Literature DB >> 29355888

Association of microRNA-200c expression levels with clinicopathological factors and prognosis in endometrioid endometrial cancer.

Milosz Wilczynski1, Justyna Danielska2, Daria Domanska-Senderowska3, Monika Dzieniecka4, Bozena Szymanska5, Andrzej Malinowski6.   

Abstract

INTRODUCTION: MicroRNAs (miRNAs) are regulators of gene expression, which play an important role in many critical cellular processes including apoptosis, proliferation and cell differentiation. Aberrant miRNA expression has been reported in a variety of human malignancies. Therefore, miRNAs may be potentially used as cancer biomarkers. miRNA-200c, which is a member of the miRNA-200 family, might play an essential role in tumor progression. The purpose of this study was to evaluate the prognostic and clinical significance of miRNA-200c in women with endometrioid endometrial cancer.
MATERIAL AND METHODS: Total RNA extraction from 90 archival formalin-fixed paraffin-embedded tissue samples of endometri-oid endometrial cancer and 10 normal endometrium samples was performed. After cDNA synthesis, real-time polymerase chain reaction was conducted and relative expression of miRNA-200c was assessed. Then, miRNA-200c expression levels were evaluated with regard to clinicopathological characteristics.
RESULTS: The expression levels of miRNA-200c were significantly increased in endometrioid endometrial cancer samples. Expression of miRNA-200c maintained at significantly higher levels in the early stage endometrioid endometrial cancer compared with more advanced stages. In the Kaplan-Meier analysis, lower levels of miRNA-200c expression were associated with inferior survival.
CONCLUSIONS: Expression levels of miRNA-200c might be associated with clinicopathological factors and survival in endometrioid endometrial cancer.
© 2018 Nordic Federation of Societies of Obstetrics and Gynecology.

Entities:  

Keywords:  Endometrial cancer; gynecological cancers; microRNA; microRNA-200c; oncology

Mesh:

Substances:

Year:  2018        PMID: 29355888     DOI: 10.1111/aogs.13306

Source DB:  PubMed          Journal:  Acta Obstet Gynecol Scand        ISSN: 0001-6349            Impact factor:   3.636


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