam
Journal:  Am J Surg       Date:  2020-09-01       Impact factor: 2.565

Review 3.  The Role of Tranexamic Acid in the Management of an Acutely Hemorrhaging Patient.

Authors:  Steven Davis; Aria Nawab; Christiaan van Nispen; Ali Pourmand
Journal:  Hosp Pharm       Date:  2020-02-13

Review 4.  Trauma-induced coagulopathy.

Authors:  Ernest E Moore; Hunter B Moore; Lucy Z Kornblith; Matthew D Neal; Maureane Hoffman; Nicola J Mutch; Herbert Schöchl; Beverley J Hunt; Angela Sauaia
Journal:  Nat Rev Dis Primers       Date:  2021-04-29       Impact factor: 65.038

5.  Fibrinolytic system activation immediately following trauma was quickly and intensely suppressed in a rat model of severe blunt trauma.

Authors:  Mineji Hayakawa; Takumi Tsuchida; Yoshinori Honma; Asumi Mizugaki; Takayoshi Ooyasu; Tomonao Yoshida; Tomoyo Saito; Kenichi Katabami; Takeshi Wada; Kunihiko Maekawa
Journal:  Sci Rep       Date:  2021-10-13       Impact factor: 4.379

6.  Not all patients benefit from the postoperative antifibrinolytic treatment: clinical evidence against the universal use of tranexamic acid following total knee arthroplasty.

Authors:  Jiacheng Liu; Han Wang; Xiangdong Wu; Yiting Lei; Wei Huang
Journal:  J Orthop Surg Res       Date:  2022-01-29       Impact factor: 2.359

7.  Modern Management of Bleeding, Clotting, and Coagulopathy in Trauma Patients: What Is the Role of Viscoelastic Assays?

Authors:  Sanjeev Dhara; Ernest E Moore; Michael B Yaffe; Hunter B Moore; Christopher D Barrett
Journal:  Curr Trauma Rep       Date:  2020-01-23
  7 in total

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