Literature DB >> 28733034

Decreased expression of miR-150, miR146a and miR424 in type 1 diabetic patients: Association with ongoing islet autoimmunity.

Guofeng Wang1, Yong Gu2, Ning Xu1, Mei Zhang3, Tao Yang4.   

Abstract

BACKGROUND: Recent studies revealed altered miRNAs profiling in patients with autoimmune diseases, including type 1 diabetes (T1D). Here, we conducted an observational study and examined the expression levels of these five miRNAs (miR-150,miR-146a,miR-424,miR-181a and miR-142-3P) in peripheral blood mononuclear cells (PBMCs) from T1D patients.
METHODS: PBMCs were obtained from T1D cohorts (n = 78), type 2 diabetes (T2D, n = 46) and healthy control subjects (n = 56). Quantitative analysis of five miRNAs were performed using SYBR quantitative real time PCR (qRT-PCR). All values were normalized to endogenous control U6. Then, We compared expression level of five miRNAs in PBMCs from T1DM and age-matched healthy controls, and related the miRNAs expression levels to beta-cell function, autoantibodies and glycaemic control in T1D cohort.
RESULTS: We identified decreased miR-150, miR-146a and miR-424 in patients with T1D, which can distinguished from non-diabetic patients and T2D patients (p < 0.05). Furthermore, they were significantly decreased in PBMCs from GADA autoantibody-positive (Ab+) patients versus GADA autoantibody-negative (Ab-) patients. However, there was no correlation between characteristics such as age, sex, diabetes duration, C-peptide, and glycaemic, and the expression level of miRNAs (p > 0.05).
CONCLUSIONS: Our study indicated miR-150, miR-146a and miR-424 may be potential biomarkers of T1DM with applications in the clinical setting as well as provided new insights into the molecular mechanisms involved in this disorder.
Copyright © 2017. Published by Elsevier Inc.

Entities:  

Keywords:  Autoantibodies; Autoimmunity; Biomarker; Type 1 diabetes; microRNA

Mesh:

Substances:

Year:  2017        PMID: 28733034     DOI: 10.1016/j.bbrc.2017.06.196

Source DB:  PubMed          Journal:  Biochem Biophys Res Commun        ISSN: 0006-291X            Impact factor:   3.575


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