Literature DB >> 29603301

Quantification of lectin fluorescence in GNE myopathy muscle biopsies.

Petcharat Leoyklang1, Bradley Class2, Satoru Noguchi3, William A Gahl1,4, Nuria Carrillo1,2, Ichizo Nishino3, Marjan Huizing1, May Christine Malicdan1,4.   

Abstract

INTRODUCTION: GNE myopathy is an adult-onset muscle disorder characterized by impaired sialylation of (muscle) glycans, detectable by lectin histochemistry. We describe a standardized method to quantify (lectin-) fluorescence in muscle sections, applicable for diagnosis and response to therapy for GNE myopathy.
METHODS: Muscle sections were fluorescently labeled with the sialic acid-binding Sambucus nigra agglutinin (SNA) lectin and antibodies to sarcolemma residence protein caveolin-3 (CAV-3). Entire tissue sections were imaged in tiles and fluorescence was quantified.
RESULTS: SNA fluorescence co-localizing with CAV-3 was ∼50% decreased in GNE myopathy biopsies compared with muscle-matched controls, confirming previous qualitative results. DISCUSSION: This quantitative fluorescence method can accurately determine sialylation status of GNE myopathy muscle biopsies. This method is adaptable for expression of other membrane-associated muscle proteins, and may be of benefit for disorders in which therapeutic changes in expression are subtle and difficult to assess by other methods. Muscle Nerve 58: 286-292, 2018.
© 2018 Wiley Periodicals, Inc.

Entities:  

Keywords:  N-acetylmannosamine (ManNAc); SNA lectin; caveolin-3; sarcolemma; sialylation

Mesh:

Substances:

Year:  2018        PMID: 29603301      PMCID: PMC6105422          DOI: 10.1002/mus.26135

Source DB:  PubMed          Journal:  Muscle Nerve        ISSN: 0148-639X            Impact factor:   3.217


  31 in total

1.  UDP-GlcNAc 2-epimerase: a regulator of cell surface sialylation.

Authors:  O T Keppler; S Hinderlich; J Langner; R Schwartz-Albiez; W Reutter; M Pawlita
Journal:  Science       Date:  1999-05-21       Impact factor: 47.728

2.  Sialylation of Thomsen-Friedenreich antigen is a noninvasive blood-based biomarker for GNE myopathy.

Authors:  Petcharat Leoyklang; May Christine Malicdan; Tal Yardeni; Frank Celeste; Carla Ciccone; Xueli Li; Rong Jiang; William A Gahl; Nuria Carrillo-Carrasco; Miao He; Marjan Huizing
Journal:  Biomark Med       Date:  2014       Impact factor: 2.851

Review 3.  Muscular dystrophies due to glycosylation defects: diagnosis and therapeutic strategies.

Authors:  Francesco Muntoni; Silvia Torelli; Dominic J Wells; Susan C Brown
Journal:  Curr Opin Neurol       Date:  2011-10       Impact factor: 5.710

4.  Caveolin-3 in muscular dystrophy.

Authors:  E M McNally; E de Sá Moreira; D J Duggan; C G Bönnemann; M P Lisanti; H G Lidov; M Vainzof; M R Passos-Bueno; E P Hoffman; M Zatz; L M Kunkel
Journal:  Hum Mol Genet       Date:  1998-05       Impact factor: 6.150

5.  NCAM is hyposialylated in hereditary inclusion body myopathy due to GNE mutations.

Authors:  E Ricci; A Broccolini; T Gidaro; R Morosetti; C Gliubizzi; R Frusciante; G M Di Lella; P A Tonali; M Mirabella
Journal:  Neurology       Date:  2006-03-14       Impact factor: 9.910

6.  Oral monosaccharide therapies to reverse renal and muscle hyposialylation in a mouse model of GNE myopathy.

Authors:  Terren K Niethamer; Tal Yardeni; Petcharat Leoyklang; Carla Ciccone; Adrian Astiz-Martinez; Katherine Jacobs; Heidi M Dorward; Patricia M Zerfas; William A Gahl; Marjan Huizing
Journal:  Mol Genet Metab       Date:  2012-10-18       Impact factor: 4.797

7.  Sialic acid-binding lectins: submolecular specificity and interaction with sialoglycoproteins and tumour cells.

Authors:  E Fischer; R Brossmer
Journal:  Glycoconj J       Date:  1995-10       Impact factor: 2.916

8.  Reduction of UDP-N-acetylglucosamine 2-epimerase/N-acetylmannosamine kinase activity and sialylation in distal myopathy with rimmed vacuoles.

Authors:  Satoru Noguchi; Yoko Keira; Kumiko Murayama; Megumu Ogawa; Masako Fujita; Genri Kawahara; Yasushi Oya; Masaoki Imazawa; Yu-Ichi Goto; Yukiko K Hayashi; Ikuya Nonaka; Ichizo Nishino
Journal:  J Biol Chem       Date:  2004-01-05       Impact factor: 5.157

9.  Mutations in the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy.

Authors:  C Minetti; F Sotgia; C Bruno; P Scartezzini; P Broda; M Bado; E Masetti; M Mazzocco; A Egeo; M A Donati; D Volonte; F Galbiati; G Cordone; F D Bricarelli; M P Lisanti; F Zara
Journal:  Nat Genet       Date:  1998-04       Impact factor: 38.330

Review 10.  Sialic acids as regulators of molecular and cellular interactions.

Authors:  Roland Schauer
Journal:  Curr Opin Struct Biol       Date:  2009-08-19       Impact factor: 6.809

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  4 in total

Review 1.  The role of amyloid β in the pathological mechanism of GNE myopathy.

Authors:  Tongtong Zhang; Ren Shang; Jing Miao
Journal:  Neurol Sci       Date:  2022-07-29       Impact factor: 3.830

Review 2.  GNE Myopathy: Etiology, Diagnosis, and Therapeutic Challenges.

Authors:  Nuria Carrillo; May C Malicdan; Marjan Huizing
Journal:  Neurotherapeutics       Date:  2018-10       Impact factor: 7.620

3.  Population Pharmacokinetic Model of N-acetylmannosamine (ManNAc) and N-acetylneuraminic acid (Neu5Ac) in Subjects with GNE Myopathy.

Authors:  Scott Van Wart; Donald E Mager; Cindy J Bednasz; Marjan Huizing; Nuria Carrillo
Journal:  Drugs R D       Date:  2021-04-24

4.  Congenital thrombocytopenia associated with GNE mutations in twin sisters: a case report and literature review.

Authors:  Xin Li; Ying Li; Min Lei; Jing Tian; Zuocheng Yang; Shoujin Kuang; Yanjuan Tan; Tao Bo
Journal:  BMC Med Genet       Date:  2020-11-16       Impact factor: 2.103

  4 in total

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