Literature DB >> 30716477

No Association Between Vitamin D Status and Risk of Barrett's Esophagus or Esophageal Adenocarcinoma: A Mendelian Randomization Study.

Jing Dong1, Puya Gharahkhani2, Wong-Ho Chow3, Marilie D Gammon4, Geoffrey Liu5, Carlos Caldas6, Anna H Wu7, Weimin Ye8, Lynn Onstad9, Lesley A Anderson10, Leslie Bernstein11, Paul D Pharoah12, Harvey A Risch13, Douglas A Corley14, Rebecca C Fitzgerald15, Prasad G Iyer16, Brian J Reid9, Jesper Lagergren17, Nicholas J Shaheen18, Thomas L Vaughan9, Stuart MacGregor2, Sharon Love19, Claire Palles20, Ian Tomlinson20, Ines Gockel21, Andrea May22, Christian Gerges23, Mario Anders24, Anne C Böhmer25, Jessica Becker26, Nicole Kreuser21, Rene Thieme21, Tania Noder27, Marino Venerito28, Lothar Veits29, Thomas Schmidt30, Claudia Schmidt31, Jakob R Izbicki32, Arnulf H Hölscher31, Hauke Lang33, Dietmar Lorenz34, Brigitte Schumacher35, Rupert Mayershofer36, Yogesh Vashist37, Katja Ott38, Michael Vieth29, Josef Weismüller39, Markus M Nöthen25, Susanne Moebus40, Michael Knapp41, Wilbert H M Peters42, Horst Neuhaus43, Thomas Rösch23, Christian Ell22, Janusz Jankowski44, Johannes Schumacher25, Rachel E Neale45, David C Whiteman46, Aaron P Thrift47.   

Abstract

BACKGROUND & AIMS: Epidemiology studies of circulating concentrations of 25 hydroxy vitamin D (25(OH)D) and risk of esophageal adenocarcinoma (EAC) have produced conflicting results. We conducted a Mendelian randomization study to determine the associations between circulating concentrations of 25(OH)D and risks of EAC and its precursor, Barrett's esophagus (BE).
METHODS: We conducted a Mendelian randomization study using a 2-sample (summary data) approach. Six single-nucleotide polymorphisms (SNPs; rs3755967, rs10741657, rs12785878, rs10745742, rs8018720, and rs17216707) associated with circulating concentrations of 25(OH)D were used as instrumental variables. We collected data from 6167 patients with BE, 4112 patients with EAC, and 17,159 individuals without BE or EAC (controls) participating in the Barrett's and Esophageal Adenocarcinoma Consortium, as well as studies from Bonn, Germany, and Cambridge and Oxford, United Kingdom. Analyses were performed separately for BE and EAC.
RESULTS: Overall, we found no evidence for an association between genetically estimated 25(OH)D concentration and risk of BE or EAC. The odds ratio per 20 nmol/L increase in genetically estimated 25(OH)D concentration for BE risk estimated by combining the individual SNP association using inverse variance weighting was 1.21 (95% CI, 0.77-1.92; P = .41). The odds ratio for EAC risk, estimated by combining the individual SNP association using inverse variance weighting, was 0.68 (95% CI, 0.39-1.19; P = .18).
CONCLUSIONS: In a Mendelian randomization study, we found that low genetically estimated 25(OH)D concentrations were not associated with risk of BE or EAC.
Copyright © 2019 AGA Institute. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  BEACON; Chemoprevention; Esophageal Cancer; Etiology; Risk Factors

Year:  2019        PMID: 30716477      PMCID: PMC6675666          DOI: 10.1016/j.cgh.2019.01.041

Source DB:  PubMed          Journal:  Clin Gastroenterol Hepatol        ISSN: 1542-3565            Impact factor:   11.382


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