Literature DB >> 29016887

Phase I/II trial of vorinostat combined with temozolomide and radiation therapy for newly diagnosed glioblastoma: results of Alliance N0874/ABTC 02.

Evanthia Galanis1, S Keith Anderson1,2, C Ryan Miller3, Jann N Sarkaria4, Kurt Jaeckle5, Jan C Buckner1, Keith L Ligon6, Karla V Ballman2, Dennis F Moore7, Michael Nebozhyn8, Andrey Loboda9, David Schiff10, Manmeet Singh Ahluwalia11, Eudocia Q Lee12, Elizabeth R Gerstner13, Glenn J Lesser14, Michael Prados15, Stuart A Grossman16, Jane Cerhan17, Caterina Giannini18, Patrick Y Wen12.   

Abstract

Background: Vorinostat, a histone deacetylase (HDAC) inhibitor, has shown radiosensitizing properties in preclinical studies. This open-label, single-arm trial evaluated the maximum tolerated dose (MTD; phase I) and efficacy (phase II) of vorinostat combined with standard chemoradiation in newly diagnosed glioblastoma.
Methods: Patients received oral vorinostat (300 or 400 mg/day) on days 1-5 weekly during temozolomide chemoradiation. Following a 4- to 6-week rest, patients received up to 12 cycles of standard adjuvant temozolomide and vorinostat (400 mg/day) on days 1-7 and 15-21 of each 28-day cycle. Association between vorinostat response signatures and progression-free survival (PFS) and overall survival (OS) was assessed based on RNA sequencing of baseline tumor tissue.
Results: Phase I and phase II enrolled 15 and 107 patients, respectively. The combination therapy MTD was vorinostat 300 mg/day and temozolomide 75 mg/m2/day. Dose-limiting toxicities were grade 4 neutropenia and thrombocytopenia and grade 3 aspartate aminotransferase elevation, hyperglycemia, fatigue, and wound dehiscence. The primary efficacy endpoint in the phase II cohort, OS rate at 15 months, was 55.1% (median OS 16.1 mo), and consequently, the study did not meet its efficacy objective. Most common treatment-related grade 3/4 toxicities in the phase II component were lymphopenia (32.7%), thrombocytopenia (28.0%), and neutropenia (21.5%). RNA expression profiling of baseline tumors (N = 76) demonstrated that vorinostat resistance (sig-79) and sensitivity (sig-139) signatures had a reverse and positive association with OS/PFS, respectively. Conclusions: Vorinostat combined with standard chemoradiation had acceptable tolerability in newly diagnosed glioblastoma. Although the primary efficacy endpoint was not met, vorinostat sensitivity and resistance signatures could facilitate patient selection in future trials.

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Year:  2018        PMID: 29016887      PMCID: PMC5909661          DOI: 10.1093/neuonc/nox161

Source DB:  PubMed          Journal:  Neuro Oncol        ISSN: 1522-8517            Impact factor:   12.300


  50 in total

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3.  Modulation of radiation response by histone deacetylase inhibition.

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Review 8.  Grand rounds at the National Institutes of Health: HDAC inhibitors as radiation modifiers, from bench to clinic.

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10.  IDH mutation, 1p19q codeletion and ATRX loss in WHO grade II gliomas.

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