Literature DB >> 30793327

Lung disease in STAT3 hyper-IgE syndrome requires intense therapy.

Carolin Kröner1,2, Jens Neumann3, Julia Ley-Zaporozhan4, Beate Hagl1,5, Iris Meixner1, Benedikt D Spielberger1,5, Gregor Dückers6, Bernd H Belohradsky1, Tim Niehues6, Michael Borte7, Joseph Rosenecker1, Matthias Kappler1, Susanne Nährig8, Simone Reu3, Matthias Griese1,2, Ellen D Renner1,5,9.   

Abstract

BACKGROUND: Pulmonary complications are responsible for high morbidity and mortality rates in patients with the rare immunodeficiency disorder STAT3 hyper-IgE syndrome (STAT3-HIES). The aim of this study was to expand knowledge about lung disease in STAT3-HIES.
METHODS: The course of pulmonary disease, radiological and histopathological interrelations, therapeutic management, and the outcome of 14 STAT3-HIES patients were assessed.
RESULTS: The patients' quality of life was compromised most by pulmonary disease. All 14 patients showed first signs of lung disease at a median onset of 1.5 years of age. Lung function revealed a mixed obstructive-restrictive impairment with reduced FEV1 and FVC in 75% of the patients. The severity of lung function impairment was associated with Aspergillus fumigatus infection and prior lung surgery. Severe lung tissue damage, with reduced numbers of ATP-binding cassette sub-family A member 3 (ABCA3) positive type II pneumocytes, was observed in the histological assessment of two deceased patients. Imaging studies of all patients above 6 years of age showed severe airway and parenchyma destruction. Lung surgeries frequently led to complications, including fistula formation. Long-term antifungal and antibacterial treatment proved to be beneficial, as were inhalation therapy, chest physiotherapy, and exercise. Regular immunoglobulin replacement therapy tended to stabilize lung function.
CONCLUSIONS: Due to its severity, pulmonary disease in STAT3-HIES patients requires strict monitoring and intensive therapy.
© 2019 EAACI and John Wiley and Sons A/S. Published by John Wiley and Sons Ltd.

Entities:  

Keywords:  ABCA3; STAT3 hyper-IgE syndrome; bronchiectasis; lung disease; pneumatocele; primary immunodeficiency

Year:  2019        PMID: 30793327     DOI: 10.1111/all.13753

Source DB:  PubMed          Journal:  Allergy        ISSN: 0105-4538            Impact factor:   13.146


  5 in total

Review 1.  Respiratory Mycoses in COPD and Bronchiectasis.

Authors:  Pei Yee Tiew; Micheál Mac Aogáin; Soo Kai Ter; Stefano Aliberti; James D Chalmers; Sanjay H Chotirmall
Journal:  Mycopathologia       Date:  2021-03-11       Impact factor: 2.574

2.  Pulmonary Infections and Surgical Complications in a Young Girl with Signal Transducer and Activator of Transcription 3 Loss-of-Function Mutation Hyperimmunoglobulin E Syndrome: A Case Report.

Authors:  Crhistian Toribio-Dionicio; Dania Cubas-Guzmán; Pedro Guerra-Canchari; Vanuza García-Sánchez; Wilmer Córdova-Calderón
Journal:  Pediatr Allergy Immunol Pulmonol       Date:  2021-03       Impact factor: 1.349

Review 3.  STAT3 Hyper-IgE Syndrome-an Update and Unanswered Questions.

Authors:  Christo Tsilifis; Alexandra F Freeman; Andrew R Gennery
Journal:  J Clin Immunol       Date:  2021-05-01       Impact factor: 8.317

4.  Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients.

Authors:  Vera Schwierzeck; Renate Effner; Felicitas Abel; Matthias Reiger; Gundula Notheis; Jürgen Held; Valeska Simon; Sebastian Dintner; Reinhard Hoffmann; Beate Hagl; Johannes Huebner; Alexander Mellmann; Ellen D Renner
Journal:  J Clin Immunol       Date:  2022-06-02       Impact factor: 8.542

5.  Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential.

Authors:  Iris Meixner; Beate Hagl; Carolin I Kröner; Benedikt D Spielberger; Ekaterini Paschos; Gregor Dückers; Tim Niehues; Ronny Hesse; Ellen D Renner
Journal:  Orphanet J Rare Dis       Date:  2020-09-10       Impact factor: 4.123

  5 in total

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