Literature DB >> 30802166

DNA Damage Response Mediates Pressure Overload-Induced Cardiomyocyte Hypertrophy.

Yuji Nakada1, Ngoc Uyen Nhi Nguyen1, Feng Xiao1, Jainy J Savla1, Nicholas T Lam1, Salim Abdisalaam2, Souparno Bhattacharya2, Shibani Mukherjee2, Aroumougame Asaithamby2, Thomas G Gillette1, Joseph A Hill1,3, Hesham A Sadek1,3.   

Abstract

Entities:  

Keywords:  DNA damage; DNA, double-stranded; KU60019; angiotensin II; ataxia telangiectasia, mutated; hypertrophy; myocytes, cardiac

Mesh：

Substances：

Year:  2019        PMID： 30802166      PMCID： PMC6467068          DOI： 10.1161/CIRCULATIONAHA.118.034822

Source DB:  PubMed          Journal:  Circulation        ISSN： 0009-7322            Impact factor:   29.690


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3.  The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response.

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5.  DNA single-strand break-induced DNA damage response causes heart failure.

Authors:  Tomoaki Higo; Atsuhiko T Naito; Tomokazu Sumida; Masato Shibamoto; Katsuki Okada; Seitaro Nomura; Akito Nakagawa; Toshihiro Yamaguchi; Taku Sakai; Akihito Hashimoto; Yuki Kuramoto; Masamichi Ito; Shungo Hikoso; Hiroshi Akazawa; Jong-Kook Lee; Ichiro Shiojima; Peter J McKinnon; Yasushi Sakata; Issei Komuro
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8.  Resistin deletion protects against heart failure injury by targeting DNA damage response.

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9.  Stress-sensing in the human greying hair follicle: Ataxia Telangiectasia Mutated (ATM) depletion in hair bulb melanocytes in canities-prone scalp.

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10.  Atrial Fibrillation Underlies Cardiomyocyte Senescence and Contributes to Deleterious Atrial Remodeling during Disease Progression.

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