Literature DB >> 29251119

Association of TNF-α rs1799964 and IL-1β rs16944 polymorphisms with multiple system atrophy in Chinese Han population.

Xin Zhou1, Chunrong Wang1, Zhao Chen1, Yun Peng1, Huirong Peng1, Xuan Hou1, Wei Ye1, Rong Qiu2, Kun Xia3, Beisha Tang1,3,4, Hong Jiang1,3,4.   

Abstract

BACKGROUND: Recent evidence suggested that several single nucleotide polymorphisms (SNPs) of inflammation-related genes (TNF-α rs1799964, IL-1α rs1800587, IL-1β rs16944, IL-8 rs4073, ICAM-1 rs5498) were associated with multiple system atrophy (MSA). Herein, we conducted this case-control study to evaluate the possible correlation between the five SNPs related to inflammation and MSA in Chinese Han population. METHODS AND PATIENTS: We recruited 154 sporadic patients with MSA and 223 health controls in this study. All subjects were genotyped for the five SNPs using polymerase chain reaction amplification and Sanger sequencing.
RESULTS: TNF-α rs1799964, genotype distribution and minor allele frequency (MAF) showed significant differences between patients and controls, which might illustrate the minor allele C may increase the risk for MSA (genotype, P = 0.006, OR = 1.245, 95% CI = [1.066-1.455]; allele, P = 0.001, OR = 1.887, 95% CI = [1.303-2.733]). For rs16944, patients carrying AA genotype showed a nearly 5-year early age at onset (AAO) than GG genotype (50.52 ± 7.45 years vs. 54.90 ± 7.21 years, P = 0.037). No differences were found in genotype distribution and MAF of the five SNPs between patients with MSA with predominant cerebellar ataxia (MSA-C) and with predominant Parkinsonism (MSA-P).
CONCLUSION: Our study suggests that rs1799964 of TNF-α may act as a risk factor for MSA and the IL-1β rs16944 might be a genetic factor that modifies the AAO in MSA. Moreover, the exact mechanism of neuroinflammatory response in MSA deserves further exploration.

Entities:  

Keywords:  IL-1β rs16944; Multiple system atrophy; TNF-α rs1799964; genetic risk factor; polymorphisms

Mesh:

Substances:

Year:  2018        PMID: 29251119     DOI: 10.1080/00207454.2017.1418346

Source DB:  PubMed          Journal:  Int J Neurosci        ISSN: 0020-7454            Impact factor:   2.292


  5 in total

1.  Analysis of (CAG)n expansion in ATXN1, ATXN2 and ATXN3 in Chinese patients with multiple system atrophy.

Authors:  X Zhou; C Wang; D Ding; Z Chen; Y Peng; H Peng; X Hou; P Wang; X Hou; W Ye; T Li; H Yang; R Qiu; K Xia; J Sequeiros; B Tang; H Jiang
Journal:  Sci Rep       Date:  2018-03-01       Impact factor: 4.379

Review 2.  Cross-examining candidate genes implicated in multiple system atrophy.

Authors:  Jared S Katzeff; Katherine Phan; Sivaraman Purushothuman; Glenda M Halliday; Woojin Scott Kim
Journal:  Acta Neuropathol Commun       Date:  2019-07-24       Impact factor: 7.801

Review 3.  MicroRNAs Dysregulation and Metabolism in Multiple System Atrophy.

Authors:  Chunchen Xiang; Shunchang Han; Jianfei Nao; Shuyan Cong
Journal:  Front Neurosci       Date:  2019-10-17       Impact factor: 4.677

4.  Alterations of the Gut Microbiota in Multiple System Atrophy Patients.

Authors:  Linlin Wan; Xin Zhou; Chunrong Wang; Zhao Chen; Huirong Peng; Xuan Hou; Yun Peng; Puzhi Wang; Tianjiao Li; Hongyu Yuan; Yuting Shi; Xiaocan Hou; Keqin Xu; Yue Xie; Lang He; Kun Xia; Beisha Tang; Hong Jiang
Journal:  Front Neurosci       Date:  2019-10-18       Impact factor: 4.677

5.  High neutrophil-to-lymphocyte ratio predicts short survival in multiple system atrophy.

Authors:  LingYu Zhang; Bei Cao; Yanbing Hou; Qianqian Wei; RuWei Ou; Bi Zhao; Huifang Shang
Journal:  NPJ Parkinsons Dis       Date:  2022-01-20
  5 in total

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