Literature DB >> 28650356

Trending Fibrinolytic Dysregulation: Fibrinolysis Shutdown in the Days After Injury Is Associated With Poor Outcome in Severely Injured Children.

Christine M Leeper1, Matthew D Neal, Christine J McKenna, Barbara A Gaines.   

Abstract

OBJECTIVE: To trend fibrinolysis after injury and determine the influence of traumatic brain injury (TBI) and massive transfusion on fibrinolysis status.
BACKGROUND: Admission fibrinolytic derangement is common in injured children and adults, and is associated with poor outcome. No studies examine fibrinolysis days after injury.
METHODS: Prospective study of severely injured children at a level 1 pediatric trauma center. Rapid thromboelastography was obtained on admission and daily for up to 7 days. Standard definitions of hyperfibrinolysis (HF; LY30 ≥3), fibrinolysis shutdown (SD; LY30 ≤0.8), and normal (LY30 = 0.9-2.9) were applied. Antifibrinolytic use was documented. Outcomes were death, disability, and thromboembolic complications. Wilcoxon rank-sum and Fisher exact tests were performed. Exploratory subgroups included massively transfused and severe TBI patients.
RESULTS: In all, 83 patients were analyzed with median (interquartile ranges) age 8 (4-12) and Injury Severity Score 22 (13-34), 73.5% blunt mechanism, 47% severe TBI, 20.5% massively transfused. Outcomes were 14.5% mortality, 43.7% disability, and 9.8% deep vein thrombosis. Remaining in or trending to SD was associated with death (P = 0.007), disability (P = 0.012), and deep vein thrombosis (P = 0.048). Median LY30 was lower on post-trauma day (PTD)1 to PTD4 in patients with poor compared with good outcome; median LY30 was lower on PTD1 to PTD3 in TBI patients compared with non-TBI patients. HF without associated shutdown was not related to poor outcome, but extreme HF (LY30 >30%, n = 3) was lethal. Also, 50% of massively transfused patients in hemorrhagic shock demonstrated SD physiology on admission. All with HF (fc31.2%) corrected after hemostatic resuscitation without tranexamic acid.
CONCLUSIONS: Fibrinolysis shutdown is common postinjury and predicts poor outcomes. Severe TBI is associated with sustained shutdown. Empiric antifibrinolytics for children should be questioned; thromboelastography-directed selective use should be considered for documented HF.

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Mesh:

Year:  2017        PMID: 28650356     DOI: 10.1097/SLA.0000000000002355

Source DB:  PubMed          Journal:  Ann Surg        ISSN: 0003-4932            Impact factor:   12.969


  12 in total

Review 1.  Fibrinolysis Shutdown in Trauma: Historical Review and Clinical Implications.

Authors:  Hunter B Moore; Ernest E Moore; Matthew D Neal; Forest R Sheppard; Lucy Z Kornblith; Dominik F Draxler; Mark Walsh; Robert L Medcalf; Mitch J Cohen; Bryan A Cotton; Scott G Thomas; Christine M Leeper; Barbara A Gaines; Angela Sauaia
Journal:  Anesth Analg       Date:  2019-09       Impact factor: 5.108

2.  Does Tranexamic Acid Improve Clot Strength in Severely Injured Patients Who Have Elevated Fibrin Degradation Products and Low Fibrinolytic Activity, Measured by Thrombelastography?

Authors:  Hunter B Moore; Ernest E Moore; Michael P Chapman; Kirk C Hansen; Mitchell J Cohen; Frederic M Pieracci; James Chandler; Angela Sauaia
Journal:  J Am Coll Surg       Date:  2019-03-29       Impact factor: 6.113

Review 3.  Fibrinolysis in trauma: a review.

Authors:  M J Madurska; K A Sachse; J O Jansen; T E Rasmussen; J J Morrison
Journal:  Eur J Trauma Emerg Surg       Date:  2017-09-16       Impact factor: 3.693

Review 4.  Blood Loss and Transfusion in a Pediatric Scoliosis Surgery Cohort in the Antifibrinolytic Era.

Authors:  Carolyn G Ahlers; Matthews Lan; Jonathan G Schoenecker; Alexandra J Borst
Journal:  J Pediatr Hematol Oncol       Date:  2022-04-01       Impact factor: 1.289

5.  Endotheliopathy is Associated with a 24-hour Fibrinolysis Phenotype Described by Low TEG Lysis and High D-Dimer after Trauma: a Secondary Analysis of the PROPPR Study.

Authors:  Robert P Richter; Danielle M Joiner; Russell L Griffin; Jan O Jansen; Jeffrey D Kerby; Charles E Wade; John B Holcomb; Jessica C Cardenas; Jillian R Richter
Journal:  Ann Surg Open       Date:  2022-03

6.  It's sooner than you think: Blunt solid organ injury patients are already hypercoagulable upon hospital admission - Results of a bi-institutional, prospective study.

Authors:  Julia R Coleman; Annika B Kay; Ernest E Moore; Hunter B Moore; Eduardo Gonzalez; Sarah Majercik; Mitchell J Cohen; Thomas White; Fredric M Pieracci
Journal:  Am J Surg       Date:  2019-09-10       Impact factor: 2.565

Review 7.  Time Course of Hemostatic Disruptions After Traumatic Brain Injury: A Systematic Review of the Literature.

Authors:  Alexander Fletcher-Sandersjöö; Eric Peter Thelin; Marc Maegele; Mikael Svensson; Bo-Michael Bellander
Journal:  Neurocrit Care       Date:  2021-04       Impact factor: 3.210

Review 8.  Trauma-induced coagulopathy.

Authors:  Ernest E Moore; Hunter B Moore; Lucy Z Kornblith; Matthew D Neal; Maureane Hoffman; Nicola J Mutch; Herbert Schöchl; Beverley J Hunt; Angela Sauaia
Journal:  Nat Rev Dis Primers       Date:  2021-04-29       Impact factor: 65.038

Review 9.  Does Complement-Mediated Hemostatic Disturbance Occur in Traumatic Brain Injury? A Literature Review and Observational Study Protocol.

Authors:  Alexander Fletcher-Sandersjöö; Marc Maegele; Bo-Michael Bellander
Journal:  Int J Mol Sci       Date:  2020-02-26       Impact factor: 5.923

10.  A rat model of orthopedic injury-induced hypercoagulability and fibrinolytic shutdown.

Authors:  Kristen T Carter; Ana C Palei; Frank T Spradley; Brycen M Witcher; Larry Martin; Robert L Hester; Matthew E Kutcher
Journal:  J Trauma Acute Care Surg       Date:  2020-11       Impact factor: 3.697

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