Literature DB >> 28955739

Post-translationally modified human lens crystallin fragments show aggregation in vitro.

O P Srivastava1, K Srivastava1, J M Chaves1, A K Gill1.   

Abstract

BACKGROUND: Crystallin fragments are known to aggregate and cross-link that lead to cataract development. This study has been focused on determination of post-translational modifications (PTMs) of human lens crystallin fragments, and their aggregation properties.
METHODS: Four crystallin fragments-containing fractions (Fraction I [∼3.5 kDa species], Fraction II [∼3.5-7 kDa species], Fraction III [∼7-10 kDa species] and Fraction IV [>10-18 kDa species]), and water soluble high molecular weight (WS-HMW) protein fraction were isolated from water soluble (WS) protein fraction of human lenses of 50-70 year old-donors. The crystallin fragments of the Fractions I-IV were separated by two-dimensional (2D)-gel electrophoresis followed by analysis of their gel-spots by mass spectrometry. The Fractions I-IV were examined for their molecular mass, particle-diameters, amyloid fibril formation, and for their aggregation by themselves and with WS-HMW proteins.
RESULTS: Crystallin fragments in Fractions I-IV were derived from α-, β- and γ-crystallins, and their 2D-gel separated spots contained multiple crystallins with PTMs such as oxidation, deamidation, methylation and acetylation. Crystallin fragments from all the four fractions exhibited self-aggregated complexes ranging in Mr from 5.5×105 to 1.0×108 Da, with diameters of 10-28 nm, and amyloid fibril-like formation, and aggregation with WS-HMW proteins.
CONCLUSION: The crystallin fragments exhibited several PTMs, and were capable of forming aggregated species by themselves and with WS-HMW proteins, suggesting their potential role in aggregation process during cataract development. GENERAL SIGNIFICANCE: Crystallin fragments play a major role in human cataract development.

Entities:  

Keywords:  Amyloid; Cataract; Crystallins; Lens; Post-translational modifications; Two-dimensional gel electrophoresis

Year:  2017        PMID: 28955739      PMCID: PMC5614626          DOI: 10.1016/j.bbrep.2017.01.011

Source DB:  PubMed          Journal:  Biochem Biophys Rep        ISSN: 2405-5808


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