Literature DB >> 29460236

A double-blind, randomised, placebo-controlled, cross-over study assessing the use of XEN-D0103 in patients with paroxysmal atrial fibrillation and implanted pacemakers allowing continuous beat-to-beat monitoring of drug efficacy.

S R Shunmugam1, Conn Sugihara2, Nick Freemantle3, Patrick Round4, Steve Furniss2, Neil Sulke2.   

Abstract

PURPOSE: The ultrarapid delayed rectifier current (IKur) carried by Kv1.5 channels, which are solely expressed in the atrium, is a potential target for safer treatment of paroxysmal atrial fibrillation (PAF). XEN-D0103 is a nanomolar ion channel blocker that selectively inhibits potassium ion flux through the Kv1.5 ion channel. The efficacy of XEN-D0103 in reducing AF burden was assessed in patients with DDDRp permanent pacemakers (PPMs) and PAF.
METHODS: A double-blind, placebo-controlled, cross-over study was performed in patients with PAF and DDDRp PPMs with advanced atrial and ventricular Holters allowing beat-to-beat arrhythmia follow-up. All anti-arrhythmic drugs were withdrawn before randomised treatment. After baseline assessment, patients were randomly assigned to two treatment periods of placebo then XEN-D0103 50 mg bd, or XEN-D0103 50 mg bd then placebo.
RESULTS: Fifty-four patients were screened and 21 patients were eligible and included in the randomised trial. All 21 patients completed both treatment periods. The primary endpoint was change in AF burden assessed by PPM. There was no significant difference in AF burden on treatment with XEN-D0103 versus placebo. There was a reduction in the mean frequency of AF episodes (relative reduction 0.72, 95% CI 0.66 to 0.77; p < 0.0001). XEN-D0103 was safe and well tolerated, and there were no serious adverse events. XEN-D0103 did not have any apparent effect on heart rate compared to placebo.
CONCLUSIONS: XEN-D0103 did not reduce AF burden in patients with PAF and dual chamber pacemakers providing beat-to-beat monitoring. XEN-D0103 was well tolerated and did not have any apparent effect on heart rate. Although single-ion channel blockade with XEN-D0103 did not affect AF in this study, there might be a potential for this agent to be used in combination with other atrially specific drugs in the treatment of AF. EUDRACT TRIAL REGISTRATION NUMBER: 2013-004456-38.

Entities:  

Keywords:  Dual chamber pacemakers; I Kur channel blocker; Paroxysmal AF

Mesh:

Substances:

Year:  2018        PMID: 29460236     DOI: 10.1007/s10840-018-0318-2

Source DB:  PubMed          Journal:  J Interv Card Electrophysiol        ISSN: 1383-875X            Impact factor:   1.900


  21 in total

1.  AVE0118, blocker of the transient outward current (I(to)) and ultrarapid delayed rectifier current (I(Kur)), fully restores atrial contractility after cardioversion of atrial fibrillation in the goat.

Authors:  Sunniva de Haan; Maura Greiser; Erik Harks; Yuri Blaauw; Arne van Hunnik; Sander Verheule; Maurits Allessie; Ulrich Schotten
Journal:  Circulation       Date:  2006-08-28       Impact factor: 29.690

2.  Primary structure and functional expression of a mouse inward rectifier potassium channel.

Authors:  Y Kubo; T J Baldwin; Y N Jan; L Y Jan
Journal:  Nature       Date:  1993-03-11       Impact factor: 49.962

3.  Ionic targets for drug therapy and atrial fibrillation-induced electrical remodeling: insights from a mathematical model.

Authors:  M Courtemanche; R J Ramirez; S Nattel
Journal:  Cardiovasc Res       Date:  1999-05       Impact factor: 10.787

Review 4.  Controversies in atrial fibrillation.

Authors:  Stanley Nattel; Lionel H Opie
Journal:  Lancet       Date:  2006-01-21       Impact factor: 79.321

5.  A randomized, placebo-controlled study of vernakalant (oral) for the prevention of atrial fibrillation recurrence after cardioversion.

Authors:  Christian Torp-Pedersen; Dimitar H Raev; Garth Dickinson; Noam N Butterfield; Brian Mangal; Gregory N Beatch
Journal:  Circ Arrhythm Electrophysiol       Date:  2011-08-14

Review 6.  New drugs targeting the cardiac ultra-rapid delayed-rectifier current (I Kur): rationale, pharmacology and evidence for potential therapeutic value.

Authors:  John W Ford; James T Milnes
Journal:  J Cardiovasc Pharmacol       Date:  2008-08       Impact factor: 3.105

7.  Sustained depolarization-induced outward current in human atrial myocytes. Evidence for a novel delayed rectifier K+ current similar to Kv1.5 cloned channel currents.

Authors:  Z Wang; B Fermini; S Nattel
Journal:  Circ Res       Date:  1993-12       Impact factor: 17.367

8.  Pathology-specific effects of the IKur/Ito/IK,ACh blocker AVE0118 on ion channels in human chronic atrial fibrillation.

Authors:  T Christ; E Wettwer; N Voigt; O Hála; S Radicke; K Matschke; A Várro; D Dobrev; U Ravens
Journal:  Br J Pharmacol       Date:  2008-06-09       Impact factor: 8.739

9.  Coronary ischemic events after first atrial fibrillation: risk and survival.

Authors:  Yoko Miyasaka; Marion E Barnes; Bernard J Gersh; Stephen S Cha; Kent R Bailey; James B Seward; Toshiji Iwasaka; Teresa S M Tsang
Journal:  Am J Med       Date:  2007-04       Impact factor: 4.965

10.  Opportunistic detection of atrial fibrillation in subjects aged 65 years or older in primare care: a randomised clinical trial of efficacy. DOFA-AP study protocol.

Authors:  Luis Á Pérula-de-Torres; Miguel Á Martínez-Adell; Virginia González-Blanco; José M Baena-Díez; Enrique Martín-Rioboó; Juan M Parras-Rejano; Jesús González-Lama; Remedios Martín-Alvarez; Roger Ruiz-Moral; José Á Fernández-García; Modesto Pérez-Díaz; Joaquin Ruiz-de-Castroviejo; Carlos Pérula-de-Torres; Antonio Valero-Martín; Ana Roldán-Villalobos; Margarita Criado-Larumbe; Emili Burdoy-Joaquín; Montserrat Coma-Solé; Mercè Cervera-León; Lluís Cuixart-Costa
Journal:  BMC Fam Pract       Date:  2012-10-30       Impact factor: 2.497

View more
  8 in total

1.  Populations of in silico myocytes and tissues reveal synergy of multiatrial-predominant K+ -current block in atrial fibrillation.

Authors:  Haibo Ni; Alex Fogli Iseppe; Wayne R Giles; Sanjiv M Narayan; Henggui Zhang; Andrew G Edwards; Stefano Morotti; Eleonora Grandi
Journal:  Br J Pharmacol       Date:  2020-08-09       Impact factor: 8.739

Review 2.  Why translation from basic discoveries to clinical applications is so difficult for atrial fibrillation and possible approaches to improving it.

Authors:  Stanley Nattel; Philip T Sager; Jörg Hüser; Jordi Heijman; Dobromir Dobrev
Journal:  Cardiovasc Res       Date:  2021-06-16       Impact factor: 10.787

Review 3.  Computational Modeling of Electrophysiology and Pharmacotherapy of Atrial Fibrillation: Recent Advances and Future Challenges.

Authors:  Márcia Vagos; Ilsbeth G M van Herck; Joakim Sundnes; Hermenegild J Arevalo; Andrew G Edwards; Jussi T Koivumäki
Journal:  Front Physiol       Date:  2018-09-04       Impact factor: 4.566

4.  Small Conductance Ca2 +-Activated K+ (SK) Channel mRNA Expression in Human Atrial and Ventricular Tissue: Comparison Between Donor, Atrial Fibrillation and Heart Failure Tissue.

Authors:  Elisa Darkow; Thong T Nguyen; Marina Stolina; Fabian A Kari; Constanze Schmidt; Felix Wiedmann; István Baczkó; Peter Kohl; Sridharan Rajamani; Ursula Ravens; Rémi Peyronnet
Journal:  Front Physiol       Date:  2021-04-01       Impact factor: 4.566

Review 5.  The contribution of ion channels to shaping macrophage behaviour.

Authors:  Anna Selezneva; Alasdair J Gibb; Dean Willis
Journal:  Front Pharmacol       Date:  2022-09-07       Impact factor: 5.988

6.  The KCa2 Channel Inhibitor AP30663 Selectively Increases Atrial Refractoriness, Converts Vernakalant-Resistant Atrial Fibrillation and Prevents Its Reinduction in Conscious Pigs.

Authors:  Jonas Goldin Diness; Jeppe Egedal Kirchhoff; Tobias Speerschneider; Lea Abildgaard; Nils Edvardsson; Ulrik S Sørensen; Morten Grunnet; Bo Hjorth Bentzen
Journal:  Front Pharmacol       Date:  2020-02-28       Impact factor: 5.810

Review 7.  Computational models of atrial fibrillation: achievements, challenges, and perspectives for improving clinical care.

Authors:  Jordi Heijman; Henry Sutanto; Harry J G M Crijns; Stanley Nattel; Natalia A Trayanova
Journal:  Cardiovasc Res       Date:  2021-06-16       Impact factor: 10.787

Review 8.  Peptide Inhibitors of Kv1.5: An Option for the Treatment of Atrial Fibrillation.

Authors:  Jesús Borrego; Adam Feher; Norbert Jost; Gyorgy Panyi; Zoltan Varga; Ferenc Papp
Journal:  Pharmaceuticals (Basel)       Date:  2021-12-14
  8 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.