Literature DB >> 29594656

Glioblastoma (GBM) effects on quantitative MRI of contralateral normal appearing white matter.

Hatef Mehrabian1,2, Wilfred W Lam3, Sten Myrehaug4,5, Arjun Sahgal6,4,5, Greg J Stanisz3,6,7.   

Abstract

PURPOSE: The objective was to investigate (with quantitative MRI) whether the normal appearing white matter (NAWM) of glioblastoma (GBM) patients on the contralateral side (cNAWM) was different from NAWM of healthy controls.
METHODS: Thirteen patients with newly diagnosed GBM and nine healthy age-matched controls were MRI-scanned with quantitative magnetization transfer (qMT), chemical exchange saturation transfer (CEST), and transverse relaxation time (T2)-mapping. MRI scans were performed after surgery and before chemo-radiation treatment. Comprehensive qMT, CEST, T2 data were acquired. A two-pool MT model was fit to qMT data in transient state, to calculate MT model parameters [Formula: see text]. CEST signal was isolated by removing the contributions from the MT and direct water saturation, and CEST signal was calculated for Amide (CESTAmide), Amine (CESTAmine) and nuclear overhauser effect, NOE (CESTNOE).
RESULTS: There was no difference between GBM patients and normal controls in the qMT properties of the macromolecular pool [Formula: see text]. However, their free water pool spectrum was different (1/RaT2a,patient = 28.1 ± 3.9, 1/RaT2a,control = 25.0 ± 1.1, p = 0.03). This difference could be attributed to the difference in their T2 time ([Formula: see text] = 83 ± 4, [Formula: see text] = 88 ± 1, p = 0.004). CEST signals were statistically significantly different with the CESTAmide having the largest difference between the two cohorts (CESTAmide,patient = 2.8 ± 0.4, CESTAmide,control = 3.4 ± 0.5, p = 0.009).
CONCLUSIONS: CEST in cNAWM of GBM patients was lower than healthy controls which could be caused by modified brain metabolism due to tumor cell infiltration. There was no difference in MT properties of the patients and controls, however, the differences in free water pool properties were mainly due to reduced T2 in cNAWM of the patients (resulting from structural changes and increased cellularity).

Entities:  

Keywords:  Chemical exchange saturation transfer (CEST); Glioblastoma (GBM); Normal appearing white matter (NAWM); Quantitative magnetization transfer (qMT)

Mesh:

Year:  2018        PMID: 29594656     DOI: 10.1007/s11060-018-2846-0

Source DB:  PubMed          Journal:  J Neurooncol        ISSN: 0167-594X            Impact factor:   4.130


  43 in total

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3.  Whole-brain N-acetylaspartate spectroscopy and diffusion tensor imaging in patients with newly diagnosed gliomas: a preliminary study.

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4.  Guidelines for the treatment of newly diagnosed glioblastoma: introduction.

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5.  Adult glioblastoma multiforme survival in the temozolomide era: a population-based analysis of Surveillance, Epidemiology, and End Results registries.

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6.  Quantitative assessment of amide proton transfer (APT) and nuclear overhauser enhancement (NOE) imaging with extrapolated semisolid magnetization transfer reference (EMR) signals: II. Comparison of three EMR models and application to human brain glioma at 3 Tesla.

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7.  Mapping of amide, amine, and aliphatic peaks in the CEST spectra of murine xenografts at 7 T.

Authors:  Kimberly L Desmond; Firas Moosvi; Greg J Stanisz
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2.  Quantitative CEST and MT at 1.5T for monitoring treatment response in glioblastoma: early and late tumor progression during chemoradiation.

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4.  Does the magnetization transfer effect bias chemical exchange saturation transfer effects? Quantifying chemical exchange saturation transfer in the presence of magnetization transfer.

Authors:  Alex K Smith; Kevin J Ray; James R Larkin; Martin Craig; Seth A Smith; Michael A Chappell
Journal:  Magn Reson Med       Date:  2020-02-18       Impact factor: 4.668

5.  3D gradient echo snapshot CEST MRI with low power saturation for human studies at 3T.

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7.  The Value of APTw CEST MRI in Routine Clinical Assessment of Human Brain Tumor Patients at 3T.

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8.  3D APT and NOE CEST-MRI of healthy volunteers and patients with non-enhancing glioma at 3 T.

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10.  Whole brain apparent diffusion coefficient measurements correlate with survival in glioblastoma patients.

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