Literature DB >> 30465177

Exosomal miR-155 Derived from Hepatocellular Carcinoma Cells Under Hypoxia Promotes Angiogenesis in Endothelial Cells.

Yusuke Matsuura1, Hiroshi Wada1, Hidetoshi Eguchi2, Kunihito Gotoh1, Shogo Kobayashi1, Mitsuru Kinoshita1, Masahiko Kubo1, Koji Hayashi1, Yoshifumi Iwagami1, Daisaku Yamada1, Tadafumi Asaoka1, Takehiro Noda1, Koichi Kawamoto1, Yutaka Takeda1,3, Masahiro Tanemura1,4, Koji Umeshita5, Yuichiro Doki1, Masaki Mori1.   

Abstract

PURPOSE: In this study, we aim to clarify whether exosomes secreted from hepatocellular carcinoma (HCC) cells under hypoxia affect angiogenesis in endothelial cells.
METHODS: Exosomes derived from human liver cancer cell lines were cultured under hypoxic or normoxic conditions for 24 h, isolated using ExoQuick-TC®, and co-cultured with HUVECs to evaluate angiogenic activity. We also evaluated the expression of miR-155 in the exosomes from 40 patients with HCC.
RESULTS: Exosomes under hypoxia remarkably enhanced tube formation of HUVECs. Both cellular and exosomal miR-155 were significantly up-regulated under hypoxic conditions. Knockdown of miR-155 in HCC cells attenuated the promotion of tube formation by exosomes under hypoxia in HUVECs, and high expression of exosomal miR-155 in preoperative plasma was significantly correlated with early recurrence.
CONCLUSION: These results suggest that exosomes derived from HCC cells under hypoxia induce tube formation of HUVECs and that exosomal miR-155 may affect angiogenic activity in HCC.

Entities:  

Keywords:  Angiogenesis; Exosome; HCC; Hypoxia; miR-155

Mesh:

Substances:

Year:  2018        PMID: 30465177     DOI: 10.1007/s10620-018-5380-1

Source DB:  PubMed          Journal:  Dig Dis Sci        ISSN: 0163-2116            Impact factor:   3.199


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