Literature DB >> 30257283

Is internal thoracic artery resistant to reperfusion injury? Evaluation of the storage of free internal thoracic artery grafts.

Gábor Veres1, Harald Schmidt2, Péter Hegedűs2, Sevil Korkmaz-Icöz2, Tamás Radovits3, Sivakkanan Loganathan2, Paige Brlecic2, Shiliang Li2, Matthias Karck2, Gábor Szabó2.   

Abstract

OBJECTIVES: The in situ internal thoracic artery (ITA) is recognized as the best conduit for coronary artery bypass surgery. The ITA-if it is used as an in situ graft-has a much higher late patency rate than any other arterial graft, including a free ITA graft. We sought to determine if the use of the ITA as an in situ/free graft and its storage in preservation solutions, have an effect on endothelial function.
METHODS: The ITA was harvested as either a free or in situ graft in a porcine model. Free grafts were stored in different preservation solutions (saline, Custodiol and Tiprotec [both Köhler Chemie GmbH, Bensheim, Germany]). The ITA was anastomosed off pump to the left anterior descending artery (as in situ/free graft). Freshly harvested ITA served as a control. After 2 hours of reperfusion, the implanted grafts were harvested. The assessment of endothelial function, histopathological analysis, and gene expression were performed.
RESULTS: Endothelial function and integrity were severely impaired after reperfusion in the free ITA groups, however, it was partially preserved in the Tiprotec group. Reperfusion injury resulted in increased nitro-oxidative stress, DNA breakage, vascular cell adhesion protein 1, intercellular adhesion molecule-1, and caspase-3 scores, and a decreased endothelial nitric oxide synthase score in the free ITA groups. The in situ ITA graft showed no signs of injury. mRNA levels were significantly altered among the groups.
CONCLUSIONS: An early, severe endothelial dysfunction of the stored, free ITA as described, could be completely prevented by the use of an in situ ITA graft. Tiprotec might be a feasible option for storage of free arterial grafts during coronary artery bypass grafting.
Copyright © 2018 The American Association for Thoracic Surgery. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  CABG; endothelial dysfunction; internal thoracic artery; preservation solution

Mesh:

Year:  2018        PMID: 30257283     DOI: 10.1016/j.jtcvs.2018.05.079

Source DB:  PubMed          Journal:  J Thorac Cardiovasc Surg        ISSN: 0022-5223            Impact factor:   5.209


  3 in total

1.  Pharmacological activation of soluble guanylate cyclase improves vascular graft function.

Authors:  Gábor Veres; Yang Bai; Klára Aliz Stark; Harald Schmidt; Tamás Radovits; Sivakkanan Loganathan; Sevil Korkmaz-Icöz; Gábor Szabó
Journal:  Interact Cardiovasc Thorac Surg       Date:  2021-05-10

2.  Impact of skeletonized harvesting of the internal thoracic artery on intrasternal microcirculation considering preparation quality.

Authors:  Lars Saemann; Alina Zubarevich; Folker Wenzel; Jasmin Soethoff; Sivakkanan Loganathan; Sevil Korkmaz-Icöz; Matthias Karck; Gábor Szabó; Gábor Veres
Journal:  Interact Cardiovasc Thorac Surg       Date:  2021-10-29

3.  Aspirin Reduces Ischemia-Reperfusion Injury Induced Endothelial Cell Damage of Arterial Grafts in a Rodent Model.

Authors:  Gábor Veres; Kálmán Benke; Roland Stengl; Yang Bai; Klára Aliz Stark; Alex Ali Sayour; Tamás Radovits; Sivakkanan Loganathan; Sevil Korkmaz-Icöz; Matthias Karck; Gábor Szabó
Journal:  Antioxidants (Basel)       Date:  2022-01-18
  3 in total

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