Literature DB >> 28643456

IRF6 expression in basal epithelium partially rescues Irf6 knockout mice.

Youssef A Kousa1,2, Dina Moussa3, Brian C Schutte1,3,4.   

Abstract

BACKGROUND: Mutations in IRF6, CHUK (IKKA), and RIPK4 can lead to a disease spectrum that includes cutaneous, limb, and craniofacial malformations. Loss of these alleles in the mouse leads to perinatal lethality and severe cutaneous, limb, and craniofacial defects also. Genetic rescue in the mouse has been shown for Ikka and Ripk4.
RESULTS: Here, we show partial genetic rescue of Irf6 knockout embryos using the KRT14 promoter to drive Irf6 expression in the basal epithelium. In contrast to Irf6 knockout embryos, rescue embryos survive the immediate perinatal period. Macroscopic examination reveals rescue of skin adhesions between the axial and appendicular skeleton. Unexpectedly, KRT14-driven Irf6 expression does not completely rescue orofacial clefting and adhesions between the palate and tongue, suggesting the importance of cell-autonomous IRF6 expression in periderm. Like knockout embryos, Irf6 rescue embryos also have persistent esophageal adhesions, which likely contribute to postnatal demise.
CONCLUSIONS: Together, these data suggest that targeted expression of IRF6 can significantly reduce disease severity, but that a minimum level of Irf6 in both periderm and basal epithelial cells is necessary for orofacial development. Therefore, homologous human and mouse phenotypes are observed for IRF6, IKKA, and RIPK4. In this work, we show that altering the expression level of IRF6 dramatically modified this phenotype in utero. Developmental Dynamics 246:670-681, 2017.
© 2017 Wiley Periodicals, Inc. © 2017 Wiley Periodicals, Inc.

Entities:  

Keywords:  GRHL3; IRF6; Van der Woude syndrome; cleft lip and palate; epidermis; epithelium; genetic rescue; limb defects; mouse models; oral periderm; popliteal pterygium syndrome

Mesh:

Substances:

Year:  2017        PMID: 28643456      PMCID: PMC5552062          DOI: 10.1002/dvdy.24537

Source DB:  PubMed          Journal:  Dev Dyn        ISSN: 1058-8388            Impact factor:   3.780


  27 in total

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4.  Dominant mutations in GRHL3 cause Van der Woude Syndrome and disrupt oral periderm development.

Authors:  Myriam Peyrard-Janvid; Elizabeth J Leslie; Youssef A Kousa; Tiffany L Smith; Martine Dunnwald; Måns Magnusson; Brian A Lentz; Per Unneberg; Ingegerd Fransson; Hannele K Koillinen; Jorma Rautio; Marie Pegelow; Agneta Karsten; Lina Basel-Vanagaite; William Gordon; Bogi Andersen; Thomas Svensson; Jeffrey C Murray; Robert A Cornell; Juha Kere; Brian C Schutte
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Review 5.  Toward an orofacial gene regulatory network.

Authors:  Youssef A Kousa; Brian C Schutte
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9.  Cell-autonomous and non-cell-autonomous roles for IRF6 during development of the tongue.

Authors:  Steven Goudy; Peggi Angel; Britni Jacobs; Cynthia Hill; Veronica Mainini; Arianna L Smith; Youssef A Kousa; Richard Caprioli; Lawrence S Prince; Scott Baldwin; Brian C Schutte
Journal:  PLoS One       Date:  2013-02-22       Impact factor: 3.240

10.  Disruption of an AP-2alpha binding site in an IRF6 enhancer is associated with cleft lip.

Authors:  Fedik Rahimov; Mary L Marazita; Axel Visel; Margaret E Cooper; Michael J Hitchler; Michele Rubini; Frederick E Domann; Manika Govil; Kaare Christensen; Camille Bille; Mads Melbye; Astanand Jugessur; Rolv T Lie; Allen J Wilcox; David R Fitzpatrick; Eric D Green; Peter A Mossey; Julian Little; Regine P Steegers-Theunissen; Len A Pennacchio; Brian C Schutte; Jeffrey C Murray
Journal:  Nat Genet       Date:  2008-10-05       Impact factor: 38.330

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6.  SPECC1L regulates palate development downstream of IRF6.

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