Literature DB >> 29352507

Nuclear and membrane estrogen receptor antagonists induce similar mTORC2 activation-reversible changes in synaptic protein expression and actin polymerization in the mouse hippocampus.

Fang-Zhou Xing1, Yan-Gang Zhao2, Yuan-Yuan Zhang2, Li He3, Ji-Kai Zhao2, Meng-Ying Liu2, Yan Liu1, Ji-Qiang Zhang2.   

Abstract

AIMS: Estrogens play pivotal roles in hippocampal synaptic plasticity through nuclear receptors (nERs; including ERα and ERβ) and the membrane receptor (mER; also called GPR30), but the underlying mechanism and the contributions of nERs and mER remain unclear. Mammalian target of rapamycin complex 2 (mTORC2) is involved in actin cytoskeleton polymerization and long-term memory, but whether mTORC2 is involved in the regulation of hippocampal synaptic plasticity by ERs is unclear.
METHODS: We treated animals with nER antagonists (MPP/PHTPP) or the mER antagonist (G15) alone or in combination with A-443654, an activator of mTORC2. Then, we examined the changes in hippocampal SRC-1 expression, mTORC2 signaling (rictor and phospho-AKTSer473), actin polymerization (phospho-cofilin and profilin-1), synaptic protein expression (GluR1, PSD95, spinophilin, and synaptophysin), CA1 spine density, and synapse density.
RESULTS: All of the examined parameters except synaptophysin expression were significantly decreased by MPP/PHTPP and G15 treatment. MPP/PHTPP and G15 induced a similar decrease in most parameters except p-cofilin, GluR1, and spinophilin expression. The ER antagonist-induced decreases in these parameters were significantly reversed by mTORC2 activation, except for the change in SRC-1, rictor, and synaptophysin expression.
CONCLUSIONS: nERs and mER contribute similarly to the changes in proteins and structures associated with synaptic plasticity, and mTORC2 may be a novel target of hippocampal-dependent dementia such as Alzheimer's disease as proposed by previous studies.
© 2018 John Wiley & Sons Ltd.

Entities:  

Keywords:  actin polymerization; estrogen receptors; hippocampus; mTORC2; synaptic plasticity

Mesh:

Substances:

Year:  2018        PMID: 29352507      PMCID: PMC6490049          DOI: 10.1111/cns.12806

Source DB:  PubMed          Journal:  CNS Neurosci Ther        ISSN: 1755-5930            Impact factor:   5.243


  61 in total

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  9 in total

1.  Nuclear and membrane estrogen receptor antagonists induce similar mTORC2 activation-reversible changes in synaptic protein expression and actin polymerization in the mouse hippocampus.

Authors:  Fang-Zhou Xing; Yan-Gang Zhao; Yuan-Yuan Zhang; Li He; Ji-Kai Zhao; Meng-Ying Liu; Yan Liu; Ji-Qiang Zhang
Journal:  CNS Neurosci Ther       Date:  2018-01-19       Impact factor: 5.243

Review 2.  Estrogenic hormones receptors in Alzheimer's disease.

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Review 4.  Neurodegenerative Disease: Roles for Sex, Hormones, and Oxidative Stress.

Authors:  Nathalie Sumien; J Thomas Cunningham; Delaney L Davis; Rachel Engelland; Oluwadarasimi Fadeyibi; George E Farmer; Steve Mabry; Paapa Mensah-Kane; Oanh T P Trinh; Philip H Vann; E Nicole Wilson; Rebecca L Cunningham
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Review 5.  Steroid receptor coactivator-1: The central intermediator linking multiple signals and functions in the brain and spinal cord.

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Authors:  Karyn M Frick; Jaekyoon Kim
Journal:  Horm Behav       Date:  2018-05-09       Impact factor: 3.587

7.  GPR30-mediated estrogenic regulation of actin polymerization and spatial memory involves SRC-1 and PI3K-mTORC2 in the hippocampus of female mice.

Authors:  Yuan-Yuan Zhang; Meng-Ying Liu; Zhi Liu; Ji-Kai Zhao; Yan-Gang Zhao; Li He; Wei Li; Ji-Qiang Zhang
Journal:  CNS Neurosci Ther       Date:  2019-02-03       Impact factor: 5.243

8.  MHC II-PI3K/Akt/mTOR Signaling Pathway Regulates Intestinal Immune Response Induced by Soy Glycinin in Hybrid Grouper: Protective Effects of Sodium Butyrate.

Authors:  Bin Yin; Hongyu Liu; Beiping Tan; Xiaohui Dong; Shuyan Chi; Qihui Yang; Shuang Zhang
Journal:  Front Immunol       Date:  2021-01-18       Impact factor: 7.561

9.  Vulnerability to psychological stress-induced anorexia in female mice depends on blockade of ghrelin signal in nucleus tractus solitarius.

Authors:  Chihiro Yamada; Seiichi Iizuka; Miwa Nahata; Tomohisa Hattori; Hiroshi Takeda
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  9 in total

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