Literature DB >> 28794251

Clinicopathologic features of myositis patients with CD8-MHC-1 complex pathology.

Chiseko Ikenaga1, Akatsuki Kubota1, Masato Kadoya1, Kenichiro Taira1, Naohiro Uchio1, Ayumi Hida1, Meiko Hashimoto Maeda1, Yu Nagashima1, Hiroyuki Ishiura1, Kenichi Kaida1, Jun Goto1, Shoji Tsuji1, Jun Shimizu2.   

Abstract

OBJECTIVE: To determine the clinical features of myositis patients with the histopathologic finding of CD8-positive T cells invading non-necrotic muscle fibers expressing major histocompatibility complex class 1 (CD8-MHC-1 complex), which is shared by polymyositis (PM) and inclusion body myositis (IBM), in relation to the p62 immunostaining pattern of muscle fibers.
METHODS: All 93 myositis patients with CD8-MHC-1 complex who were referred to our hospital from 1993 to 2015 were classified on the basis of the European Neuromuscular Center (ENMC) diagnostic criteria for IBM (Rose, 2013) or PM (Hoogendijk, 2004) and analyzed.
RESULTS: The 93 patients included were 17 patients with PM, 70 patients with IBM, and 6 patients who neither met the criteria for PM nor IBM in terms of muscle weakness distribution (unclassifiable group). For these PM, IBM, and unclassifiable patients, their mean ages at diagnosis were 63, 70, and 64 years; autoimmune disease was present in 7 (41%), 13 (19%), and 4 (67%); hepatitis C virus infection was detected in 0%, 13 (20%), and 2 (33%); and p62 was immunopositive in 0%, 66 (94%), and 2 (33%), respectively. Of the treated patients, 11 of 16 PM patients and 4 of 6 p62-immunonegative patients in the unclassifiable group showed responses to immunotherapy, whereas all 44 patients with IBM and 2 p62-immunopositive patients in the unclassifiable group were unresponsive to immunotherapy.
CONCLUSIONS: CD8-MHC-1 complex is present in patients with PM, IBM, or unclassifiable group. The data may serve as an argument for a trial of immunosuppressive treatment in p62-immunonegative patients with unclassifiable myositis.
© 2017 American Academy of Neurology.

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Year:  2017        PMID: 28794251     DOI: 10.1212/WNL.0000000000004333

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  9 in total

Review 1.  Update on Inclusion Body Myositis.

Authors:  Duaa Jabari; V V Vedanarayanan; Richard J Barohn; Mazen M Dimachkie
Journal:  Curr Rheumatol Rep       Date:  2018-06-28       Impact factor: 4.592

Review 2.  New Developments in the Genetics of Inclusion Body Myositis.

Authors:  Kyla A Britson; Stephanie Y Yang; Thomas E Lloyd
Journal:  Curr Rheumatol Rep       Date:  2018-04-02       Impact factor: 4.592

Review 3.  Updates on the Immunopathology in Idiopathic Inflammatory Myopathies.

Authors:  Akinori Uruha; Hans-Hilmar Goebel; Werner Stenzel
Journal:  Curr Rheumatol Rep       Date:  2021-07-01       Impact factor: 4.592

4.  Inflammatory myopathy with myasthenia gravis: Thymoma association and polymyositis pathology.

Authors:  Naohiro Uchio; Kenichiro Taira; Chiseko Ikenaga; Masato Kadoya; Atsushi Unuma; Kenji Yoshida; Setsu Nakatani-Enomoto; Yuki Hatanaka; Yasuhisa Sakurai; Yasushi Shiio; Kenichi Kaida; Akatsuki Kubota; Tatsushi Toda; Jun Shimizu
Journal:  Neurol Neuroimmunol Neuroinflamm       Date:  2018-12-24

5.  Comprehensive transcriptomic analysis shows disturbed calcium homeostasis and deregulation of T lymphocyte apoptosis in inclusion body myositis.

Authors:  Mridul Johari; Anna Vihola; Johanna Palmio; Manu Jokela; Per Harald Jonson; Jaakko Sarparanta; Sanna Huovinen; Marco Savarese; Peter Hackman; Bjarne Udd
Journal:  J Neurol       Date:  2022-03-02       Impact factor: 6.682

6.  NLRP3 inflammasome up-regulates major histocompatibility complex class I expression and promotes inflammatory infiltration in polymyositis.

Authors:  Ping Xia; Yu-Quan Shao; Cong-Cong Yu; Yu Xie; Zhi-Jie Zhou
Journal:  BMC Immunol       Date:  2022-08-14       Impact factor: 3.594

Review 7.  Current Classification and Management of Inflammatory Myopathies.

Authors:  Jens Schmidt
Journal:  J Neuromuscul Dis       Date:  2018

8.  A Patient with HCV Infection and a Sustained Virological Response to Direct-acting Antiviral Treatment Who Developed Inclusion Body Myositis.

Authors:  Toru Kuwano; Norio Akuta; Fumitaka Suzuki; Shunichiro Fujiyama; Yusuke Kawamura; Hitomi Sezaki; Tetsuya Hosaka; Satoshi Saitoh; Masahiro Kobayashi; Yoshiyuki Suzuki; Mariko Kobayashi; Yasuji Arase; Kenji Ikeda; Hiromitsu Kumada
Journal:  Intern Med       Date:  2018-03-30       Impact factor: 1.271

9.  Muscle Transcriptomics Shows Overexpression of Cadherin 1 in Inclusion Body Myositis.

Authors:  Chiseko Ikenaga; Hidetoshi Date; Motoi Kanagawa; Jun Mitsui; Hiroyuki Ishiura; Jun Yoshimura; Iago Pinal-Fernandez; Andrew L Mammen; Thomas E Lloyd; Shoji Tsuji; Jun Shimizu; Tatsushi Toda; Jun Goto
Journal:  Ann Neurol       Date:  2022-02-11       Impact factor: 11.274

  9 in total

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